[00:00:00] Carl Lanore: [00:00:00] welcome back to another episode of super human radio. If you didn't know this already, we do these shows. Facebook live now, so you can actually join us, watch us post questions and all that sort of stuff. If you're near your computer, I, a lot of people probably need their computers. A lot of people at home, the kids are home.

[00:00:15] They're home. I only have my guests today because the ore is closed for, you know, non life threatening surgeries. And so, uh, we take advantage of opportunities when they come along because we're intelligent. We are the superhuman nation. After all. Today is March 18th, 2020. We are deep into the beginnings of a self incarceration, uh, cutting back on social contact in the face of the, uh.

[00:00:46] Contagious covert 19 virus. This is a good time. You know, people out there beating each other up at the, at the grocery stores trying to buy food and toilet paper. But we live in a great world today [00:01:00] where you can do everything online and one of the things you can do online right now is go to eat legendary.com.

[00:01:06] And order the most amazing nut butters in the world. Elisa said, can't believe I finished, uh, a jar of the, a Apple pie, a nut butter in two days. I use it as dessert. I take a spoonful of it and put it in my mouth, and I feel like I had dessert and I'm. While you're there, check out the tasty pastry, which is the a version of the pop tart that you can actually give to your kids and feel good about because it has nine grams of protein, less than one gram of sugar.

[00:01:37] A great stuff tastes great. They'll think they're eating junk. Don't tell them, don't tell them. Uh, go to eat legendary.com today and let them know you heard about them here. At super human radio. And so now we bring my guest on and he's been on the show before and that is, Oh, I gotta turn my solo off.

[00:01:56] Here we go. There he is. Dr. William seeds. How you doing, dr [00:02:00] seeds

[00:02:01] Dr. William Seeds, MD: [00:02:01] are all doing great. Where's your cap? Usually you got your cap on what's going on?

[00:02:05] Carl Lanore: [00:02:05] I got my hair colored so now I don't have to wear it. You know, I'm not brave like you, you know, you look better. You look really good. I don't look good with gray hair.

[00:02:14] I look, I look very, very old. Very old.

[00:02:16] Dr. William Seeds, MD: [00:02:16] I've had gray hair, I think since I was 12

[00:02:20] Carl Lanore: [00:02:20] that's interesting. Some people do, and my friend Billy Mitchell went gray pretty young as well. Yeah, that's really interesting. So anyway, we have you today because the O R is closed, right? Doc.

[00:02:31] Dr. William Seeds, MD: [00:02:31] Yes, it is. Yes, it is that the, uh, state put its mandation is in.

[00:02:37] And, uh, I think we as a community of physicians also felt that, uh, you know, we have to do our part and trying to do our best to social distance. And really the, I think more importantly, it's, it's keeping our patients safe and keeping them away from the hospitals if we can, and keeping them isolated to, to get through this as quickly as we can.

[00:02:59] So. [00:03:00] You know, we're all in full support and we want this under control so we can keep this, uh, safe for.

[00:03:09] Carl Lanore: [00:03:09] Oh,

[00:03:10] Dr. William Seeds, MD: [00:03:10] yeah.

[00:03:12] Carl Lanore: [00:03:12] We want it to end sooner rather than later. Right. We don't, we're not trying to prove anything here. No, it's, I'm all for it. You know, I've got a home gym. I know you have an amazing home gym. Uh, you have actually w w some people would consider a commercial gym out at, back, at the back of your home.

[00:03:28] Uh, but yeah, you know, Elisa and I, uh, with it, we're training at home. I tried to get a Prowler real quick. I'm going to have to resort to pushing my car for a little while. Oh,

[00:03:37] Dr. William Seeds, MD: [00:03:37] hello, Carla. I showed you my new, uh, my new squat belt, uh, uh, platform. I hooked up to my, uh, my, uh, uh, my rack might swap back.

[00:03:49] Carl Lanore: [00:03:49] No, no, you haven't.

[00:03:50] I know you told me you got it. It's really nice, you know.

[00:03:53] Dr. William Seeds, MD: [00:03:53] Oh my gosh. Talk about what have I been missing for years? It's just a, you know, it's another way to train and it's [00:04:00] a. Makes it fun. It puts, takes that stress off my back and my shoulders. So I'm excited to, uh, to continue that, uh, process. So where I go with,

[00:04:12] Carl Lanore: [00:04:12] yeah.

[00:04:12] Very, very good. So let's, let's let, let me kind of lead us down this path because I think this is a very, very intriguing discussion today. All right. We've gone, hello, Oakey, all has been well known for a long time for a variety of things. Including, uh, uh, you know, mitigating stress and, and stuff like that.

[00:04:34] But the, the reality is it wasn't really an effective therapeutic because it has a super duper short half life. Correct me if I'm wrong, but it's like 30 minutes or something like that,

[00:04:47] Dr. William Seeds, MD: [00:04:47] right? Yeah. It's shorter than that, and it's. You need high levels and in humans too, to really be effective and, uh, to, uh, penetrate the cell, get across the [00:05:00] blood, brain barrier, uh, those types of things.

[00:05:02] And so, uh, we had a brilliant chemist, uh, Justin Curtin who, uh, who so took this on and he, uh, he made this into a cyclic compound that is about 30. Has about three times the power of the, uh, original Hinoki IOL. And it's a, what an advancement. I mean, I can't tell you, I cannot tell you how significant this supplement has been and in my ability to help people, uh, through a multitude of, of aspects, not just as a, you know, as an anti, uh, as an anoxic lytic as an antianxiety type of medication.

[00:05:46] But as a. As a supplement is as an antiinflammatory, any depressant and from biotic, all of these things are real and. Um, anti-spasmodic spasmodic, antidepressive [00:06:00] antitumor genic which I think we're going to talk

[00:06:02] Carl Lanore: [00:06:02] about. Cause that's, that's one of, that's one of the areas that people don't really aren't clear on it.

[00:06:07] So, but first I gotta say Justin Kirkland is a chemical superhero whose name you probably never heard of, but he is behind more formulas. We're using novel molecules from, from companies like, uh, like Gaspari nutrition all the way down. You've never heard his name before, but the guy is not only brilliant, but he has the inquisitiveness of a child.

[00:06:32] So he discovers things all the time and people don't know who he is, but they will know who he is in upcoming months. I promise you that. So. The real, the real problem then with Hinoki oil, even though we've known all along that it's got some magical benefits, is that you, you'd have to dose it to people every 10 minutes to see any effectiveness, and that's not a real, that's not a a possibility.

[00:06:58] So this form of [00:07:00] honokiol stays around longer, but it is also stronger as well. Is that correct? It's not just that it stays around longer. It's also stronger? Correct. Okay.

[00:07:09] Dr. William Seeds, MD: [00:07:09] It has, it has real therapeutic effects in the, in the cell line, in vitro and in vivo. And I'll tell you what, even, I mean, I know we're going to talk about cancer, but I don't think anybody realizes the significance of the antiviral effects that  has and, and how that can play a significant role today for people who, you know, there, there's so many things out there where people are trying to do whatever they can to.

[00:07:36] To upregulate their immune system to help them work against something where there really aren't a lot of alternatives other than what we've discussed. I think there are significant things that we've discussed and had been brought up about peptides, which I won't continue with, but I'm going to tell you this cheap alternative of a supplement is very, very intriguing where we know how Nokia has [00:08:00] profound effects on.

[00:08:02] Herpes, a viral replication on CMV viral replication, um, on a hepatitis C, hepatitis C, viral replication. I mean, it can control viral replication in the cell, and that's significant. And also Hinoki IOL has an acute effect of increasing interferon. And interferon is the key, I think, to working against. Uh, any type of viral replication, because that's what the body does.

