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Transcript to SHR # 2522 :: The Epigenetic Effects of Tobacco and Alcohol PLUS Shielding Your Body From RF Soup PLUS Busting the 5G = COVID-19 Myth

[00:00:00] Carl Lanore: [00:00:00] Welcome back to another episode of super human radio. We have a really good show today. And it's good on a lot of different levels. And I'll tell you why, because there's a lot of us who lead what we consider clean lives, uh, but many of us do certain things that maybe really aren't in line with the desired outcomes that we want in our lives.

[00:00:18] One of those things is alcohol, and another one of those things is tobacco use. Uh, whether you're chewing tobacco or smoking tobacco, uh, these are the two most abused substances, substances in our population. They both have addictive qualities. They both rewire the brain to desire more. The big difference between addiction and, uh, and, and, uh, what's the other word I'm looking for?

[00:00:39] Um, but anyway, uh, addiction has specific. Uh, signatures. When you look at brain and how it changes, and we're going to talk about that from a, uh, an aspect of epigenetics. What is it doing for us? There's a lot of people that read studies and it says, Oh, you know, a glass of wine a day is good for you. I personally don't believe that if it's good for [00:01:00] you, give it to your babies and give it to your dogs, right?

[00:01:02] Why? Why hold them out and not let them benefit from the good things from alcohol. But we're going to talk about what it really does to the body. Uh, and, and whether or not it is. Uh, something that you should be partaking in, in just a minute with Dr. William seeds and Dr. Daniel Elias, Martine, herons. And before we do that, we have to, of course, pay homage to our title sponsor, and that is legendary foods makers of amazing snacks, uh, that you can eat and feel like you're cheating, but you're not.

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[00:02:16] And uh, Oh, this is, this is what it's about. When you produce your own show as you're hosting your own show. And, uh, we have, are joined by both Dr. William seeds, which many of you already know. And, uh, Dr. Daniel LIS, Martine, herons, all the way from the UK. How are you guys doing? Okay,

[00:02:41] Dr. Daniel Elias Martin Herranz, PhD: [00:02:41] well thank you. Here we go again.

[00:02:43] Carl Lanore: [00:02:43] I got worried there. I was going to, Oh no, we lost the microphone. Um, so I think we could all agree that there is a large push, not by the tobacco industry so much. But definitely by the alcohol industry to somehow [00:03:00] normalize alcohol use by saying it's actually good for you. When we look at epigenetic testing, uh, do we see evidence, Daniel, that, uh, that alcohol is good for you?

[00:03:17] So

[00:03:19] Dr. Daniel Elias Martin Herranz, PhD: [00:03:19] low levels of alcohol, um. Have been, as you said, for a long time, you know, promoted as something that is healthy for the general population and according to the latest studies, um, and more specifically, a study, for example, that was done in 2018 in the landslide, which is one of the most prestigious. Uh, medicine magazines or journals, uh, where they look at, you know, data from 195 countries, uh, females, males.

[00:03:50] Uh, what they show is that there is no amount of alcohol that is good for your body, right? So I think, you know, obviously, um, you know, [00:04:00] science on 90 put immunology keeps changing. And, you know, as we have more data, we discover new things. But I would say, you know. From a scientific point of view, while we do know until today is that no level of alcohol is as good for our bodies.

[00:04:15] Carl Lanore: [00:04:15] And then you know that that just validates something I've been saying on the show for over a decade. I like getting a buzz from alcohol. I like the way it makes me feel when I'm out with friends. It lubricates everything. It makes everything easier and fun and. I sound so much more intelligent to myself when I'm drunk.

[00:04:32] Uh, but you know, the reality is that I've never believed that was good for me, and I just say that's an, that's an accepted risk that I'm a risk I'm willing to accept. But the reality is, I've always said that I don't think alcohol is good, but tobacco is another story. I think it's pretty obvious that tobacco is bad.

[00:04:51] And I think when we think about tobacco products, we do think that they're bad. Yet people start smoking today. [00:05:00] Right now there's people who are going to start using tobacco products, smoking and so on. Dr seeds, what happens when someone is exposed to tobacco products on an epigenetic level?

[00:05:14] Dr. William Seeds, MD: [00:05:14] So the. The process is really the same as what we do.

[00:05:21] And when we're looking at, uh, at any disease, it's, it's a change of a method, a methylation change that happens. Um, on the cytosine. It's a, it's a methylation that happens and, uh, it changes the signature of that gene. Uh, and it's, we've shown that even low. Exposure, uh, to, uh, smoking versus high exposure still shows epigenetic changes of methylation.

[00:05:52] Uh, sites and they've, they've identified up to, I'm pretty sure it's been up to over a thousand [00:06:00] sites. I think that's right. Danny, close to a thousand sites that they have recognized where there are true methylation changes. Um, and in particular, uh, the, uh, alcohol, uh, hydrocarbon, uh, um, gene repressor is one specific that's followed all the way through that is affected with.

[00:06:21] Uh, smoking and it's, it's, uh, they've even shown that smoking affects that methylation changes in a smoke, in a pregnant woman smoking have a genetic or epigenetic changes in a child. So that, that's, uh, those are significant, I think, significant factors of, of showing that there are real changes that happen in what that gene is able to do after it's affected by that.

[00:06:48] By that change. Even more so with the epigenetic changing, uh, or, uh, studies we've been able to show, you know, that you can reverse, depending on [00:07:00] timeframe, you can reverse some of these changes where you can get them back to what a non-smokers, uh, epigenic profile would look like

[00:07:08] Carl Lanore: [00:07:08] and that sort of thing.

[00:07:09] But something, there has to be an intervention to do that. If not, of course, I noticed that some of the studies I looked at indicate that just cessation. A will in the host will, will change things. But what about the transgenerational effects that we have? We have a bullet item up right now that talks about that there's this trans-generational effect going upstream to a future, uh, children and grandchildren.

[00:07:37] If you stop smoking. Before you get pregnant or before you father a child, because we know it's in the germline of the father as well. Daniel, can we save our future generations or are they stuck with it because of our stupid decision?

[00:07:52] Dr. Daniel Elias Martin Herranz, PhD: [00:07:52] Yeah, so obviously, uh, this is, this is quite a difficult topic on, you know, there are some things that we do know already and many things that [00:08:00] are unknown, uh, still in, you know, in humans in us.

[00:08:04] Um, one of the things that we do know is that if the mother smokes while she's pregnant, uh, from, from the future baby, uh, and that is considered on intergenerational effects, or not transgenerational but intergenerational, um, then we can detect. Uh, some of that signature has stopped their seats said, uh, in that, you know, baby and then, and then adult even, you know, decades after, after that happened.

[00:08:31] So that is truly, if you think about it as scary, um, in the sense that we know that it is possible that somehow those people there, that the health of those people are being affected. They catch up towards because their mothers smoked in the. You know, while while they were pregnant. So obviously we don't truly understand yet what are the big consequences for the health of those people.

[00:08:56] We do know that those signatures exist. Um, and [00:09:00] I think, you know, in the next few years, we will start to understand what are the consequences of this much better.

[00:09:05] Carl Lanore: [00:09:05] You know, it's funny, my mother, my mother smoked when she was pregnant with me, and she also took one of the earliest benzodiazepines Librium.

[00:09:15] When she was pregnant with me and my sister used to joke with me when we were older because I always had allergies. I had respiratory problems when I was a kid, and my sister used to laugh and say, mommy smoked when she was pregnant with you. And like, like it was like she was putting the hex on me. But you know, I'm listening to this and I actually feel sad.

[00:09:34] I feel like, Oh my God. So the scales were already tilted the day I entered the world because my mother smoked while she was pregnant with me, and no doctor ever told her not to. Yeah, I

[00:09:45] Dr. Daniel Elias Martin Herranz, PhD: [00:09:45] mean, obviously we do need to make, uh, you know, I think now everyone is much more conscious, died, you know, you should never smoke to start with.

[00:09:54] Uh, but if you do a smoke and you're going to be a mother or a father, you should quit, uh, [00:10:00] months, if not years before you're thinking about having a baby. And, you know, I think that's, that's a recommendation that everyone should follow, is one of the best investments that you can do for your health and that of your children.

[00:10:11] Uh, and I think there's, there's no doubt that as we understand better, they're making a sense of how this happens. Uh, you know, it'd be become, it will become more and more clear how, you know, mutations and also epigenetic changes, uh, cost these things. But, um, yeah, I think everyone should try to avoid that as much as they can.

[00:10:31] Carl Lanore: [00:10:31] I'm going to start a go fund me page for me. No, I'm kidding around.

[00:10:37] Dr. William Seeds, MD: [00:10:37] I think Danny made a really good point that people should. Should really pay attention to, and that is the female that is interested in getting pregnant and not having these factor, these methylation factors affecting the, the baby's, uh, genomic profile.

[00:10:56] We know that if you're a heavy smoker and you've been smoking over, [00:11:00] you know, five to seven years, it can take over two years, two years to get a profile that can be. Normal for smoking. So that means, like Danny said, it can be years that you have to be prepared to understand what you're going to do in, in potentially changing your offspring.

[00:11:21] And I think that, you know, those kinds of messages need to be made really clear, so people understand there are very, there are very significant consequences. Uh, with, with trying to, um, when you're, it's not just, you stopped smoking in, in six months, you know, things are all better and heavy smokers. It's a big deal.

[00:11:42] And it, that's a long time to get that normal pattern back, that normal a methylation pattern.

[00:11:48] Carl Lanore: [00:11:48] And so, and every, every time we talk about methalate, uh, epigenetics, we talk about methylation. So it seems to me. That everyone's in agreement that while senescent cells [00:12:00] are the watermark of bad aging, it seems like the real magic and aging better is establishing the appropriate levels of methylation and D methylation.

[00:12:13] Is that correct?