[00:08:34] When the body upregulates its immune system, it turns on interferon and interferon in the cell. What does that do? It sets in motion. This process of where it tries to starve the virus through what's called the moon mevalonate a sterile pathway. It starves it from replicating, and I'm going to tell you something Carl.

[00:09:00] [00:08:59] That is a very well known mechanism of what we do to upregulate the immune system. And I think everybody should be taking the advantage of trying to do everything they can to keep their immune system to where they can challenge. Any type of viral, um, influx and replication in the cell. And I think I have really, you know, all our, all our studies of a honokiol are in vitro, in vivo studies that are in the animals.

[00:09:32] And, and they have shown human studies with the viral replication. Uh, we have nothing specific to show you that it works against. This Corona virus is, uh, coven 19. Bottle replication is Bao replication.

[00:09:48] Carl Lanore: [00:09:48] Oh wait, wait, wait, wait. But, but the, the one unique thing about VI this virus and if it's a family of retroviruses, is that they harness the [00:10:00] enzyme reverse transcriptase in order to build themselves into our cellular DNA, and they turn every cell into a factory.

[00:10:06] So would we, would interferon help with a retrovirus? You think.

[00:10:12] Dr. William Seeds, MD: [00:10:12] Well, it's all basic stuff, Carl, that that has nothing to do. The cell, the virus is going to replicate. It needs them Valant the mevalonate sterile pathway in every cell. It needs that to replicate. If it doesn't have that as energy, it's not replicated.

[00:10:30] That's a fact. So that's the way our immune system works in, in trying it first to starve off the replication. And then it up-regulates our own inflamma zones if it produces cytokines of chemokine Institute, cause a pop ptosis of the cell, that that's how we attack these viruses. Um, so the. And in fact, in fact, with this specific virus, it's becoming, [00:11:00] it's very similar in homology to the SARS Kovi one virus in 2001 to 2003 it's got like 72% amino acid sequence homology.

[00:11:11] Very, very similar. And the tertiary domain is very similar. So we're finding out that all of the symptoms. The way this approaches and and infiltrates into the cell, what it, what it's doing is it has this spike protein. That goes after what's called, um, angiotensin converting enzyme to, and that's a receptor for it.

[00:11:38] And that's how it enters into the cell. And in fact, we learned from SARS, you know, in 2002, 2003 in which is being used now. Uh, in, uh, Italy. In China, in France, they're using, um, NG Tensen blocking receptor agents. [00:12:00] These, the ACE two enzyme, which is kind of a paradoxical thing, but they're trying to upregulate it to fight this in the people that are severely

[00:12:09] Carl Lanore: [00:12:09] sick.

[00:12:10] So I wait a minute, I want to better understand that. So what you're saying is that. Upregulating uh, ACE actually may have a protective effect. So taking an ACE inhibitor is a bad idea, but an ARB where you're blocking the receptor may actually be a good idea.

[00:12:27] Dr. William Seeds, MD: [00:12:27] Yeah, it's a, it's a ACE too. Not, not a SWAT ACE one.

[00:12:32] ACE one's what's converts engine tents into angiotensin two. All right, so Andy Tensen two's the bad player. Um, and that Angie Tencent too. What happens is you have eight two, which is angiotensin converting enzyme. Two you have that around to control the amount of angiotensin. What it does is it, it changes NG tens and two into angiotensin [00:13:00] one seven.

[00:13:01] And then that works on a mass receptor and it has a, uh, it has a positive effect. We're NG tents and. Two works on the angiotensin one receptor and has a negative effect. So what's happening is, this is why people, this is why the older people with hypertension, diabetes, they have an activated reninangiotencin system.

[00:13:24] It's, and it's why younger people don't really have issues with this because they have plenty of ACE too around. Older people don't have a lot of A's too, because it's been, it's not being converted. And that's exactly why this is a, a disease more specific to, uh, to people with, with w where, where we're realizing Grennan angiotensin system as an inflammatory, uh, cascade problem in the lungs is, is all about this process.

[00:13:56] And this is why it's so exciting to see. [00:14:00] Um, to see the revelations of what we learned from SAR, uh, in 2000 and, uh, in two, 2003 and now we have some ways that are actually working, um, with these arms in, in trying to upregulate that, uh, paradoxical shift and getting enough of this ACE to, and, um. Uh, I, I think that's really, really important to understand that we, we really do know some of these mechanisms.

[00:14:28] Now. This is all, this is all theoretical and, and we have no, you know, we have no papers other than what will be produced after we go through this process. Um, but boy, we've got some really, really, I think solid things to work on it, helping these people that already hype, that are, you know, hypertensive, uh.

[00:14:50] In the beginning of their state of, uh, going through this inflammatory cascade of lung disease. And, and I think that's, that's significant that we [00:15:00] have this understanding

[00:15:01] Carl Lanore: [00:15:01] what role does, uh, the cytokine storm that we see in people with, uh, with autoimmunity, uh, have to do with possibly being at greater risk of, of, of death from a virus like this.

[00:15:20] Dr. William Seeds, MD: [00:15:20] Well, you've already locked. So, so this has a lot to do with, with this. It's this player, this upregulation of this transcription factor that's called nuclear factor Kappa B. And this transcription factors, what the nucleus transcribes when it's.

[00:15:44] Carl Lanore: [00:15:44] Okay. Well you drop the drop down. You dropped out for just a second. Could you, could you just go back, I'm sorry to make you do this, but we lost your, uh, your connection for a second. Just start again with this, this comment you're making right now, please.

[00:16:00] [00:15:59] Dr. William Seeds, MD: [00:15:59] So this has to do with what's called a transcription factor in the nucleus is all nuclear factor Kappa B.

[00:16:08] That is an important transcription factor that the nucleus will transcribe to produce the cytokines like interleukin one, beta Kuma necrosis factor, interleukin six, and other, uh, chemo tactic factors that are, that are very significant in the initiation of this innate immune response. That's going after this virus that has struck.

[00:16:36] It's like it's, it didn't get, so you'd look at the cell that we talked about before where it was trying to star at first with the interferon, it was trying to starve that viral replication. And that viral replication over took that part. So the second part that's turned on in the cell is this inflamma zone, which has an alarm system that starts that transcription of [00:17:00] nuclear factor Kappa B to make these cytokines and chemo kinds to, to kill the virus and draw your immune system in the kill the cell and go through a pop ptosis.

[00:17:10] But what happens. Is that gets overwritten to and, and gets into higher function and start causing more chemokine cytokines to come into play. And that's when you get into your, you're more into your septic type of roles. And so people who are immune compromised are already there and don't have that ability to.

[00:17:33] You know, to directly attack, um, the, uh, th that viral load and as you would like to initially. So it's a little more complicated, but that's a good way to think about it, I think in understanding it.

[00:17:46] Carl Lanore: [00:17:46] Yeah, no, a unique medical has a question. So are you saying, uh, or possibly suggesting that the AR B's, angiotensin receptor blockers could be used for prophylaxis?

[00:18:00] [00:18:00] Dr. William Seeds, MD: [00:18:00] Well, obviously you don't want to put anybody into, you know, you don't want to make anybody, um, hypotensive, right? So these are mainly, these are air bees. Uh, ARBs are typically used for hypertension and some glommed, uh, some kidney diseases. Um, uh, telmisartan is something that is an orbit that is used, uh.

[00:18:25] Prophylactically for higher glucose levels, hyperglycemia or prevention. That may be something you could, you could utilize as an Arbonne, may not cause the hypotensive things you're looking at. So could it be a prophylactic? Yes, possibly. Um, you have to be careful with it though. And again, we're speculating, you know, that that's the important part here, but, but the science is there.

[00:18:50] There's no doubt about that. The science that we're discussing is. Is, uh, I'm, I'm going to say that not a lot of people know about [00:19:00] this, this ACE to stuff that was discovered from, from the stars. But I think it's a very, you know, that's a great question that unique brings up. Um, I think if you, if you handle it, if you go with lower doses, I think with something like a telmisartan, which is T, which is used as a prophylaxis for higher, you know, to stop A1C growth, that things like that.

[00:19:24] I think that possibly could be used like that. That's a, that's a really novel way of thinking of it. Uh, I, I haven't really put a lot of thought to that,

[00:19:33] Carl Lanore: [00:19:33] but I would think that it would only have value in people that are suffering from some sort of hypertensive condition to begin with. Because theoretically, if you're not hypertensive, then your whole.

[00:19:44] H one H two is all balanced and it's working good. And, and

[00:19:51] Dr. William Seeds, MD: [00:19:51] it goes further than that. So I think unique has a different perspective, but I, because what they're thinking about is this older person that we may think is, [00:20:00] has normal blood pressure, um, who, who may be healthy. Remember as we age, we're dealing against senescence.

[00:20:08] We have disease burden that's already there. So this reninangiotencin system is a real deal and is activated and is elevated as we get older. There's no knowing that. So I think he's looking at it or she, or they're looking at

[00:20:30] Carl Lanore: [00:20:30] Oh, I knew that was going to happen.  he's on a wireless network. He'll reconnect quickly. Here's what I'm going to do. I'm going to run a quick commercial break anyway, and we will get dr seeds back on and finish that comment. Stay tuned. Evolution just got kicked up a notch.

[00:20:51] Welcome back. Back to super Meridia. We lost you that doc, but it's okay. I think we've covered the AR BS and the possible. [00:21:00] Uh, benefits, uh, in, in, in this, uh, virus. I want to talk more about, uh, D, H H B, however, how it could be used, and then we're going to go onto cancer. How can it be used? How do you see people using it to add yet one more protective layer, let's say, for no, for lack of better terms?

[00:21:21] Dr. William Seeds, MD: [00:21:21] Well, as you know, I think it's an, it's a phenomenal, um, supplement and. Besides, it's the, the reason we brought it to the market for the, um, for the anoxia lytic or the, you know, the, or the anxiety issues that it is so capable of and it's so fantastic and, and helping people with it really has an, uh, uh, direct, uh, effect on that nuclear respiratory Kappa.

[00:21:52] That the, I'm sorry, that, um, nuclear factor, Kappa B transcription factor we were just talking about, it can inhibit [00:22:00] that, that mechanism. And that's a direct mechanism involved. Once the viral replication, um, and viruses take hold in the body, they can have a significant effect. And being a. A modulator of that, but at the same time, it can work on the interferon side and up-regulate that aspect of starving the cell of, of what it needs.

[00:22:23] So I, like right now on, every one of my patients has been directed to utilize this. I just, I don't have them using it at, uh, in higher doses presently. I just haven't taken one pill in the morning and one pill at night when they go to bed. And. My belief is that it can have significant effects in giving you a better equipped immune system in working against this viral load.

[00:22:52] And then if the load calm, let's say you get symptomatic, then we've got, we've got some room where we can add more of [00:23:00] this end, you know, taking two pills a day in morning. The day at night. I mean, we can really go after this and I like it because it's a, it's an option everyone can afford and it's an option that I don't think, you know, when we're working in an in an uncertain area at this time, but we know mechanisms, we understand the possibilities and we've got some science behind something.

[00:23:27] My attitude is, my gosh, why or

[00:23:30] Carl Lanore: [00:23:30] why not use it? Exactly. I feel the same way. Um, so let, let's talk a little bit about cancer. Now, the effects on cancer and a variety of cancers, including like some, some brain tumors, uh, and honokiol have been well understood. But as we said at the beginning of the show, it wasn't a practical, uh, therapeutic option because it had such a short, short half life and you had to give the patients so much, so frequently it was like, Oh, we can't do this.

[00:23:55] But now with DHH be in the picture, it seems that it could be a [00:24:00] potential therapeutic. What are we looking at. From the chemo protective side of this unique molecule.

[00:24:09] Dr. William Seeds, MD: [00:24:09] Well, I think this is where, you know, I'm really glad you wanted to spend some time to, to bring this up outside of, you know, why we have it, um, marketed as a antianxiety, uh, supplement.

[00:24:22] I think the power on this side of the cancer research is tremendous and. Let me, let me just go through some basics so everybody can understand and it may make sense. So when you have a tumor, you have a cell that becomes all of a sudden atonomous and it grows and it just wants to keep polyphony operating and dividing and self renewing and wants to just dominated takeover.

[00:24:53] Just think of it like that, right. And we've, uh, we've, uh, commonly we've approached cancer in a way of [00:25:00] where we just try to go at it and just kill it. You know, we use chemotherapy, we use radiation. Um, we use surgery. We just try to go at it and stop it. Well, what if we looked at cancer in another way? We know that cancer depends to grow and proliferate.

[00:25:21] It needs to continue to self renew, right? It needs to go through that cell cycle and continue the process. Well, what if we understanding that we know what senescence does in controlling a cell cycle? What if we use senescence against the cancer cell?

[00:25:40] Carl Lanore: [00:25:40] Wow.

[00:25:41] Dr. William Seeds, MD: [00:25:41] What if we turn off that cell cycle? We put it at hole, we put it at Bay.

[00:25:50] We'll guess what. This is where it gets amazing. Hinoki. Yael can arrest cancer cells in the cell cycle, so it can [00:26:00] become, basically, it could become a cell supplement that induces senescence, so it's going to stop that cancer cell from replicating. Number one. And number two, that it enhance it. It gives the settle that ability, so when a cell stops replicating, it gives a cell some time to take a look at what's going wrong and what cancer is very good at.

[00:26:28] It's good at using the cells own ability to make antioxidants and and so forth to, to keep it alive, basically, to, to keep it surviving. So if we can stop that cell cycle, let that cell try to clean itself up a bit, and then go through what's called a pop ptosis. Uh, where this, there's this, there's a specific tumor suppressor gene called P 53 that can be [00:27:00] activated, um, that will enable the cell to go through what's called a pop ptosis.

[00:27:07] It just, it basically kills itself. That I think is where you're seeing a tremendous amount of research now and looking at how we can go at cells, put cancer cells into senescence, stop them from replicating, and then do everything we can to attack what that cell has been able to do to take advantage of the of its environment.

[00:27:32] And that's exactly, so with brain tumors, like gliomas. What's your 70% of malignant tumors of the brain? Gliomas. What they do like a glioblastoma multiforme me, which is the worst type of cancer, one in the brain. It's, you know, five year, 5% survival rate. Um, we've shown specifically that with Hinoki UL in vitro and in vivo, in Xeno graph [00:28:00] studies in animals, that we can actually take that glial cell.

[00:28:04] That takes advantage of the STEM cell in the brain. That's how it keeps manipulating itself and growing. It utilizes the STEM cell, the glial STEM cell, and then transforms, well, Hinoki. Yael can go right at that and stop that, pull that cell replication by turning off the cell cycle, and then it gives also this cell, the ability to turn off.

[00:28:31] Um, it's a, it's mechanisms of what's called there. There are these mechanisms that are mitogenic mechanisms. It's, it's the mitogen activating protein kinases, and it's how  tours involved in this too, so it can turn all of that off. Then the cell loses its ability to where it needs energy, right to proliferate.