[00:12:16] Dr. Daniel Elias Martin Herranz, PhD: [00:12:16] So, yeah, it's part of the, it's part of the story. Definitely. And we are unraveling this as we, as we speak. So, you know, this is a really, really hot topic in, in biological research, on aging research at the moment, trying to really understand what are the main, you know, molecular mechanisms behind the aging process and what we can do to, to reverse that, or at least.

[00:12:36] Slow it down. And as you mentioned, we know that we accumulate senescent cells as we age in different tissues. And this senescent cells are like some sort of Somby cells that secrete a lot of inflammatory molecules, uh, and that, you know, reduces that, that way or like it makes the tissues not work so efficiently anymore.

[00:12:55] Um, and I know that also things that we've seen is that there this epigenetic changes. [00:13:00] Uh, I'm more specifically at type of opportunity market. I assume it's known as DNA methylation. Uh, that also happens to be an agent and, and also across tissues. Right. Um, so now the question is, uh, are this, uh, methylation changes causing aging, or are they a consequence of all the things that are happening during an agent?

[00:13:20] Uh, and I think probably the answer is going to be that it's a combination of both. Um, so I do think that there will be, you know, part of the, of the biggest story, uh, of what is really driving agent will, will be at the epigenetic level. But I do think also that many of the markers that we use might be a more downstream consequences of the courageous process.

[00:13:40] And I say most of the emphasis in biology, the, you know, the answer is normally called complex.

[00:13:47] Carl Lanore: [00:13:47] And that's what makes us all sad because people are very, very monolithic. We want to know, just tell me the one thing I need to do, and it's never one thing. And that's actually an insult. Uh, [00:14:00] into the reality of the complexity of the human being.

[00:14:04] I mean, it's just, it's, you know, we have to start looking at ourselves the way we look at computer systems as opposed to a laptop, you know, uh, there, there are so many systems is, uh, thank you. Thank you, Scott. I was actually picking up my laptop by my phone because Tommy said he had no audio. And he is having a good audio.

[00:14:23] Thank you. Thank you. I was distracted. Sorry about that. So, you know, we are so much more complex and, and sadly even the pharmaceutical agenda tries to, to distill things down to this one pill, that one thing. And you know, I'm sorry to tell you if that is all your bandwidth can handle your, your loss.

[00:14:43] Because this is about addressing systems. It's not one thing. Like, I have some problems I'm dealing with. And I realize now it's not one thing. It's not just going to be my diet that has to change. There's a lot of things that are going to have to change. And so it's a really good point. So tell me, there's a [00:15:00] new word out there now that I see in some studies.

[00:15:03] Poly epigenetic DNA methylation scores. What is poly epigenetic.

[00:15:10] Dr. Daniel Elias Martin Herranz, PhD: [00:15:10] Yeah. So obviously, uh, the word comes from polygenic risk scores. So when we look at not epigenetic, but genetic information, uh, what we realize over the years is that looking only at one position in the DNA to try to explain a phenotype or your risk of disease, et cetera, is many times not enough.

[00:15:29] Uh, because most of these phenotypes or most of this risks are quite complex. So you need to use thousands and thousands. Uh, of, of different positions in your DNA to build biomarkers that are really accurate. Um, so a poli epigenetic risk score would be the same, but looking at epigenetic data. So instead of using only, you know, one or two positions of one persistent of the, of the DNA where we're looking at the methylation pattern.

[00:15:55] We look at thousands of them or millions of them, and we try to build [00:16:00] a biomarker that is much more accurate using, uh, the information from all those markers. And that's, that's really what we do,

[00:16:07] Carl Lanore: [00:16:07] what it makes. I was going to say, dr seeds, one of the things that you will always excited about the chronometer test, and I felt like this was a good opportunity to, to run the, uh, special to, uh, because my audience is the only audience in the world that can get 70% off.

[00:16:23] The single best epigenetic test in the world, and it's a saliva test, which means you don't have to go to a doctor. No one has to draw blood. You just spit in the tube. I just did it the other day and sent mine in. I can't wait to see it because I really feel it's going to give me a direction on how to approach.

[00:16:38] Getting feeling better again, but dr seeds, one of the things that you said to me that you were so excited was that this particular test tests 20 million data points in your DNA as opposed to the nearest, uh, closest match of a epigenetic test is only like what, 2000 or 80,000 or [00:17:00] something like that.

[00:17:02] Dr. William Seeds, MD: [00:17:02] Um, uh, maybe up to 400,000, maybe more.

[00:17:06] Carl Lanore: [00:17:06] This is 20. This is 20 million. So when you talk about poly epigenetics, which I see this word being used more and more in epigenetic research, it's like, Oh, God, you know, we've been looking at this one target when we have all of those targets out there that are, that are hurting us.

[00:17:23] Dr. William Seeds, MD: [00:17:23] Yeah,

[00:17:24] Dr. Daniel Elias Martin Herranz, PhD: [00:17:24] that's right. Yeah.

[00:17:25] The more markers, the better.

[00:17:27] Carl Lanore: [00:17:27] Yeah.

[00:17:27] Dr. William Seeds, MD: [00:17:27] I mean, any, any statistician will tell you that it's all statistics. The, the, the higher number of variables you have to evaluate, the more accurate and the more predictability, um, you can have in your testing. And that's, that's really the most, I think that a very important point to bring up here.

[00:17:48] It's. It's predictability and how can you translate this information? And that's where we're going with all of this information, is being able to [00:18:00] predict and translate. Where does this progress into disease? You know, where do you stratify into any disease process? Uh, just like we're talking about right now about smoking, you know, what is the with are, what are the markers to push you into.

[00:18:18] Cancer, esophageal cancer or lung cancer or bladder cancer, or what are the markers that are going to push you more towards the chronic obstructive pulmonary disease, and that that information is tremendous. In fact, we have information from looking at epigenetic changes that

[00:18:38] Carl Lanore: [00:18:38] there's, Oh, okay, we may have lost dr feeds.

[00:18:44] Dr. William Seeds, MD: [00:18:44] Probably nothing to do with smoking. What's that?

[00:18:48] Carl Lanore: [00:18:48] No, no, you're back. You're fine. Yeah. That's a great point. Some of these things may have nothing to do with your smoking at all. And,

[00:18:54] Dr. William Seeds, MD: [00:18:54] and that's, that's crucial information did to start building. And, and [00:19:00] this is where I think the, you know, all the work that, uh, Danny and his team are doing are just, uh, it's just remarkable to see how this is going to advance and change the way we, we look at Madison because this, these are the things we need.

[00:19:14] To help our patients make better choices and to convince them that you can make better choices and that they potentially can be reversible. And that's what's, that's what's so I think that's the point. No one should, should devalue about the epigenome that it is reversible. And. That's why this is, this is why we're so focused on it.

[00:19:38] Carl Lanore: [00:19:38] This is such a great discussion because that's one of the things that people need to understand. The beauty of the epigenetic model is you cause the problem. You can fix the problem, but you first have to get your compass. Pointing in the right direction. And the only way to find that out is the compass has to have seen the bad direction.

[00:19:56] And that's what establishes the role of the epigenetic tests. [00:20:00] When you see where you went wrong, you can then go right. But if you're just shooting in the dark and trying this and trying that, or I'll try the carnivore diet for awhile. I don't know what's working. The carnival diet may work for you, but there may be other things that you need to add into it.

[00:20:14] To see the results you want. I love this. I'm so excited because I really believe that getting a picture of my current epigenetic status, my digital twin. Is going to allow me to get on the right path. And that could be a myriad of things I could find out that I've been trained in way too hard for too long.

[00:20:35] And that's part of the reason I feel the way I do. And I'm willing to acknowledge that and, and move on that, uh, dr seeds, I have to just mention John peaks, just comment that he has a. A physical for work coming up and nothing quells the white coat syndrome, like the chill pill. You just reminded him he needs to order some.

[00:20:55] So I thought that was kind of cute. Um, there, there is, [00:21:00] um, there's really is really a great opportunity here, a hopefulness. I kind of think that this is like optimistic medicine where other medicine is pessimistic. You go in, they say, Oh, you got cancer. Oh, but here we go. Oh, you have to change paths. And it's, it's really exciting to me, so I've got to

[00:21:20] Dr. Daniel Elias Martin Herranz, PhD: [00:21:20] fully agree with that.

[00:21:21] With that, Carl, I think, you know, in the DNA testing a space, I think their previous, you know, only looking at genetics sometimes can be a bit of a pessimistic medicine in that sense. Um. So sometimes, you know, in the case, for example, of, of genetic variance that allow you to choose to choose treatments, etc.

[00:21:39] There you can, you know, you do have a choice on, and it's very useful. But in other cases, it will just tell you that you have this risk, uh, and that there is little that you can do to change that because you were born that way. And I think, you know, some people might want to know, but in general, I think everyone wants to.

[00:21:58] Take information that [00:22:00] you can do something about right. And, and that, and that is the positive side of things. And that's where, you know, every genetics is, it's so powerful in that sense, in first of all, being able to measure all the stages over time. Uh, compare against yourself, against your baseline and build, as you said, your digital twin.

[00:22:16] And then take a specific interventions on half a page to see if they are working or not. Uh, you know, over, over time. And I think that completely changes. The way that we think about preventative healthcare, the way we do medicine and the way that we avoid all this

[00:22:33] Carl Lanore: [00:22:33] complex diseases. And then this, this tier bullet I just put up, speaks to something that dr seed said that there's longitudinal data that shows that the methylation changes accumulate.

[00:22:44] With increased use of tobacco and you can't, that while they can attenuate, you can, by quitting, you can attenuate them. The longer you smoke, the longer you'll have to quit before they go back to normal because there's this, this accumulation, you know, we forget about accumulation. [00:23:00] I'm a big fan of acknowledging that part of the reason things happen as we age is because of the accumulation.

[00:23:07] Of things that aren't good for us. That could be iron. Uh, it could be toxins. It could be, uh, the, the components of tobacco use that are, but they accumulate over time. It's like junk. You can't just stop and expect all that junk to just disappear. You gotta wait for it to come out of your body.