[00:28:55] And then it brings in this process of [00:29:00] upregulating what are called the box genes, which are Bax and BAK. They're the genes that cause a pop ptosis of a cell. And they deregulate these BCL two genes, which are the anti-apoptotic. So, so there's a real mechanism behind this. And Carl, it's been proven in glioma cell lines.

[00:29:24] So I think this is, this is,

[00:29:27] Carl Lanore: [00:29:27] this is way more exciting than antianxiety.

[00:29:30] Dr. William Seeds, MD: [00:29:30] Oh my gosh. Yeah. Oh, absolutely. Well,

[00:29:34] Carl Lanore: [00:29:34] I mean, I mean, don't get me wrong, for people who suffer from anxiety right now, they're going, F you call, what are you talking about? Because anxiety is a horrible thing, and we know, I mean, we have people that are using this, uh, this form of DHH.

[00:29:48] It gets called the chill pill. Uh, you know, I, I don't know, I guess I, uh, hold on a second. Let me just do this real quick. So, all right, it's called the chill pill. So those of you listening going, okay, but where do I get this stuff? Uh, you [00:30:00] can actually go to dr seeds.com use the coupon code SHR save 20% off.

[00:30:05] It's called the chill pill because originally it was used to help people who suffered from anxiety, social anxiety, and so on. Take the edge off without feeling sleepy or groggy or dopey like benzos and stuff like that. But the reality is that it may have a greater benefit to those who are worried about who feel they're genetically prone to, who filled their lifestyles are prone to cancers.

[00:30:31] It may be a more important supplement than that for them.

[00:30:36] Dr. William Seeds, MD: [00:30:36] Well. Yeah, it's so the re the research is hot and heavy right now and, and  and, and what it's done in, um, solid tumors, brain tumors. Um, hematologic. It's, uh, you know, specifically with this glioma, we were talking about gliomas, like, uh, uh, uh, uh, specifically this, uh, this, uh, tumor.

[00:31:00] [00:30:59] Uh, uh, what, uh, glioma, uh, Malta. Glioblastoma. Yeah. Sorry about that. Um, so the, the, the key to that tumor is this thing called epidermal growth factor. And that epidermal growth factor is what's enhancing the growth of the tumor, enhancing that neural cell, the glioma cell line enhancing that. That PI three, K, a, K, T, M, a P, K and poor pathway that leads to proliferation, differentiation, self renewal, all that stuff.

[00:31:36] When Oak Hill cuts it off, it deregulates that epidermal growth factor itself, it arrest the cell. It goes through all those mechanisms I described so. I mean, there's tremendous implications where we can go with this. And it's being, it's being actively looked at right now as agiment to, you know, to, uh, with, with other type of, uh, [00:32:00] um,

[00:32:00] Carl Lanore: [00:32:00] therapies,

[00:32:01] Dr. William Seeds, MD: [00:32:01] molecules, therapies, surgery,

[00:32:03] Carl Lanore: [00:32:03] and editing it into a standard of care type environment.

[00:32:07] Where they're already doing the things that they've always been doing, but the adding this to it, and they're looking to see if there's a higher recidivism rate and stuff like that. Question I have for you is that we, we know cancer leverages everything that's important and beneficial to healthy cells.

[00:32:22] Uh, but it just does it better, more efficiently and faster. Um, and this is one of the reasons why I've always been. Uh, more against than for the idea of suppressing IGF one to deal with cancer because the healthy cells need IGF one as well, and starving them of something they need, uh, is sacrificing yourself to starve the thing that you don't want any longer.

[00:32:48] Do these unique characteristics and mechanisms of action that we see with honokiol and DHH B. Uh, in cancer lines [00:33:00] affect healthy cells. All right.

[00:33:03] Dr. William Seeds, MD: [00:33:03] I'm not sure what your question is,

[00:33:05] Carl Lanore: [00:33:05] so, so does, does it, does it do this, does it shut off the cell's ability to repair itself and move on on healthy cells as well as cancer cells?

[00:33:16] Obviously we don't want it to do that to healthy cells, right?

[00:33:20] Dr. William Seeds, MD: [00:33:20] Yeah. No, it's, uh, it has played a trophic effects. It's a, it's an interesting. It's, it's how the cell utilizes the, the, the, the extract itself. Um, it does not influence the, um, it does not down-regulate itself from being able to repair itself.

[00:33:40] Carl Lanore: [00:33:40] And it doesn't speed up a pop ptosis in healthy cells, in other words. Right.

[00:33:44] Dr. William Seeds, MD: [00:33:44] No, it does not.

[00:33:45] Carl Lanore: [00:33:45] That's important. That's good.

[00:33:47] Dr. William Seeds, MD: [00:33:47] No, it does not do

[00:33:49] Carl Lanore: [00:33:49] that. Interesting. Interesting. So, um. What other therapeutic benefits do you see from this unique molecule so far?

[00:34:00] [00:34:00] Dr. William Seeds, MD: [00:34:00] Well, I think, uh, I think from just the perspective of just the multitude of, uh, of aspects of what it works with.

[00:34:10] And I mean, we've just talked about, we've talked about the potential of what, what it can do, um, in cancer and how it can relate to. Um, putting, arresting the cell cycle, um, which I think is, is invaluable, but on the same side. So this is where it gets interesting. And as you said, you know, can it have effects on regular cells?

[00:34:39] Well, if we look, if we look again at this process that you've heard me talk about, where as we age, we develop more senescent type of cells. That want to go into self cycle arrest. They don't want to proliferate or differentiate. They just want to stay in a swollen state and cause a [00:35:00] lot of problems with other cells, basically.

[00:35:02] Right? That's what leads to diabetes, heart disease. All the diseases we know about is all secondary to that process. Cell senescence. That's what's becoming evident today, right? Well, here's where Hinoki Eyal works. Also on the other side of the track where that process has to occur in, again, the key factor is this, NF, KB, this nuclear factor, Kappa B, a transcription factor that is over transcribed and starts processing this cell into where it gets its senescence associated phenotypical.

[00:35:43] Um, a state where it's making those cytokines and chemokine and it's what's creating, depending on where it is, um, where the senescent cells are, you know, if they're around the gut with Crohn's, inflammatory diseases, altered colitis, or if [00:36:00] they're around the heart with heart disease. Um, or, uh, there around the lungs or they're in the brain.

[00:36:07] I mean, it can be anywhere, right? Well, this has a direct modulating effect on the production of this. And FKB. I think that's tremendous. I mean, you know, we're all looking for these Sinhala Lytics and we're looking for the, I think we're, I think we have to be very careful because you, you really hit a great point about what does it do to a normal cell?

[00:36:32] Well, this  works specifically on cells that are dysregulated, so. So as the cells losing its efficiency and it is potentially becoming a senescent cell, we now have a modulator which is very cheap and goes and has more specific precision oriented that can go to [00:37:00] that cell and help modulate the amount of that that, uh, that FKB that's transcribed, that's making.

[00:37:10] All those inflammatory reagents and dampen it down and at the same time improve the cell's ability to go through a tophi G and my topic G and that's all our owls cleaning in the cell, right? So it gives the cell the ability to change what we would call an epigenic signature. Something how, how a cell, even though the genes dictate a certain way.

[00:37:39] It should transcribe certain proteins, and that's what you're born with. Well, we know that those genes can be influenced by many, many factors, and that's called epigenetic influence, and that will change what the cell is going to make. Well, Carl, this is an Irv supplement [00:38:00] that can I, that I, I have been, uh, immersed in.

[00:38:05] That I believe has a direct effect as a, an excellent Sunoco modulating type of supplement. And what I like about it, it's specific to bad cells and does nothing to normal cells. And that's where you get into issues, you know, like with the course of 10 and, uh,  and those things that, that have affects on every cell.

[00:38:29] And that's what I don't like about that. When, when people. Talk about utilizing them and doing them three times a. So, you know, three, three days out of three weeks and then you do it twice a year. I don't know how smart that is when we're going after every cell in the body, right? Because we need, we need senescence.

[00:38:49] We need cells like that to help us. Um, I, I'm more precision oriented. Let's just go right at it. If we know the mechanism. Why aren't we going

[00:38:58] Carl Lanore: [00:38:58] right? Try that. The mechanism [00:39:00] for that particular characteristic, I just, I knew I read this a couple months ago. I was doing some research on inflammation. It's always been a fascination of mine.

[00:39:08] And I found a study that linked, uh, inflammation to, uh, drug abuse and alcoholism. Uh, that actually, um, that, that the inflammatory effects of certain drugs of abuse as well as of alcohol increase. And this is why I was looking for it. NF Kappa B. And that brain inflammatory response is possibly the most damaging portion of some of these, uh, of alcoholism for obviously your liver too.

[00:39:37] And so it sounds to me like the chill pill. I'm not pandering here, but th this, this unique molecule molecule DHH B, uh, may have a role in helping people, uh, that have a Dick that have addictions and, and getting their brain straightened out and reducing inflammation in the brain because it's the study points at [00:40:00] the, uh, uh, reduction of NF Kappa B and the lack of the loss of desire for the drug or alcohol because of what it's doing to the brain.

[00:40:10] Dr. William Seeds, MD: [00:40:10] Yeah. And so, so here's, here's the unique thing also. So yeah, it's another, it's, it's the same, it's the same story in

[00:40:24] Carl Lanore: [00:40:24] a different outcome with a different reason for the outcome, right?

[00:40:27] Dr. William Seeds, MD: [00:40:27] Yeah. It's this same process, and it's what a senescent cell or an inflamed the pre senescent cell is becoming. It's, it's making, it starts.

[00:40:37] It makes that when, when it starts going through that transcription factor and making too much of it and where, cause sometimes it needs it to like attack viruses, attack bacteria. Uh, you need senescence for certain activities. But, but once it overcomes the cell in overuse, um, then it [00:41:00] becomes a problem.

[00:41:00] And the nice thing about this, uh, uh, DH, uh, about the, the Pinocchio. Is, it crosses the blood brain barrier too, and it has profound effects in the brain as we already know with what it does. Just with the GABA a receptor agonists aspect of, uh, helping, uh, call them the brain down. And it also works on the NSO for those people that have issues with addiction.

[00:41:29] They have some dysregulation of the, uh, MDA receptors and. This specifically. So it's all, it's about a little bit of glutamate toxicity and it ha it's over it that that MDA, MDA receptor is not functioning properly and there's too much calcium influx and this has specific mechanism of where it works and down-regulating that MDA receptor.

[00:41:57] So it really has some profound [00:42:00] mechanisms that, um, that we've known about requite a long time. But like you said, we haven't had the ability to use our knowledge of where we can keep something around long enough and have something that has a significant enough effect

[00:42:19] Carl Lanore: [00:42:19] to

[00:42:19] Dr. William Seeds, MD: [00:42:19] change those factors, those transcription factors.

[00:42:23] And. What happens down the road. And I think, I think the reason, the, the best evidence that I can give anybody about this HHB and, and its effectiveness is that direct anti-anxiety effect that people feel when they take a couple of those pills. I mean, it's, it's within 30 minutes or most people, they are going to feel that clean.

[00:42:49] Control, decrease of anxiety. And, and that's, I think that's, that gives you an idea that of the power of this supplement.

[00:42:58] Carl Lanore: [00:42:58] Any, any evidence that [00:43:00] it increases growth hormone levels, pulsatile nature, the trough, any of that. Because we know that, uh, some. GABA,  things do that, like, like the amino acid, the freeform amino acid, GABA, but also back in the day GBL and things like that.

[00:43:18] We know that they, they PR, they cause the body to produce them more like secreted dogs. Any evidence that the HHB is a secreted dog?

[00:43:25] Dr. William Seeds, MD: [00:43:25] No. Only in the aspect of if we, if you follow this, so let's go down this pathway again. If you believe in this process that as we age, we build up senescent cells. So let's say that process is going on in the hypothalamus or in the inter pituitary where we have, we have some dysfunctional inflammatory cells that are decreasing the ability of that anterior pituitary secrete a God to release growth hormone.

[00:44:00] [00:44:00] Or it's up-regulating somatostatin to hold on to that release. Okay. And, and we, we actually do see that, that there is that inflammatory process. We just don't know the mechanisms, but there it is. Again, it's that upregulation of an inflammatory process that may be controlling as we age. The release. Um, that hasn't been, of course, that hasn't been looked at.

[00:44:27] It's not a direct effect, but it sure makes sense if we follow this theory of, of how senescence affects. Um, all aspects,

[00:44:36] Carl Lanore: [00:44:36] all of this. Yeah, exactly. I look, we're going to take a less commercial break with dr seeds. We'll be right back. If you have any questions, put 'em up on Facebook.

[00:44:46] Dr. William Seeds, MD: [00:44:46] This is

[00:44:46] Carl Lanore: [00:44:46] the superhuman channel where we use oxygen for the power of good

[00:44:58] Goldston. The system [00:45:00] by computer has a coronavirus. All right. So if you want to explore and use D H HB, it's called the chill pill. It's at dr seeds.com you can save 20% off if we use the code SHR if you use the code SHR. And so, I mean, I think we've kind of covered all the benefits of the chill pill or do you think that we need to hit it more?

[00:45:23] Cause I want to talk more about your skiing expositions exposition.

[00:45:26] Dr. William Seeds, MD: [00:45:26] Ah, yeah, that I, I mean, w we can pick another day to talk about more on DHB. There's plenty to cover. So

[00:45:35] Carl Lanore: [00:45:35] Kevin Fryman wants to know what's the difference between the chill pill and other sources of DHB? I didn't know that. Where are the sources?

[00:45:41] Dr. William Seeds, MD: [00:45:41] Are there, uh, well, he'd have tiny, then they're pirated because there's only one, there's only one formula

[00:45:48] Carl Lanore: [00:45:48] and it's patented, right?

[00:45:50] Dr. William Seeds, MD: [00:45:50] Yes.

[00:45:51] Carl Lanore: [00:45:51] Yeah. So if somebody else has out there making DHH B. Uh, and they're not, and they're not on board with your group, then they're making it illegally.

[00:46:00] [00:46:00] Dr. William Seeds, MD: [00:46:00] They'll get a letter soon.

[00:46:02] Carl Lanore: [00:46:02] Yeah. Or just tell them to cut you in. That's all. Anyway, so a, there you go. So it's, it's patented, Kevin. So let us know. Could you post up here where you're getting it or where you think you've seen it? I'd love to know. So, so you are an avid skier. You love to ski when you're do have fine time away from your busy schedule.

[00:46:22] And I know that you love to ski with your sons. And of course, Jocelyn too. Uh, but, uh, you used to send me crazy pictures like when you, uh, upstate New York with your, when you, when Joss got you the new glasses that had the cameras built into them, where you would literally do this whole cross country ski thing where you would just go for hours and hours, but recently you climbed a mountain in Vale, Colorado.

[00:46:43] Is that right?