[00:23:29] Dr. Daniel Elias Martin Herranz, PhD: [00:23:29] That's right. Yeah. And I think, you know, oldest societies that are super interesting in the sense that they have shown us the complexity of, of this dynamics, right. And before, we didn't really have a way to quantify this in the longterm, uh, because the main problem with. Previous biomarkers to, uh, you know, that look at, for example, a tobacco exposure.

[00:23:50] They normally look at things like cotton in, in the body, which is some metabolite that you can quantify. But the problem is that the half life of that is max a couple of weeks, right? So [00:24:00] after one month, approximately, you don't have a way to know how that person is recovering. For example, from quitting the smoking.

[00:24:06] And that is where epigenetics become super powerful, right? If you're a former smoker, you decided to quit. You want to see how you're improving not only during the first month, but over the next month, over the next year, and you know, during the rest of your lifetime really and see how you're improving because that's a very positive message.

[00:24:23] If you have a way to really see your improvement, uh, you know, there are tons of studies that show that from our behavioral or psychological point of view, that's so powerful for people to, to stick to those interventions. And you know, this, this type of femininity biomarkers really give you the possibility for the first time to quantify these things in the longterm, rather than just, uh, during the, during the first month

[00:24:46] Carl Lanore: [00:24:46] dr seeds, it's like people who train with weights.

[00:24:50] And they're not sure that the program they're on is going to give them the results they want. So they hop from program to program without ever seeing any results. And [00:25:00] had they stuck with one program, they would have seen it. But the problem is the evidence isn't always. Fast enough for us to say, Oh, I'm accomplishing my goal.

[00:25:09] And it's even more evident when you're looking for health. You, you, how do you quantify your health? So maybe you get a bad night's sleep, you think, Oh, it's, I'm getting worse. I got to stop doing what I did yesterday. This gives you an opportunity to truly quantify, Oh, I just got to stay with the program because things are working.

[00:25:28] Dr. William Seeds, MD: [00:25:28] Yeah. And I think that's a, it's a really important points and bring up, when I'm sitting down with a patient. And I go over lab values with patients. You might as well just stop there because you, they have no idea. Or they're having a difficult time and following how you evaluate those lab values and, and, and how that correlates to them.

[00:25:53] Whereas you have a graph, you have a picture, you've got a picture in time, and you can relate it and [00:26:00] stratify it to other people around the world. That's pretty powerful. And that's what people take home, that snapshot and go, Holy smokes. Okay, this, this is, this is something that is valid. And, um, and I'm being compared to this tremendous bank of data, uh, and I can see it and I can see where I need to go.

[00:26:24] This is fantastic. And, and that's, that's, that's such a valuable tool in trying to change. You know, you can't, I can't, it really doesn't matter what I say. Um, it matters how I say it and how I get the message across to a patient where they're the ones who determined, you know, how committed they want to be, the change and the more valuable I can make that message, the better I'm doing my job.

[00:26:53] And that's where this is. So this is, uh, this has changed the way you have a conversation with patients. Never had [00:27:00] anything this powerful before in, in having discussions of where your patient gets it instantly, it's not, it's not an, it's not one of those things that takes a while to, to look at something and then to digest it.

[00:27:14] They get it right away.

[00:27:15] Carl Lanore: [00:27:15] So do you think there's something magical to, uh, to the heat factor and tobacco, or do we see some of these same problems with methylation with people vaping. Uh, instead of, you know, they're, they're still putting certain, certain chemicals in their lungs that are getting distributed to the body.

[00:27:32] Is this a, is this a problem with nicotine? Is it a problem with the toxic, uh, particulate matter that's going down? And I love, what do you think, Daniel?

[00:27:42] Dr. Daniel Elias Martin Herranz, PhD: [00:27:42] Yeah, no, that's signing probably interesting question. And I think one of the, one of the areas where, you know, a lot of research is going on now, uh, not only from the opportunity side of things, but, you know, in general.

[00:27:53] Obviously there are more and more people that are starting to vape instead of smoking. Uh, I'm, you know, I think it certainly, [00:28:00] they should really know what are the longterm consequences of Fabian. Right. Uh, but there are a few studies that are starting to look at this, for example, from the epigenetic side of things.

[00:28:08] Um, in one of them, they looked at a specific type of, uh, you know, part of, of our DNA, which is known as line one. And these are repeats. So regions of the DNA that are the same. And, and they occupy a great proportion of our entire genome, uh, and they actually come from, from mains and viruses that inserted long time ago.

[00:28:28] And then, you know, they just stay there. So they've been associated with many things, you know, that have to do with our predisposition to develop cancer. Um, and things like that because they can be unstable sometimes. And what keeps them on their control. It's actually DNA methylation. So, you know, in general, more DNA methylation in those regions.

[00:28:49] Keep them, you know, without, without causing too much trouble. And, uh, what these people in this study, so, uh, looking at both vapers and smokers, [00:29:00] uh, what they saw is that there was hypomethylation. So I lose of methylation in this line. One elements, uh, which could be a proxy for increasing your chances of developing cancer for both.

[00:29:10] Um, so having said this, this was a relatively smaller study, uh, and you know, it's early days. Um, but I wouldn't be surprised if it turns out that, you know, some of the damaging effects that we see in our epigenomes in our genome, uh, caused by smoking, are also observed in vaping. Uh, but I'm also sure that there will be different.

[00:29:31] Ways by which they can affect us.

[00:29:33] Dr. William Seeds, MD: [00:29:33] And I think is that Danny, where, so when that hypo methylation in that region occurs, it changes the promoter region where the methylation as aspects occur. And so that changes from that methylation. Even though there's hypo methylation, the promoter region changes its methylation.

[00:29:53] Is that

[00:29:53] Dr. Daniel Elias Martin Herranz, PhD: [00:29:53] correct? So line ones, uh, can be all over the, all over the genome. So they are, [00:30:00] you know, a specific type of sequence that can be in many places. Uh, I'm not a hundred percent sure to be honest with you. Like whether they are, uh, they will be founding promoters or I'm assuming there might be some cases where that's the case.

[00:30:14] Um, but I think in this case, the main problem is that if we don't keep them under control. Uh, they themselves can then cause, you know, making stability. Sure. Um, so it's a different, yeah, it's a different mechanism by which they can become a dangerous team in our genomes.

[00:30:30] Carl Lanore: [00:30:30] Right. We're going to take a quick commercial break.

[00:30:32] We have a lot of questions and comments piling up here. We're going to get to all of them. When we come back from this break, we're going to take a quick commercial break. When we come back, we're going to talk about alcohol consumption because this is something that all of us seem to think well. You know, it's okay.

[00:30:46] I read a study. It's okay. It's good for me. Uh, but it really isn't. And we're going to discuss the effects on the epigenome, uh, from alcohol and get to your questions as well. So stay tuned. We'll be right back with more. [00:31:00] Superhuman radio. It you'll gain with it. You are listening to the superhuman channel where ripped and we're ready.

[00:31:13] Welcome back to super gonorrhea. We got a lot of comments, some questions I want to put up before we move on to alcohol. Um, so Rigo Vargas, who is actually been on my show in the past. Has a really good question. He says, I'm hesitant to get genetic testing because I'm afraid to have my genetic information on a database somewhere.

[00:31:33] Am I paranoid? Change my mind.

[00:31:38] Dr. Daniel Elias Martin Herranz, PhD: [00:31:38] Yeah. Thanks. Thanks, Rico for the question. I think, I think you're not paranoid. I think you're asking the right question. Um, obviously genetic information and epigenetic information is very sensitive data, and you should be very careful where, where that data is being stored on, who's taking care of it.

[00:31:53] Um, I think I kind of speak from comics, you know, very proudly in the sense that we have embraced, [00:32:00] uh, type of legislation, uh, which is the European law for this, which is known as CVPR. Uh, so we are a European company. And what that means is that, uh, you know, we have the highest standards of data privacy and security in the world.

[00:32:14] And, you know, I said, company, we've always believed that. Um, you know, people own their data where just like, you know, a story on it carefully. I'm using it to give you the best that we can with it. Uh, and in that sense, you know, our users are always in control of it. Uh, so first of all, in terms of security, uh, you know, the highest standards for this in terms of encryption, both in, uh, at rest and in transit, et cetera, et cetera, are taking into account, uh, on that in terms of privacy.

[00:32:43] The data is only used for those things that, that our users want it to be used. Uh, and they have complete control, uh, on this all the time. So at any time, if you contact us and say, Hey, I want you guys to give me my data and delete everything that you have on me, uh, you can [00:33:00] do it at any time. Uh, and you know, we love, you know, this philosophy where, uh, where the users really empower, uh, to, to be in complete control of the data that is being stored in a secure way.

[00:33:12] So. Uh, I would say in that sense, we go, uh, you can trust us. But, uh, obviously, you know, I understand that it's a very sensitive type of data and, uh, that you should, you should do. Why you feel like it's better for you. But

[00:33:27] Carl Lanore: [00:33:27] I think about a doctor, you spoke about this before, you actually spoke about it in terms of what we have here in the United States, right?

[00:33:35] Dr. William Seeds, MD: [00:33:35] Yeah, exactly. I think that needs to be emphasized how their rules are stricter. And that that's very important, that the, this data is very much your data and you control it. And this, these are the strictest rules in the world, uh, on this type of data. And this is what they're following. And I think that really resonates to [00:34:00] patients and, and people when they understand that those are questions you need to ask.

[00:34:04] That should be one of the first questions you ask is where, where is this data going? What control do I have over it? And are you utilizing this data outside of your institution with other, you know, are you collaborating and selling my data? And, uh, those are, those are really, really big points to bring up.

[00:34:22] And I think that's what makes this, um, for me so valuable to know because I'm always looking out for the best interest of my patients. And. That's why we're talking about this.

[00:34:33] Carl Lanore: [00:34:33] Well, one of the things that I think has come to the forefront now, aside from just what the company does with the data, is what, what happens when the company is sold 23 and me was sold to a pharmaceutical company, if I'm correct.