[00:46:45] Dr. William Seeds, MD: [00:46:45] Yeah, it's, it's, you know, it's all those things. I'm getting ready for my training, for my hustle felt stone deal that it's when I turned 60. But, uh, I, uh, my wife and my kids known that, uh, uh, I had this [00:47:00] goal of, uh, I wanted to, uh, skinny up Vail mountain, which is a little over two miles, up about 10, 10,000, 300 feet.

[00:47:09] And, uh, I was ready for it. I just had just done a mastermind course out there and my kids were off for spring break. And so I met them up in Vale. And, um, I told him one morning, I said that guys, this is the day I'm going to skinny up the mountain. And, uh, I, um, my wife had gotten me, you know, she's always the one who, who's always behind everything.

[00:47:33] And she had gotten my skins that I put on my skis and I've got, I've got a good set. Where I can, I can do a lot of back country stuff. But I, I, uh, prepared for, I started I think a little bit before six in the morning and it took me about two and a half hours to get up the top of the mountain. And what's so funny, cause

[00:47:56] Carl Lanore: [00:47:56] that's 10,000, 300 feet right.

[00:47:59] Dr. William Seeds, MD: [00:47:59] Yeah. Okay. [00:48:00] So, so let's, uh, what, what you'll appreciate, Carl, is, you know, I'm one of those people that I just put it in my mind and I'm going to do it right. So the first hundred yards of this track. I stopped and I'm like, breathing heavily.

[00:48:15] Carl Lanore: [00:48:15] Yeah. What did I get myself into? Right? I'm like,

[00:48:18] Dr. William Seeds, MD: [00:48:18] what the hell am I doing?

[00:48:21] I'm not, I'm, I'm, I'm, maybe I need to rethink this, you know, and, and I just had to tell myself, okay, this is where the brain starts messing with you. You were too. You know, you need to pace it. You need to be the turtle. You need to get your brain right. And do this because you can do it. And I'm telling you, Carl, I w I almost just said, what the hell am I doing?

[00:48:44] Because I was like dying after a hundred yards. So I just had to, you know, yeah. It's what everybody goes through. You had to get that mental preparation back. And, uh, I'll tell you how invigorating, you know, I, I was prepared. I had my, you [00:49:00] know, I, I'm, I think I'm optimized as much as I can be. And the. I had my ketone esters that I had, Ray and I sipped my esters about every hour as I, uh, you know, before I started.

[00:49:13] Carl Lanore: [00:49:13] Yeah. As you were saying,

[00:49:14] Dr. William Seeds, MD: [00:49:14] and I tell you what, it was exciting and I was, I was just, I was sending Johs back videos cause she wanted proof of life at re, you know, half an hour to make sure

[00:49:25] Carl Lanore: [00:49:25] we, yeah, sure. I mean, so, so let's, let's, let me ask you a question. Skins, they create traction. Is that why you put the skins on the skis?

[00:49:33] Dr. William Seeds, MD: [00:49:33] Yeah. They're there so that you can, you can hold your ski one, you're on an incline and you're not

[00:49:40] Carl Lanore: [00:49:40] like using backwards. Okay, good, good. But then I got to believe that once you got to the top, you took the skins off and you skied down, correct. Yes. That has, that had to be a gratifying ski down. Right.

[00:49:52] Dr. William Seeds, MD: [00:49:52] It was awesome.

[00:49:53] So I got, I got to the top just as the mountain started opening, so [00:50:00] I was really excited to. You know, to be able to, to kind of cruise down the mountain. And I actually, I, I, I really appreciate it more skiing down that mountain of what I just, what I had just done. And, and what's really funny is I know the mountain really well and, and I, as I was coming up, I thought, I still had, like, I thought I still had half, half the trip done.

[00:50:26] I thought I was going to be up there for four hours, five hours. And I'm looking up the Hill and I'm thinking, how am I going to do this? How am I going to keep going? And I saw this big tree and I'm like, boy, that tree looks familiar. And as I got closer to it. I'm like, Oh my God, that's the tree. If I go around the corner, I'm going to see the gondola as it comes to the top.

[00:50:45] And there it was. And I was like, Oh my God, I'm, I'm all right. I've made it. So that tree became my favorite tree on the mountain.

[00:50:53] Carl Lanore: [00:50:53] I bet. Because it was like an old friend giving you, uh, giving you a hand up the Hill though. Okay. You, your perception of where you are changed [00:51:00] immediately once you turn that around, that tree.

[00:51:03] Dr. William Seeds, MD: [00:51:03] I think I constantly was trying to talk myself out of it the whole way.

[00:51:08] Carl Lanore: [00:51:08] So let me ask you a question. I've been doing that, by the way, pushing the sled, like, because I haven't pushed a sled and well over three years, and I have the first couple of reps, but you know, I call them reps paths back and forth.

[00:51:18] I go on. That's it. I think that's my last set. And then I sit and I breathe and I recover and I go, I'm going to do one more set and next to, you know, you do success. And I, it's the mind. The mind doesn't really want discomfort. It doesn't, that's the bottom line. So tell me something, they took you two and a half hours to literally ski up that Hill and no skiing.

[00:51:40] Really climbed that Hill. Awkward, awkward shoes on. Uh, how long did it take you to get to the bottom once you skied down?

[00:51:48] Dr. William Seeds, MD: [00:51:48] Five minutes

[00:51:49] Carl Lanore: [00:51:49] seriously, right.

[00:51:53] Dr. William Seeds, MD: [00:51:53] It was about five minutes. I was in less than five minutes.

[00:51:57] Carl Lanore: [00:51:57] That's so cool. That's so cool. You rock on. [00:52:00] Look, we have to, this is a great question. I want to answer this before I let you go. So Kevin Fryman, who asked about buying DHH from somebody else said, somebody told me it's the same thing as Magnolia Barkin that's why I want to talk.

[00:52:10] I'm going to reiterate this again cause Kevin May not have heard the beginning of the show. But Hinoki all, which is also Magnolia. All I think they call it, uh, is actually from Magnolia bark from the tree. But the problem with it is that it has a stupid half, like it clears your body in like minutes. So it's not an effective choice for any of the things that it has the ability to do.

[00:52:36] It wasn't until the creation of this molecule, which is di hydro honokiol. That it actually sticks around in the body for what doc, like six hours

[00:52:48] Dr. William Seeds, MD: [00:52:48] or six hours.

[00:52:49] Carl Lanore: [00:52:49] So, so it's four to six hours and it also has a greater potency. It wasn't until the, the development of this molecule that Hinoki all was even viable.

[00:53:00] [00:53:00] For exploring for any of its purported benefits, because you would've had it in, put people on infusions of it around the clock to get their blood levels high enough and to keep its presence in their bloodstream long enough to see anything. So no, Kevin, this is not Magnolia bark. This is a, is a different form of what honokiol is a, it's a, it's a more advanced form.

[00:53:22] It's a, it's a, it's a better product entirely to hope that answers your question. Yeah, no, no. Go, go. That's

[00:53:30] Dr. William Seeds, MD: [00:53:30] all right. I was just going to either two parts of it, and this is the Hinoki will aspect. There's the Magnolia and whole no-kill. It's part of that, but it's specifically Pinocchio that's been, um, that, that has been cycle eyes.

[00:53:45] Then it's, it's like 30 times the strength of Pinocchio. So that's why it's called by hydrogen OPO and it's, it's, it's a patented, uh, supplement.

[00:53:53] Carl Lanore: [00:53:53] Yeah. There you go. You're not gonna find that anywhere else at this point in time, unless a. Your group starts to [00:54:00] sell it as a raw material, which I don't see them doing anytime soon, so I think it's safe to say that hun.

[00:54:05] Okey, DH HB di Heiko Huna di hydro Hinoki UL or and or the chill pill is a worthy supplement for you to consider. You will save 20% off if you go to dr seeds.com and use the code SHR if you want to give it a try. I know people who are on auto ship, they believe in it. You know, Dylan Gautreaux, he did a commercial for us who was not remunerated for it.

[00:54:29] The guy has been on benzos since he's eight years old, and he wanted off benzos. He's a grownup now, and he uses these to cope with his lifelong anxiety. He says that's the only thing he's ever tried, and he's tried it all. That literally works as, as as promoted. So.

[00:54:47] Dr. William Seeds, MD: [00:54:47] That's really a special, and I thank you for sharing that.

[00:54:50] Carl Lanore: [00:54:50] All right, look, thank you so much. So what are you going to do with all this free time on your hands, doc?

[00:54:56] Dr. William Seeds, MD: [00:54:56] Um, well, I'm still in the office, uh, seeing [00:55:00] patients right now, so that, that, that's, that's we're, we're overwhelmed with that. Um. But I see that maybe a possible shutdown on that too. Maybe for a few weeks we'll see what happens.

[00:55:12] But I think we'll always kind of be in some form, we'll be open because of not the, the ERs not wanting to be overwhelmed with traumatic injuries, you know, sprains and strains. Those things can keep coming to the office and we can, you know, we can keep dealing with that. Um. These are uncertain times, and I, you know, I just, I'm out there to support everyone and let's do our best as a, as a team to get this under control and move on with our lives.

[00:55:39] Right?

[00:55:39] Carl Lanore: [00:55:39] You know, our parents' generation, they're, their metal was built on things like this. They, they, they suffered and they held their chin up with pride and they didn't complain about it. That generation never complained. I have people telling me. That they're going to go [00:56:00] crazy. I they can't do this. And I say to some of them, when you have to go to work, you probably wish you could stay home, right?

[00:56:06] Yeah. Yeah. Now that you don't have to go to work, you're complaining about staying home. Unfortunately, and I say this and I include myself because I am, nowhere is tough as my father was. My father never complained about anything, and he had more reasons to complain than most people. But we have turned out to be a real bunch of pussies and we need to just like suck it up and shut up.

[00:56:31] If you have to not go to the gym for 15 days, you know, work in at home, do body weight work, do per general preparedness, go for a sprint, do something else. But don't complain about it because at least we're not like Italy, at least when not like some of these other countries that are literally overwhelmed with dead bodies right

[00:56:48] Dr. William Seeds, MD: [00:56:48] now.

[00:56:49] Yeah.

[00:56:49] Carl Lanore: [00:56:49] So that's it. That's my rant.

[00:56:54] Dr. William Seeds, MD: [00:56:54] All right. Thank you for having me, Carl.

[00:56:56] Carl Lanore: [00:56:56] Take care. See you soon. We're going to take a quick commercial break and when we come back, I'm going to tell you [00:57:00] an interesting story about why I think Elisa and I exposed to Corona virus in November. I'm not kidding about this, and those of you who are avid listeners of the show, you will know what I'm talking about.

[00:57:14] Evolution just got kicked up a notch

[00:57:21] fucker, man. So there's kind of like a stream of consciousness. Maybe.

[00:57:26] Dr. William Seeds, MD: [00:57:26] You know what, let me take that,

[00:57:27] Carl Lanore: [00:57:27] since dr seeds isn't here, let me try something, a new element of the show. Let me just go to no overlay. There we go. That's gone. I so I'm sure a lot of you tuned out already cause everybody came here to here with dr seeds had to say.

[00:57:43] But, uh, I've had some interesting thoughts. It started this morning. With the observation that the first case of a covert 19 and Kentucky happened in a tiny, tiny little town, a population of [00:58:00] under 18,000, uh, called Cynthiana, Kentucky, and it was a girl who worked at. The Walmart, the local Walmart, and given that it's such a small town, you know, we're not talking about a big city Walmart, right?

[00:58:13] We're not talking about tens of thousands of people walking through the Walmart every single day. That may be every couple of weeks or every month maybe. And Elisa was telling me that the officials there baffled and how she could have, you know, get it. How could she get it? You know? No one she knows has been to China or abroad.

[00:58:32] She hasn't been to China or abroad. No one she knows has the virus. In fact, I was told by an executive, I'm not going to mention his name cause he messaged me this, but, uh, of all the employees that work at Walmart, she's the only person that has caught covert 19. So maybe it's protective to work at Walmart.

[00:58:55] But, um, I digress. So. [00:59:00] Elisa said, the officials, they're just baffled. Like how could, like, they're almost making it sound like, Oh, see, um, maybe it's in, in us already and it's just coming out. No, it's not. No, it came from . There's no doubt about that. We know the epicenter, even though the, the Chinese government covered it up until December.

[00:59:24] Late December, uh, it was on their radar. Now we now have evidence, uh, the CDC has said that it was probably in their population by November in order to have created such a big spike to be observed in December. Now, hold onto that thought for the last part of my discussion. So. I said, I don't understand why they're baffled.

[00:59:54] We also know from both the CDC and other agencies that are tracking this virus close closely, that there's a [01:00:00] very large percentage of people who are exposed to the virus, but they don't get any symptoms. And this is actually stratified by age, right? You know, people on the 40 have a good possibility of not even having any symptoms, like not even having a fever, not even having a sore throat, nothing, nothing that tells them they may be sick.

[01:00:23] You remember that old game, seven degrees of Kevin bacon. You could start with any person and find somebody they know, who knows someone they know, who knows someone, they know, who know someone they know in seven steps. Who knows Kevin bacon? Oh, you know this person, that person Debbie, and she was his CoStar on some movie, so.

[01:00:45] If you think about that connectivity in our population, I mean, it's very, very accurate. Quite frankly, we're probably seven degrees away from anybody if we really work hard enough to figure it out. Um, the probability of what [01:01:00] happened to that girl in Cynthiana. Is she works at a Walmart and, okay, so she doesn't see tens of thousands of people a day, but she definitely comes in contact with hundreds of people a day.

[01:01:12] That's more than most of us. And there's a high probability or possibility that someone she came in contact with who didn't have symptoms, came in contact with someone else who didn't have symptoms, who came in contact with someone else who didn't have symptoms. Who came in contact with someone else?

[01:01:32] Who did you see what I'm getting at? It's the same thing as seven degrees of Kevin bacon. Uh, everybody can track back and may have come in contact with someone who didn't have symptoms, who got it from someone else, and someone else that someone else in all those people didn't have symptoms. So nobody, so the connectivity has dropped, like, do you know anybody who has.

[01:01:55] Coronavirus nobody. I know. We'll ask all the people you know anyway, no, they [01:02:00] don't know anybody. They ask all the them. You may end up having to go seven steps away until out here in this periphery of thousands of people. Someone goes, yeah, the guy at my school, somebody sent me a picture the other day of them escorting someone off of an airplane.

[01:02:14] I think in Connecticut, they kept the car, the plane on the tarmac, and they said she had tested positive for Corona, how she got on the plane, we don't know. And all those people, and you know, in the plane we're exposed to it. You know, air is recirculated in a, in a jet. I make jokes about this all the time.

[01:02:33] I say, you know, the lady who fought in the back, her fart is blowing on your face through that little vent that you point at yourself to keep yourself from overheating. So. Air is recirculated. If somebody has coronavirus on a jet, there's a good chance that you have been exposed to it. So the girl in Cynthiana isn't baffling to me.