[00:34:44] And a lot of people feel like, Oh great, now this pharmaceutical company has all of my information about my health and, and, and. What an, even though that pharmaceutical company may not do anything sinister with it, in fact, they may learn from it and think, Hey, you know, we've ignored this, uh, [00:35:00] these symptoms.

[00:35:00] And when the bottom line is people feel like, Oh my God, you have my genetic fingerprint. Now what happens when you sell your company? Do that. What if they don't abide by the standards and rules that you've established when you are running your company?

[00:35:17] Dr. Daniel Elias Martin Herranz, PhD: [00:35:17] That's right. So, uh, the way that it works with DDPR with, uh, you know, with their legal basis under which we operate, and as I said, we embrace, is that, you know, because their user is in complete control of the consent that is given to that company, if that consent were to be changed at any point.

[00:35:35] They will need to opt in for that. So what that means is that, um, you know, in the case of 23andme, what happens is that GSK, which is a large pharma company, licensed access to that database, uh, to, to develop tracks, right? And because their 23andme users have consent for research purposes, uh, that, that data can be used, obviously not only my Spiner, uh, with, uh, you know, any [00:36:00] research related purpose, then that was possible.

[00:36:03] Now what happens with us is that the consent that we collect from our users is that that data at the moment can only be used. Uh, to provide with our services and products and keep improving, uh, the stuff that we, that we give to you, right? So what that means is that if we wanted to do anything else, we would need to go explicitly and ask you.

[00:36:23] And that also applies, uh, in, in this type of cases. So, uh, what that means is that your, your data is being protected under the, you know, most minimum. Contract that you can hop in terms of sharing it so it cannot be shared with third parties. And then if we, for example, one to establish any research project with a university because we want to use your anonymized data to, to understand something better, most of the people will say yes, but it's all about, you know, you being in control and you deciding.

[00:36:50] While your data is being used for it, so you

[00:36:52] Carl Lanore: [00:36:52] can opt out. I liked that. So you would have to go back and say, Hey, we're selling our company and we're selling our data. Do you want the new [00:37:00] company to have your data? And you either opt in or opt out. So that's at least that's right. That allows you to control the destiny of your data.

[00:37:08] And also correct me if I'm wrong, but data is not saved. With the person's personal information. The data is just raw data with a number attached to it, and then in another database, that number is attached to the person. So people can look at the data in a very anonymous format without knowing, Oh, Carl Lenore has this gene.

[00:37:31] Dr. Daniel Elias Martin Herranz, PhD: [00:37:31] That is correct. Yeah. So we completely isolate your molecular data, your epigenetic data, or genetic data from the rest of your personal information. So in our systems, they are in two complete separate places. So it's impossible. To be able to much both of them, uh, unless you have, you know, uh, that, that, that you'll to make key if you wish.

[00:37:52] Um, so what that means is that, uh, you know, for all these purposes that have to do with research, et cetera, et cetera, uh, [00:38:00] if you were to consent often, uh, to share your data in an anonymized way, all the other party will see, let's say you're a researcher in the university of Cambridge would be raw data.

[00:38:09] Uh, but they do, they wouldn't have. Your name, they wouldn't have, you know, your postcode. They wouldn't have anything like that. So they wouldn't be able directly to go and say, Oh, this is the data from this person. So that's the way that, you know, oldest studies, uh, that involve, uh, human data are performed nowadays.

[00:38:26] And I think. You know, I think most of us, if we feel secure and we feel that our day job know, and I know any mice way is being used correctly, I think most of us will volunteer for that day.

[00:38:37] Carl Lanore: [00:38:37] Absolutely. Absolutely. You know, and I was just thinking of that. And then what about when you die, once you die, does the data, is it then available to whoever or does your right to protect your data transfer to your family?

[00:38:52] Dr. Daniel Elias Martin Herranz, PhD: [00:38:52] That's a good question. I actually don't fully know the answer to that. Um, if I'm completely honest with you. Um, I [00:39:00] think it would probably vary between countries, um, as well, uh, because obviously everything that has to do with inheritance, et cetera, et cetera, who has the right to decide on, on different aspects after someone dies, changes between countries.

[00:39:15] So I'm pretty sure. Legislation at that level will apply. Uh, but yeah, definitely it's, it's a very interesting question that

[00:39:22] Carl Lanore: [00:39:22] in my opinion, mean more and more

[00:39:23] Dr. Daniel Elias Martin Herranz, PhD: [00:39:23] important.

[00:39:23] Carl Lanore: [00:39:23] My opinion, the data becomes more valuable once you die and we know what you died from because now we also have an end point to match up your epigenetic journey and go, Oh, look at that.

[00:39:33] That's what he died from. So that is valuable information. Scott Lawler says. Uh, he's talking about the training program. I'm not about finding the best program. It's about finding a good program that works for you. I agree with you. A thousand percent. And Victor John, Andrew Mifsud, who has been on my show and doc knows, uh, said that he's waiting for his chronometer test to come back because he's excited to see the results, uh, specifically as it looks at his jeans and his, uh, he has a condition.

[00:39:58] Um, [00:40:00] I R D I think it's, uh, yeah, retinitis pigmentosa. And he wants to see if there's anything that he can do, obviously, uh, to respond to that. Um, okay. So we're going to move on now to alcohol because most people who drink smoke and most people who smoke drink and they do them both at the same time. Uh, which is really funny, but, uh, the reality is we, when we start looking at the whole methylation, uh, phenomenon, alcohol looks like it's even worse for you than tobacco.

[00:40:32] Am I right about that? Duck? Doc seeds. Yeah.

[00:40:36] Dr. William Seeds, MD: [00:40:36] Yes. So, so actually there's a, I mean, I think they're both bad. I think there's, you can, we can, we can relate that to different aspects of what, of, of how alcohol and smoking can affect you. Um, I think one of the interesting things is that we're just learning more about the metabolites of alcohol that actually [00:41:00] do have direct.

[00:41:01] Effect on, um, uh, is specifically acetone, um, where an acetol groups, um, affect the histones of DNA. And there's these post-translational changes that, that actually have a profound effect in the brain changing the epigenetic signature in the brain. And that's, uh. That kind of information I think is, is monumental now in, in moving forward of really validating that this is a, this is a serious toxin you're putting in your body.

[00:41:34] When you can validate and show right away very quickly that you're changing. You're changing the epigenome of neurons in your brain. And, and the fact that they're, they're showing, I think it's the AC, I can't remember if it's ACSS two or something. Uh, um, Danny, that it's, that, that gene, that enzyme that is important in.

[00:41:58] In changing and [00:42:00] how you develop those behavioral changes of wanting to drink more or, or so forth. So, so we're finding out so much more about that. Um, that when, when I saw that those papers that were starting to come out about the brain, I was like, okay, you know, besides everything else we know this is, this is seriously significant.

[00:42:20] And in being able to validate it epigenetically.

[00:42:24] Dr. Daniel Elias Martin Herranz, PhD: [00:42:24] That's right. Yeah. And I think it's super, you know, it's very interesting, the fact that alcohol, which is one of the most consumed things in the world, uh, is one of the least really understood drugs at the mechanistic level. Uh, so, you know, it's very, it's very interesting with all this new status, as you mentioned, dr seeds, for example, that have created very strong mechanistic links, biological links, uh, with directly the metabolites that are produced.

[00:42:50] Um, after you drink alcohol together with, uh, you know, how your genes might be regulated even in the brain. Um, so you know, these direct connections [00:43:00] really tell us that probably the story behind uncle Don, how it affects our behaviors and our health is quite a complex one. And also, uh, something that we should definitely need to, you know, we need to understand more, uh, because it's so widespread.

[00:43:14] Um, having said that, we do know. That, you know, we can use epigenetic information as I'm sure we'll discuss in a second, to quantify a how, you know, the way that you're drinking alcohol is it's affecting your health and how that is changing over time.

[00:43:29] Carl Lanore: [00:43:29] Do you think there are people out there who, um, seem to metabolize and deal with alcohol better than others?

[00:43:37] Are there some people that can get away with it and some that can't? You think Danny.

[00:43:42] Dr. Daniel Elias Martin Herranz, PhD: [00:43:42] Yeah. No, that's absolutely the case. I mean, we do see that already, uh, in our, in our data set. Um, so, you know, people can drink exactly the same amount of Fargo and yet have different consequences. And the reason for that is because, uh, there are many factors that are going to affect.

[00:43:59] The [00:44:00] response or your body to, to alcohol, right? There will be things that have to do with your core biology. So your genetic background, your sex, your age, uh, you know, your overall lifestyle. Uh, and there will be things that have to do with your behavior towards alcohol drinking, right? So there are people that, uh, will drink the same dose by in different ways.

[00:44:21] So some people might spread it across different days. I never engage in binge drinking while other people might just drink everything. One Saturday night. Uh, and obviously, you know, the consequences of that are also, uh, very different. And that's why it's so important to start to develop these biomarkers and have these biomarkers that at the end quantified the end point right now, what the way that it affected your biology at the end of the day, because all of these variables, you know, if we only look at self reported data, it's very difficult to really tell what happened, right?

[00:44:51] Because we don't have someone monitoring in real time. Okay, I'm drinking this amount today, this amount tomorrow. When you ask someone how much do you drink? They will tell [00:45:00] you, I don't know, five pints of beer a week or you know, but it's very difficult. And there are tons of studies that show tons of biases in the way that people self perceive.

[00:45:09] Carl Lanore: [00:45:09] Well, we know, we know that people under-report what they perceive as bad behavior and over report what they perceive as good behavior. So if somebody says, that's right, I just drink a couple of days a week, I know they're drinking five or six days a week. If they say, I just have one drink, I know I'm having five or six, I mean, because.

[00:45:26] They know it's something that they shouldn't be doing and they don't want to say, Oh, I'm doing this and I don't care what you think. They care what other people think. So they under-report they always do, especially when it comes to alcohol.

[00:45:38] Dr. Daniel Elias Martin Herranz, PhD: [00:45:38] Yeah. And I think that's why it's so important because, and again, I can speak from, you know, in this case, in my case, uh, so the first time that I took the test, um, I was drinking not so much about that.

[00:45:50] The national recommended guidelines, which in the UK surround. 14 units of alcohol per week, which I think is equivalent to something like maximum five fines or five beers per [00:46:00] week, or something like that. Uh, but the problem is that I was doing it mostly on the weekend. Right? And then if you do that during one or two days, then most of the time that's unhealthy.

[00:46:10] Right? So when I actually look at my exposure at the epigenetic level. It wasn't incredibly high, but it was getting close to the red. Right. So I was really shocked when I sold this because I, you know, I never really thought of, I never felt that that was really affecting me. And since then, I actually decided to, uh, not so much we use the amounts, but try to spread it across different days.

[00:46:34] Right. Uh, and I also tried to reduce it a bit. Uh, and I, in one year I managed to, you know, dramatically see the change. So I was really happy to, to see in my, in my own body how I was able to get back to the place where I should be. And I think the important thing here is that, uh, you know, what we're doing is a preclinical measurements.

[00:46:56] So we are detecting a stop that happens years [00:47:00] before you will develop proper liver damage. For example. So the current biomarkers that we use to, to look at this, things like, I mean, the transfer is the same blood, et cetera. When you have that high, you already have liver damage, and by then, yes, you might be able to recover, but it's already too late.

[00:47:14] You already got proper damage, right? Well, the stuff that we're measuring happens before that. So we're able to, to see your trend on where you're heading, um, before you get that damage. So not, and that's why this, these biomarkers are, uh, so important.

[00:47:31] Dr. William Seeds, MD: [00:47:31] That. That's a really big point that he made. Carl and Danny, thank you for bringing that up because that is, that's the most valuable tool you can bring to your patient is when you're, you're getting your preemptive and you're giving them data.

[00:47:46] Before they would even see anything that we traditionally have had, and I think you bring up a great point that I really like, is that typical. I shouldn't say typical, but doc, I only spend the weekends where, you know, I work hard all [00:48:00] week, I work out all week, and then on the weekends this is what I do.

[00:48:03] And I still say, okay, that doesn't work. And you just, you just gave a great example of, um, and, and I'm going to give you a lot of credit for doing that as a young man to be able to come to that conclusion. Cause it's, it's very difficult to convince. Um, and, and Carl will appreciate this with our, with our, the people who really do train hard, they do great things with their bodies.

[00:48:30] And then in my opinion, they go out and destroy it in one or two days and then they want to go back and they think that they're controlling it and they're not.

[00:48:40] Carl Lanore: [00:48:40] I could completely live without alcohol. I've done it for long. I mean, I went when I got married and had children, I probably didn't drink for.

[00:48:48] You know, 18 years. I mean, I had the occasional glass of wine here and there, but, and once I found physical culture, it was more important me to make gains in the gym than it was for me to have a drink on a Friday night. In fact, that [00:49:00] caused a lot of friction between me and my ex wife. And I wouldn't drink wine anymore on the weekend.

[00:49:04] But, you know, it's really, it's really sad because so many people are willing to justify alcohol use and turn to, um, studies. That are basically, uh, designed to help them, you know, bias, bias, studies that help them mitigate the guilt of doing something bad. You know, in the simplest terms, alcohol is a neurotoxin.

[00:49:32] Mercury is a neurotoxin. I'm sure that if you took small enough amounts of mercury, you'd feel high. It would probably affect your head where you feel like woozy and stuff like that, but nobody's going to start out going out and taking little bits of mercury because they go, Oh no, that's mercury poisoning.

[00:49:46] Well, it's alcohol poisoning. That's what makes you high. Alcohol poisoning makes you high. That's what it is. I mean, it's just really sad.

[00:49:56] Dr. Daniel Elias Martin Herranz, PhD: [00:49:56] I think that thing they important points will be made here, has died, you know? [00:50:00] No amount of follicle is good. And you know, the latest studies that we have, that's what they say.

[00:50:05] Um, you know, if someday that changes, then, you know, I will be very happy to update my knowledge on that. And, uh, but to this day, uh, I think that's clear. And, you know, the reason why other studies in the past have found that maybe low doses are good is because they're, you know, their tone stuff, confounding factors that can affect that.

[00:50:25] Um, you know. There are surviving viruses, all sorts of like epidemiological viruses or in many of the studies. Um, and you know, it's very, it's very, very difficult to, to, you know, at those very low doses, really have the power to, to detect these changes. Right. Um, and you know, there's also the aspect of, uh, some types of drinks, having components on them that might be healthy, such as, for example, resveratrol.

[00:50:52] In red wine, while the alcohol itself is the bad thing. So what you want to do is to have the word spiritual without, without

[00:50:58] Carl Lanore: [00:50:58] the alcohol. Right?

[00:51:00] [00:51:00] Dr. Daniel Elias Martin Herranz, PhD: [00:51:00] So, you know, that's, that's the sort of stuff that, uh, you know, I think, you know, this, this study's diet. You know, they might be true that have fun. For example, as seasons between red wine.

[00:51:11] And, uh, you know, good health outcomes, they run, you know, it's almost impossible that they will be because of the alcohol. It will likely be because of the rest of the compounds that are in red wine that are doing the good thing. Right. Um, so at the end of the day, I think the key take home messages, no amount of alcohol is good.

[00:51:29] Having said that, I'm also Spanish.

[00:51:32] Carl Lanore: [00:51:32] I know I'm Italian. I love, I love, I love,

[00:51:36] Dr. Daniel Elias Martin Herranz, PhD: [00:51:36] I love red wine. And I think, you know, like anything in life, we also need to be happy. So. Uh, from my more, I guess, philosophical point of view. I know, I think everything in moderation is fine.

[00:51:49] Carl Lanore: [00:51:49] I, and a doc will tell you, I love Sambuca.

[00:51:51] It makes me cry when I have a couple of glasses of Sambuca. I get very emotional. I love everybody. Everything is great. Uh, you know, and I, I like red wine. It's okay. But [00:52:00] I, I rather have a Negroni any day. I like Kampala. I like little compari and some gin. And. Everything is, everything is smooth. We're going to take a last commercial break.

[00:52:09] We'll answer some more of these questions and we'll wrap up the show. Again, I'm going to run this one more time for people to see if you want to see things that your doctor will never be able to see. If you want to help your doctor be a better doctor. If you go to seeds, that MD epigenetic dash test, uh, I'm sorry, seeds, that MD slash epigenetic hyphen test slash you'll be offered.

[00:52:35] A thousand dollar epigenetic test for the low price of around $200 I think it is. It's 70% off, and I know people from the show who've bought them, not just for themselves, but their wife and their children because they're going to give their children a wonderful gift. And this is something that your doctor can do for you.

[00:52:56] He checked your blood pressure. He does these rudimentary blood tests. He puts you on [00:53:00] a statin drug or something. This is, this is archaic medicine. This is the new medicine. Actually knowing. What's hurting you and what's helping you. You can now find that out with a good test like this that tests 20 million different points of your DNA.

[00:53:17] The more data, the more data points, the more information. We're going to take a quick commercial break and we'll be right back with more, so stay around. We'll finish up with some more alcohol stuff. I think here.

[00:53:30] You were listening to the superhuman channel. Don't hate us because we feel good.

[00:53:38] Welcome back. We're talking about epigenetic testing. We're talking about things that affect epigenetics. We're talking about two things that a lot of people in the United States and abroad do. I actually, I think smoking is down. In the United States compared to places like Europe and Asia. Uh, but you know, T tobacco use, alcohol use, they're, you know, they're, they're things that [00:54:00] everybody feels comfortable doing, but they are very, very damaging.

[00:54:02] And, you know, in, in light of this whole Corona virus thing, it's disingenuous for people to say, Oh, we're saving lives by shutting down the country. Um, a hundred thousand people a month die from tobacco use. I don't have the statistics on alcohol, but think about that for a second. A hundred thousand people a month die.

[00:54:26] From cigarette smoking and tobacco use. That could even be chewing tobacco and dip and all these other disgusting ways to get your caffeine or your nicotine. That was a Freudian slip because caffeine is a problem too. But when you think about it, we're worried about, you know, Oh, we've got 38,000 deaths in the United States.

[00:54:43] That's a third of what dies every single month from tobacco use. How can we say we're doing the right thing? We're worried about people. We want to help people. When the reality is a hundred thousand people a month die from tobacco use and we don't care. And [00:55:00] you know why the government gets their money?

[00:55:02] It's called, it's called taxes. They make taxes on tobacco use. So. If we really care about people, we have to address what tobacco is doing to the population. And I know I'm not asking you guys to comment on that because you're a scientist and you know, I'm just, I'm just trying to put things in perspective here a little bit.

[00:55:20] Um, so let's, let's wrap this discussion up a little bit. So in summary, these two bad actors, alcohol and tobacco work the same way. They disrupt the methylation. Landscape, and they affect our health. Is that pretty much the bottom line?

[00:55:39] Dr. Daniel Elias Martin Herranz, PhD: [00:55:39] Yeah, I mean, they will do it in a slightly different ways. It's one of them, you know, we'll have their own, uh, specifics about the way that they affect our health.

[00:55:48] Uh, but obviously both of them will have, you know, exposure to both of them are negative to our health. Uh, they are both risk factors for a lot of diseases. Uh, you know, [00:56:00] using this epigenetic information. This should be genetic tests. We can quantify over time how this is affecting us personally, taking into account our genetics, our age, our lifestyle.

[00:56:10] Um, and, you know, be able to follow this over time, as you said, with, with, if you wish, a digital twin, uh, you know, our version of yourself by, uh, with a lot of data to compare against, uh, I'm, you know, take a specific actions to, to go on improve. Um, the first point. That you required to be able to properly improve.