[01:02:52] She came in contact with someone who didn't have symptoms, so they weren't even considered. But that doesn't mean if they tested [01:03:00] everybody that they wouldn't test positive, which we're seeing in Korea now. South Korea has regularly dropped their mortality rate every time they get the results back for more tests.

[01:03:12] 96% of the people that they've tested that had respiratory symptoms did not have coronavirus. They had something else. Uh, and of the people that they're testing, they're seeing a lot now. It's estimated from the CDC and other agencies that up to 75% of people who have been exposed to this virus don't get symptoms.

[01:03:34] Think about that. That is actually scary because while this virus isn't particularly virulent. It is absolutely more a contagious than anything we've ever seen. So it's going to take out a large number of people just by fact that the volume of people that will be exposed to it, unlike influenza. So I'm not downplaying this.

[01:03:57] Please don't get me wrong, this, [01:04:00] I'm, I'm staying away from people. I'm not going to the gym. I'm training at home now. I'm not going to be stupid. I don't want to get sick to prove that I can survive. But it's very clear to me how the girl in Cynthiana got it. She came in contact with someone who was asymptomatic, so no one even thought about asking them any questions.

[01:04:21] But here's something else. Those of you who've been listening to the show regularly know that Elisa and I went on a cruise. In late October and I got so sick in November, as you remember. I mean, I continued to do the show, but I had to keep killing my mic. I was coughing. I had this dry cough, I was running a fever.

[01:04:41] I felt terrible. And I jokingly said to all of you, don't spend money on a cruise. Just go to the local hospitals, emergency room and lick the floor. The results will be the same. And those of you paid attention to the shows, though. I told you I knew where I got it. We will on a hop on hop off bus tour [01:05:00] and I, we just jumped on and sat down and did a tiny little bus.

[01:05:04] You know, everybody's crunched together. The lady sitting to my left, the leases to the right, the lady sitting to my left is packing into her hand into her hand at this dry, raspy hacking, alarming cough. Like I literally looked at Elisa when we first sat down and I'm like, Oh my God. Why did we sit here?

[01:05:24] There were no other seats, but then shortly thereafter I looked at Lisa and I said, I can feel her breath hitting me in the face. We have to get off, and we literally ran, rang, rang the bell, and we didn't even know where the next stop was. We just got up, move, move past her and got off, but it was already too late because that was at the beginning of the trip.

[01:05:46] The day before we were supposed to disembark and get off in New York. I already had a sore throat and felt horrible in the night that we spent the first night in New York. I was running a fever. I couldn't sleep that night, and we also [01:06:00] know from those who are studying this. That the mean, the average incubation period is 5.7 days, but the longest so far is about 12 days.

[01:06:12] Well, that's, I fit right into the 5.7 day model of developing my symptoms. By the time we got home, I was shot. I was wreck and I was sick for a month. It did not want to let go. It did not want to go away. I had shortness of breath. Those of you paid attention to the show, know that I had a. Mute my mic all the time and cough.

[01:06:30] Uh, I was running a fever constantly. I felt horrible. Uh, lethargic, very, very weak. Well, Elisa got sick about a week after we got home and she had the same exact symptoms, fever, uh, difficulty breathing, dry hacking, cough, and it, she, she, we talked about it this morning. She said it lasted her a little bit over a month.

[01:06:52] Now. I'm not saying that I had the Corona virus, but maybe I did. And here's my logic. [01:07:00] So we know now from the CDC that China hid the truth of when they noticed this. They didn't, they didn't talk about it until the end of December. By February, we had put in travel bands in place to keep those who in China or from China or flying home from China out of the country.

[01:07:18] And if, if, if they knew they had a, a potential pandemic cause that they waited to the last minute to tell anybody, and that was in December. Well that means that in November it was already starting to show up on their radar. And if it was showing up on their radar in November, then that means people will already starting to pass it around in late October and late October is when we were on the canard cruise that went from Quebec North and then back South down the East coast.

[01:07:49] And we sat with people for dinner every single night for lunch, for, for, for, you know, buffet and so on. Everybody was hacking on that. So much so that they were [01:08:00] making people sanitize their hands when they were walking in. They, you couldn't just walk in, they had the people standing up, please. And you'd have to pump and squirt your hand before going in because they already saw the number of people on that, uh, uh, a cruise ship that was sick already.

[01:08:15] That we're hacking that will obviously and very, very old group of people. I hope all of them live through it, but we knew that there was a problem already. They let us know, Oh yeah. You know, there's a, there's something going around the crews, you know, upper respiratory clique. Keep cleaning your hands. We probably were exposed to it because a lot of those people, like we sat with one family.

[01:08:37] Of six that were on a month and a half long cruise that went from Asia to the Mediterranean and then then to the UK, Ireland, and then down to the United States. And they still had a cruise back home, which I think they were started out in the UK or someplace like that. So we would definitely exposed [01:09:00] to something from that region of the world on that trip.

[01:09:04] And we got sick. But here's the other thing that I want to point out to you. There's a conspiracy theory out there that the Chinese government wants to get rid of its old people. So they started this disease, and now they're also suggesting that other countries are leveraging it by trying to get rid of their old people.

[01:09:23] And they're also saying that this is targeting the unproductive people in the in, in society, hence the poor. But now we know that this is actually a virus that is opportunistically affecting the affluent. When you look at the epicenters in the United States, they're not the homeless areas. They're not the homeless shelters.

[01:09:50] They're not incarcerated people. They're not in the poor communities. They're in the affluent communities. Because somebody. Had to [01:10:00] travel abroad and bring it home. And travel is something that affluent people do. Westchester and new Rochelle. Those are two of the wealthiest areas in New York, believe it or not.

[01:10:11] Hell, George Soros lives in Westchester. So those people travel, they go abroad, they go to Italy, they go to Spain, they go to England, they, they, they go to Asia, they go to China, they go to Japan, they travel. That nursing home in Washington state is a very, very expensive nursing home to stay in, number one.

[01:10:34] And they already know that the one patient who got sick got sick from somebody who had come back from China. So this is a virus that seems to be impacting the most affluent people in our population today, not the poor. Not the destitute at all because homeless people are going to China. Nobody's in the homeless community in San Diego going, Hey man, I just got back from being homeless in China.

[01:11:00] [01:11:00] No one in the homeless communities getting this because there's no affluence affecting them. So when you hear a lot of these conspiracy theories that this is a method of getting rid of the poor people in our society. It backfired if that's what they thought they were going to do. Number one, cause it's actually getting rid of the affluent people in our society.

[01:11:22] Uh, and when you also think about how this is transmitted, it is transmitted unknowingly. Often I predict, and that's going to come out in years. After all this shakes out and we do all the research you're going to hear. Some ridiculous number of people who are asymptomatic were probably the reason for these obscure cases popping up in South Dakota somewhere where like, how did it get here?

[01:11:49] No one we know went to China, but someone they knew knew someone who knew someone who knew someone, and none of them was symptomatic until that person became symptomatic. And that's how it got [01:12:00] there. So that's all I want to produce, uh, in that dish. And for you and. It's a, it's, it is, it is worrisome. I am not playing it down.

[01:12:09] I am taking precautions. So is Elisa, and we're happy to do it. You know, we love our home. We love our backyard. We love being around each other. So this is nothing much has changed for me, except I won't be training at the gym. I'll be training at home. Uh, but that's it for today. Thank you for listening. I don't think we have a show tomorrow.

[01:12:28] Uh, but of course we have, I think we have a pep talk on Friday. So tune in for that and we'll see everybody later. Have a great day. [01:13:00] .