[00:56:30] Um, I'm really know that it's working. It's too tough a way to measure it very accurately. And that's, that's really what we can give you. We give you the most accurate way to quantify in the medium and longterm the effects of this different risk factors, alcohol, uh, tobacco, but, but also the things.

[00:56:46] Carl Lanore: [00:56:46] So, dr seeds, is there anything kind of a.

[00:56:52] 30,000 foot view that we can all agree upon that can help reestablish the methylation [00:57:00] landscape in the body.

[00:57:02] Dr. William Seeds, MD: [00:57:02] Well, I think it's just a, I think the 30,000 foot view is, it's an amazing tool to be able to, you know, we're, we're used to being reactive and, and dealing with it as, as physicians, it's always, we get people when they're.

[00:57:19] At a state where things have already changed, as in they're hard to correct. We now have a method that we can actually look at something before it actually may happen and cause serious damage where we can make real changes that will affect their life for the positive, um, significantly. So I think it's really important to understand that, that we haven't had this capability before.

[00:57:48] And this is, this gives the, this gives the patient more ownership and more authority in taking care of themselves and seeking that knowledge [00:58:00] that they're desperately looking for to improve their health and make sure what they're doing is improving their health and having changes, uh, specific to where maybe we couldn't follow those with blood levels or certain lab tests.

[00:58:15] We couldn't tell you much other than, yeah, they're looking good. Well here, we can tell you, look at this. Look at where you started and look at where you are now.

[00:58:23] Carl Lanore: [00:58:23] I guess. I guess really what I was asking, and I'm gonna rephrase it, because a good friend of mine, Ron Penna, the founder of quest nutrition, loves to tell people and me.

[00:58:32] That quite often it's not what you start taking, but what you stop taking or what not, what you start eating. But what you stop eating that there seems to be a lot of emerging evidence, and I get emails from people all the time, what should I take for this? What should I take for that? And I start to ask them questions.

[00:58:47] Well, what do you do now? Because quite often the magic isn't in adding something in. It's removing something that is the insult. So in your estimation. Both of you. I'll give you both the chance to answer this. [00:59:00] The things that actually help reestablish the methylation pathways, are they things you start taking like B vitamins or they, the things that you stop doing that stop disrupting it.

[00:59:15] Good, Dan,

[00:59:16] Dr. Daniel Elias Martin Herranz, PhD: [00:59:16] that's quite out. Yeah, that's quite a complex question. So I think in some cases you always need to have the basics. Ah, so you'd need to have the basic vitamins, like some of the nutrients, for example. We know that affect directly methylation levels, so you need to get those covered. Right. But then after that, uh, I agree that a lot of this stuff that is going to affect negatively our epigenetics has to do with the stuff that we shouldn't be doing.

[00:59:42] Uh, so stop like, you know, obviously the smoke in the dream exposed to water, pollution, drinking, uh, all this sort of stuff. You know, there are actions, very simple that we can take to improve our health. We felt the need to add anything else. And in that sense, you know, I think once you have covered the basics, uh, I think [01:00:00] most of the areas right now, it's about okay, I agree with, with that view of we need to remove a lot of stuff that is, that is spine.

[01:00:07] Um, so yeah, I would say both ways by definitely looking more from removing the stuff.

[01:00:14] Carl Lanore: [01:00:14] What do you think dr feeds? I

[01:00:17] Dr. William Seeds, MD: [01:00:17] think you're right on I, and I think that just goes back to what I think that's a great question, Carl, because that's how you can really establish your relationships with your patients and saying, okay, look.

[01:00:28] I, I get it. I, and I keep going back to this example, but it's an example everybody can agree with and follow, okay, I get it. You think you're healthy, you think you're working out every day, but on the weekend, this is what you do. Let's just change that and see how it changes this. Let's just just follow with me and do that.

[01:00:47] That's a powerful way of getting that type of focus to change because what are we doing? We're removing a habit. And you know, I'm sorry, but alcohol and tobacco, they're toxins [01:01:00] and they're nothing more than toxins. And that's this. We now can absolutely start being, I think, very, yeah, yeah, absolutely.

[01:01:11] Carl Lanore: [01:01:11] Yeah.

[01:01:12] They're bad for you. Let's just get these last few questions up here and, uh, and then we'll, we'll let you go. So Rigo Vargas says, uh. Do either of you have an opinion about of some alcohol alternatives, like Buton Buton dial. I didn't know that you can get high from butane dial. Isn't that a, that's a ketone, isn't it?

[01:01:32] Dr. William Seeds, MD: [01:01:32] Eugene dial is like a floor stripper, so it's something you use to strip floors, but they use it to, uh, uh, it's, it's a gamma beautiful acetone that they use to take,

[01:01:44] Carl Lanore: [01:01:44] to feel like GHB.

[01:01:46] Dr. William Seeds, MD: [01:01:46] Yeah. Uh, yeah. It's, it's, well, it's a, it's GBA, it's GBA or I think it's GBL or something. But what happens with that actually is it's a significant toxin because it,

[01:01:59] Carl Lanore: [01:01:59] it, [01:02:00] it gives people brain problems.

[01:02:03] It's

[01:02:03] Dr. William Seeds, MD: [01:02:03] metabolized to, uh, to, uh, to, uh, give a hydrogen, uh, butyrate. And that is toxic. It causes a arrhythmias. It causes a, um, uh, eh, I mean, it's, it's significant. Uh, uh, liver failure. Um, it's a, it's a toxin for sure. So that's a really good question because I didn't think that was legal actually, but

[01:02:28] Carl Lanore: [01:02:28] it's not, it used to be a date rape drug.

[01:02:31] What they used to use it for date rape because it renders you, uh, completely, you know, unconscious and mobile people used to use that as a date rape drug. They used to put it in girl's drinks. That's why that's why GHB was taken off off the market, because guys will put it in people's drinks and knocking them out.

[01:02:50] Well, that's

[01:02:50] Dr. William Seeds, MD: [01:02:50] a metabolite of that

[01:02:52] Carl Lanore: [01:02:52] GPO GBL charge of the GHB. Right. Scott Lola said he'd ordered the test, but he's just about to pull the trigger on some [01:03:00] BPC oral. He said he will get the test eventually. Thank you, Jeff. Cliff. And I think he was talking about when I was saying about tobacco, when people really don't give a damn about health, that they continue to support the sale of tobacco.

[01:03:12] Uh, could, uh, dr seeds update us on a timeline for his peptide protocol book volume one. He was discussing it the other day with somebody. When's that book coming out, doc?

[01:03:24] Dr. William Seeds, MD: [01:03:24] That book is out to the publisher right now.

[01:03:26] Carl Lanore: [01:03:26] Oh. So it should be hitting shelves pretty soon then.

[01:03:30] Dr. William Seeds, MD: [01:03:30] It should have been out. I think it should have been out here last month, but because of this coven issue, it's been, I don't know, it's I, that's the black hole.

[01:03:40] Once it goes there, it's the black hole. It's a new black hole I've identified and I can't, I have, I have no control of, but it's, it's exciting cause it could be tomorrow. It could be next week. I, I, it's there.

[01:03:55] Carl Lanore: [01:03:55] Uh, we're going to take a commercial break and when we come back, we're gonna be joined by a guy [01:04:00] who I love his products and they make a lot of sense.

[01:04:03] Um, there's a lot of discussion about five G around right now. You can actually do things to protect yourself if you understand why you need to be protected. Of course. Before we go. Uh, I want to go ahead and just promote one more time that you can get this amazing epigenetic test that's only available to my audience at seeds.

[01:04:22] That MD slash epigenetic hyphen test slash 70% off 20 million. Data points of your DNA are tested and it's a saliva test, which means you don't have to go to the doctor, you don't have to prick your finger, you don't have to have blood drawn. You just spilled the vial. I just filled it up. You just filled the vial up with saliva, close the cap on it, screw tight, put it in the package that they give you, send it back, and it takes, how long does it take Danny for people to start to get the results back from their testing?

[01:04:52] Yeah,

[01:04:53] Dr. Daniel Elias Martin Herranz, PhD: [01:04:53] so normally it's around eight weeks. Um, obviously the current situation with like, you know, sending [01:05:00] samples, et cetera because of the Colby 19 situation, um, might be a slightly different. Um, so we're now, you know, seeing how that evolves. Uh, but that's the normal, yeah, that's the normal turnaround time.

[01:05:11] Carl Lanore: [01:05:11] I'm excited. I just sent mine and I can't wait to see it because I want to see what I can do better to age better. I'll be 62 in June, and I'm probably a little bit better off than most 62 year olds, but I think I can be better. I really do. I believe it. So we're going to find out, and I'll be talking about my results from my test openly on the air, and we'll probably do a show just about that.

[01:05:34] Uh, next time I, we're going to do this, we're going to say goodbye to our guests, uh, and we're going to take one quick commercial break. And when we come back, we're going to talk about how to protect yourself from RF. It's a very, very interesting and important discussion. And those of you who know I have a history in a land, mobile, radio paging and cell phone, and I have a very, very good understanding of how RF works [01:06:00] and the gentlemen that I'm going to be joined by in just a moment, he's going to show you just how you can protect yourself.

[01:06:06] So stay tuned. We'll be right back with more superhuman radio. You are listening to the superhuman channel. We're ripped and we're ready.

[01:06:22] Welcome back to superglue radio where we're joined and I'm going to, I'm going to take an attempt to pronounce your name properly and you have to forgive me. It's author Menard de Kalench correct? That was good. That was good. All right. That's great. I gotta be honest with you. I've been doing this show 14 years.

[01:06:39] I pride myself and being able to pronounce people's names properly because I think it's the greatest insult. Too. If you can't, if you're not sure, ask. It's the it, you know, we, we have names for a reason and I really, really strive to make sure that I pronounce it. Sometimes I'll go look up how to pronounce a name on the internet before I have a guest dog, cause I want to honor them [01:07:00] by getting their name right.

[01:07:01] Welcome to the show. I'm very excited to have you on, um, for a couple of weeks here. Yeah. So, so here, here's some interesting background about me. First. Um, when I was 22 years old, I moved to Las Vegas, Nevada. I had been working on in college to become an optometrist. And, uh, I had my heart broken and I abandoned everything and moved to Las Vegas.

[01:07:24] And, uh, I, uh, got involved in the land, mobile radio and mobile telephone and paging business. This is back when I, MTS mobile phones, 12 channels for the whole city, uh, timeout timers and paging, of course, and so on. And I actually started a business with a good friend of mine. He's a good friend now, all these years later, John Babcock.

[01:07:45] And I was the, I was the second cellular agent that Craig McCall signed up back then. He signed months up in LA and he signed us up cellular city. We had another company, cellular city with my friend alpha Santo, and he signed us up in, in, in Las [01:08:00] Vegas. And, and at that time only Chicago was online. And they built the site.

[01:08:05] And I had spent, you know, a decade in land mobile radio. I understand what re RF can do and I've told stories on this show. Some of my listeners who've been listening for years know it. When I, one day I was putting up a 462 megahertz folded dipole antenna and the technician brought a fluorescent light bulb up with them.

[01:08:25] I said, Jack, what's that for? He's going to show you something. He goes, we're going to measure the quarter wavelength of this antenna, and he had the guy down in the radio room key, the transmitter. He started right up against the Tran, the antenna. He moved it out and when he got to 462 the frequency quarterly, like the bulb lit up, no wires connected to it.

[01:08:48] Okay. That was the moment I thought, Oh my God. Like we don't see this stuff, but it's doing something. It's really doing something. It's exciting. The fluorescent tube, another time I [01:09:00] told this story on the show, I held an antenna on Mount potency when they keyed it and I felt this tingling. I didn't think nothing of it, and I just took my hand off it.

[01:09:08] Later that night when I got home, my hands split open like a hot dog when you overcook it, and I had proud flesh sticking out of my hand. I was like, wow. So I understand. We live in, and just recently on Facebook, I have a spectrum analyzer fits right here on my desk. I keep it here and I took pictures for the audience.

[01:09:29] I did a video for the audience to show them what four G looks like, and this BR, this blue wave. Transmitting from this frequency that I frequency, and you can see the little pips of the higher frequencies coming. And I said, this is a cloud of RF that we live in day in and day out. And more and more research is showing that RF does things to us on a cellular level.

[01:09:54] Does things source on the cellular level? Now enter you, you started a [01:10:00] company, how long ago? Uh, we

[01:10:02] Dr. Daniel Elias Martin Herranz, PhD: [01:10:02] got started about two years ago,

[01:10:04] Carl Lanore: [01:10:04] two years ago. Why? Why, what, what, what did you know that made you start doing this? So, it's

[01:10:10] Arthur Menard de Calenge: [01:10:10] actually an interesting story.

[01:10:12] Dr. Daniel Elias Martin Herranz, PhD: [01:10:12] We, I've got a background in

[01:10:14] Arthur Menard de Calenge: [01:10:14] engineering and biology originally, and I was at dinner with friends and we started discussing the fact that it's been years that we've heard about the fact that.

[01:10:25] Dr. Daniel Elias Martin Herranz, PhD: [01:10:25] Cell phone radiation are probably dangerous for your health, that you

[01:10:29] Arthur Menard de Calenge: [01:10:29] shouldn't be keeping them in your pockets, that you shouldn't be using wifi, et cetera, et cetera. And we were discussing about the fact, Hey, you guys are, where are your cell phones in our pockets? Who owns.

[01:10:41] Dr. Daniel Elias Martin Herranz, PhD: [01:10:41] A fitness tracker, um,

[01:10:43] Arthur Menard de Calenge: [01:10:43] who has, who has wifi.

[01:10:45] Dr. Daniel Elias Martin Herranz, PhD: [01:10:45] And, uh, that same evening

[01:10:47] Arthur Menard de Calenge: [01:10:47] I was like, all right, well, we've had fun, but let me dig into web the sciences around this. And, um, and, and if there is an issues associated with what it's radiation, and this [01:11:00] is when

[01:11:00] Dr. Daniel Elias Martin Herranz, PhD: [01:11:00] I

[01:11:01] Arthur Menard de Calenge: [01:11:01] landed on the website of the who, the world health

[01:11:05] Dr. Daniel Elias Martin Herranz, PhD: [01:11:05] organization.

[01:11:06] Arthur Menard de Calenge: [01:11:06] And found out that the categorization of where I sortation right now was the class to be human, Gustavo gen, um, which is a same categorization as car exhaust fumes,

[01:11:18] Dr. Daniel Elias Martin Herranz, PhD: [01:11:18] right?

[01:11:19] And the great thing about

[01:11:21] Arthur Menard de Calenge: [01:11:21] cogs as

[01:11:22] Dr. Daniel Elias Martin Herranz, PhD: [01:11:22] students is that you can smell them. And

[01:11:24] Arthur Menard de Calenge: [01:11:24] if in

[01:11:25] Dr. Daniel Elias Martin Herranz, PhD: [01:11:25] this room right now,

[01:11:26] Arthur Menard de Calenge: [01:11:26] or the call was running.

[01:11:27] Dr. Daniel Elias Martin Herranz, PhD: [01:11:27] A cousin Jenna was running, I would be

[01:11:30] Arthur Menard de Calenge: [01:11:30] out of here

[01:11:31] Carl Lanore: [01:11:31] and you'd know, you'd know. You'd recognize it. You say, what's that smell? Let me, I mean, they even put, they put us a cent in natural gas so that you know that it's leaking in your home, otherwise you wouldn't smell it and you die.

[01:11:48] Arthur Menard de Calenge: [01:11:48] Very tournament. And so

[01:11:50] Dr. Daniel Elias Martin Herranz, PhD: [01:11:50] that was really my

[01:11:51] Arthur Menard de Calenge: [01:11:51] aha moment where it was like, wow, um,

[01:11:53] Dr. Daniel Elias Martin Herranz, PhD: [01:11:53] I

[01:11:54] Arthur Menard de Calenge: [01:11:54] can't feel this things like on

[01:11:55] Dr. Daniel Elias Martin Herranz, PhD: [01:11:55] see them, but they're definitely here.

[01:11:58] Arthur Menard de Calenge: [01:11:58] And at the same

[01:11:58] Dr. Daniel Elias Martin Herranz, PhD: [01:11:58] time,

[01:11:59] Arthur Menard de Calenge: [01:11:59] contrary [01:12:00] to, um, I mean, Corazon not the right example, but conferred to smoking that we're discussing before. Um, technology

[01:12:08] Dr. Daniel Elias Martin Herranz, PhD: [01:12:08] is amazing.

[01:12:09] Uh, and the fact

[01:12:10] Arthur Menard de Calenge: [01:12:10] that we're able to do this interview right now, and it's true me to thousands of people. It's just

[01:12:16] Dr. Daniel Elias Martin Herranz, PhD: [01:12:16] incredible.

[01:12:17] Arthur Menard de Calenge: [01:12:17] And, um, wireless radiation are enabling all this. And that's kind of how the lens story got started, which was about keeping using technology and not changing anything about the way we live today.

[01:12:30] Dr. Daniel Elias Martin Herranz, PhD: [01:12:30] Um, but

[01:12:31] Arthur Menard de Calenge: [01:12:31] being able to do so without putting our health and our, uh, general, uh, wellbeing at

[01:12:38] Carl Lanore: [01:12:38] risk. So there are more and more studies. And here's the funny thing, a lot of these really good studies are not being done in the United States, which makes me very suspicious probably because. No one can get funding here in the United States for these studies, but if you go to Scandinavia, Sweden has been way ahead of us.

[01:12:54] You know, back in the day, Nokia, Sweden has been way ahead of us. Uh, uh, [01:13:00] ironically, uh, Egypt is way ahead of us in this particular area. Uh, even Iran. There's some great studies that came out of Iran, but none out of the United States. And when I see that, I think that's weird, but it's really not when you think about it.

[01:13:16] And that's because we love our phones. We don't want to give up our phones. Don't tell me there's something, the earlier in the show, I'm telling people not to drink and smoke anymore. Oh, now you want me to get rid of my cell phone to call? Forget it. I'm not going to listen to you anymore. But listen, I'm telling you something.

[01:13:32] There are studies that show, and I don't mean one, numerous studies that show the following, that having a cell phone call 30 minutes before going to bed increases deep sleep latency by as much as 50% there are studies that show that exposure to different frequencies of RF changed the landscape of the microbiome in your gut.

[01:13:57] There are studies that show that wearing [01:14:00] your phone. Now, it doesn't say this specifically, but you can deduce this. There we did a show about changes in heart valve function, uh, idiopathic mitral valve prolapse. There are cilia, there are cilia on each cell. They, there are anywhere from two microns to two millimeters long.

[01:14:19] And these cilia are specifically there for cellular to cellular communication. They're basically the cells antenna. That's what they are, and they're showing that cilia is picking up something. They said something in this study because here in the United States that's confusing the valve and causing mitral valve prolapse, and I connected the dots.

[01:14:41] It's picking up RF. How many times do you see a guy with his pocket? His is his phone in his pocket, right over his heart. It's causing rhythm issues with people's hearts. Okay. Remember what I said before, the quarter wavelength. Some of these little Selia, the cilia are exactly the right quarter wavelength for some of the [01:15:00] new emerging 5g frequencies coming out.

[01:15:02] Six gigahertz and up. And later in the show, I'm going to talk about the myth that people are trying to confuse. They're saying, Oh, five G is causing COBIT. It's not. I'm going to talk about that later and I'm gonna. I'm going to let you stay away from that because I'm going to splash crazy all over everybody in a little while, but the reality is that R F is affecting us on a cellular level.

[01:15:23] Egypt, they did the earliest study of using the old two G and three G generation, second generation, third generation phones. They just put them beneath. The rat's cage, male rats cage, and they turned them on, but didn't make any phone calls because people think if you're not using your phone, it's not transmitting.

[01:15:41] It's transmitting constantly. It's sending data to the cell tower that tells cell tower sending data back. Are you there? Here I am to know, go to that tower, go to this town. Constantly. You're not on the phone. It's constantly transmitting. That's why if you sleep with your phone next to your head, your sleep probably sucks, and I can fix that right now.

[01:16:00] [01:15:59] Turn your phone off and go to sleep without it, and your sleep will get better. But when you look at some of these studies, the rodents, sperm motility dropped by 80% 80% we have a huge problem right now with male infertility. A lot of these guys are carrying their phones in their pocket. It's easy to fix.

[01:16:18] Get the phone away or. Where a pair of underwear, that RF shielded, that's where you come into the picture. You saw this developing need and you created a whole line of, of, of a apparel, right? You have hats, underwear. What else? Don't you even have shirts.

[01:16:35] Dr. Daniel Elias Martin Herranz, PhD: [01:16:35] We do.

[01:16:35] Arthur Menard de Calenge: [01:16:35] Yeah. We have t-shirts. Um, and we have a lot more coming as well.

[01:16:39] Um, and, and

[01:16:40] Dr. Daniel Elias Martin Herranz, PhD: [01:16:40] yeah, the

[01:16:41] Arthur Menard de Calenge: [01:16:41] D

[01:16:41] Dr. Daniel Elias Martin Herranz, PhD: [01:16:41] idea, we, we read the same

[01:16:43] Arthur Menard de Calenge: [01:16:43] studies. And you're just given

[01:16:47] Dr. Daniel Elias Martin Herranz, PhD: [01:16:47] that you're barely

[01:16:48] Arthur Menard de Calenge: [01:16:48] scraping the surface of what's out there today because we're talking about. Thousands of studies out there, uh, showing the adverse effects of where it is radiation. And at the same [01:17:00] time, as I was saying, like keeping your cell phone away from your pocket.

[01:17:03] In a world where we check our phone and thinks 350 times a day

[01:17:07] Dr. Daniel Elias Martin Herranz, PhD: [01:17:07] on average

[01:17:08] Arthur Menard de Calenge: [01:17:08] is just not something that people are willing to do naturally. And so we are developing the next best alternative.

[01:17:15] Dr. Daniel Elias Martin Herranz, PhD: [01:17:15] Which was,

[01:17:15] Arthur Menard de Calenge: [01:17:15] okay, if you want to keep yourself on your money in your pocket, can we shield your buddy from

[01:17:20] Dr. Daniel Elias Martin Herranz, PhD: [01:17:20] those waves?

[01:17:21] And

[01:17:23] Arthur Menard de Calenge: [01:17:23] what we look into is,

[01:17:24] Dr. Daniel Elias Martin Herranz, PhD: [01:17:24] okay, is there any

[01:17:26] Arthur Menard de Calenge: [01:17:26] physical principles that we can use? Is there any technology are out there,

[01:17:30] Dr. Daniel Elias Martin Herranz, PhD: [01:17:30] are

[01:17:30] Arthur Menard de Calenge: [01:17:30] already allowing us to do this? And we found out that NASA spacesuits have a built in relation Perth technology. Because if you're an astronaut and you're going to space on a six month journey, you're.

[01:17:43] Exposed to approximately 1600 chest X rays in terms of mountain of exposure to radiation that you're taking. And so, um, the NASA engineers, I've developed a relation for technology that is directly

[01:17:55] Dr. Daniel Elias Martin Herranz, PhD: [01:17:55] embedded within

[01:17:56] Arthur Menard de Calenge: [01:17:56] the space suits of the astronauts. And so we [01:18:00] use the same physical plants. People. Which is called electromagnetic shielding.

[01:18:05] Dr. Daniel Elias Martin Herranz, PhD: [01:18:05] And we integrated this into a fabric

[01:18:08] Arthur Menard de Calenge: [01:18:08] that we can wear, um, on in normal clothing, and that is comfortable and soft, and that would act just as a regular, uh, regular fabric. Um, and yeah,

[01:18:20] Dr. Daniel Elias Martin Herranz, PhD: [01:18:20] that's the idea behind that

[01:18:21] Carl Lanore: [01:18:21] RF shielding is measured in decibel. DB. So the hat or the shirt for instance, how, how much of a DB reduction do you see in incident RF?

[01:18:34] That is, that is hitting you.

[01:18:37] Yeah.

[01:18:37] Dr. Daniel Elias Martin Herranz, PhD: [01:18:37] We're up to site the DB reduction.

[01:18:40] Carl Lanore: [01:18:40] We'll say that again.  that's great. So theoretically you, you're not getting a hundred percent out, but you're probably getting about 90% of the RF out of, of,

[01:18:51] Dr. Daniel Elias Martin Herranz, PhD: [01:18:51] uh. 99 point 99

[01:18:53] Carl Lanore: [01:18:53] okay. Okay. Excellent. So, so theoretically if someone, and you know this is something that's interesting because [01:19:00] if you ask people today in the United States, everybody's sleep sucks, right?

[01:19:05] And even if you unplug your phone and put it away and go to the extremes that I do, I unplugged the router. Before I go to bed and I unplugged my cordless base station before I go to bed. I wish I had a, I wish I could just flip the power on the house completely and just leave the heater and air conditioning and running.

[01:19:22] That next, next house I build, I'm going to modify some things, but. That still doesn't solve the problem that my neighbor's got a router that neighbor's got a router, they've got cell phones, they've got cordless phones. So by me doing this at home, I can't really protect myself, but if I slept with the get lambs had on, I probably would stop the RF from reaching the brain and causing any type of activity at night.

[01:19:47] What do you think?

[01:19:49] Dr. Daniel Elias Martin Herranz, PhD: [01:19:49] Very true.

[01:19:50] Arthur Menard de Calenge: [01:19:50] And this is Nisha that is even more present in big cities like New York or. Uh, Los Angeles or wherever you're, you might be living if [01:20:00] you're living in a, in a big building with, um, tons of neighbors. So I was doing this experiment at home the other time. I'm turning on my wifi and counting.

[01:20:10] Dr. Daniel Elias Martin Herranz, PhD: [01:20:10] I think I'm at 25

[01:20:12] Arthur Menard de Calenge: [01:20:12] or anywhere between 45 to study wifi sources, um,

[01:20:16] Dr. Daniel Elias Martin Herranz, PhD: [01:20:16] like in my bedroom

[01:20:18] Arthur Menard de Calenge: [01:20:18] at the moment. And so that's, uh, yeah, that's, that's pretty interesting when you, when you do cut it this way

[01:20:24] Carl Lanore: [01:20:24] now, have you ever, have you ever put. You're the, the, the, the cap, the skull cap on and slept with it.

[01:20:31] Arthur Menard de Calenge: [01:20:31] Um, I do.

[01:20:32] Yeah. Um,

[01:20:33] Dr. Daniel Elias Martin Herranz, PhD: [01:20:33] not every night,

[01:20:34] Arthur Menard de Calenge: [01:20:34] especially right now. I live in Los Angeles. It's pretty hot here.

[01:20:38] Dr. Daniel Elias Martin Herranz, PhD: [01:20:38] Um. But I do sometimes in the winter,

[01:20:41] Arthur Menard de Calenge: [01:20:41] and I've noticed a significant difference on

[01:20:45] Dr. Daniel Elias Martin Herranz, PhD: [01:20:45] my GC actually

[01:20:46] Arthur Menard de Calenge: [01:20:46] when doing it versus not doing it. It's something that

[01:20:50] Dr. Daniel Elias Martin Herranz, PhD: [01:20:50] I, I'm

[01:20:51] Arthur Menard de Calenge: [01:20:51] one of those guys who Sgt. Sleep as well. Uh, and I've tried a lot of things to improve it.

[01:20:57] And that was actually one of the steps that [01:21:00] was most helpful. I turn off my cell phone at night as well. Um, and I turn off my wifi. So I've set up a quick tip for your call. Um, what you can do is buy a programming plug, which will automatically turn out, turn out five nights, um, during the Yellowstone, which just sleep and then you don't have to worry about it.

[01:21:19] Carl Lanore: [01:21:19] That's nice. Um, how about, how about coming out? Remember back in the old days. People slept with a nightcap on, right, because there was this, it was like a little, little, very, very light hat with a little ball on the top, or it looked like a skull cap. And how about coming out with one specifically for sleep?

[01:21:35] So on the warm weather, people can still sleep with it on their head.

[01:21:39] Arthur Menard de Calenge: [01:21:39] It's a great idea, and I'd love to bring back this style to be honest.

[01:21:44] Carl Lanore: [01:21:44] And I see pajamas following too, right? So if you've got to get the RF off of your brain, you may as well get it off of as much of your body as possible because there's, you know, people think that grounding sheets do this.

[01:21:55] They don't do this incident. RF is still going to rain down on your body, [01:22:00] even if you're on a grounding sheet. And in fact, I would go as far as saying a grounding sheet may turn you into an antenna. Very true.

[01:22:09] Arthur Menard de Calenge: [01:22:09] And, and I love that you touched on that because that's something that people do not realize. Is that in order to

[01:22:16] Dr. Daniel Elias Martin Herranz, PhD: [01:22:16] create the

[01:22:17] Arthur Menard de Calenge: [01:22:17] electromagnetic shielding that we're talking about, or also cold as

[01:22:22] Dr. Daniel Elias Martin Herranz, PhD: [01:22:22] a fire to cage, what you need

[01:22:24] Arthur Menard de Calenge: [01:22:24] is to shield completely the body

[01:22:28] Dr. Daniel Elias Martin Herranz, PhD: [01:22:28] that you're trying to shield.

[01:22:29] Arthur Menard de Calenge: [01:22:29] So if you look at your microwave that's using the same pricy principle. Um, look at the door of your microwave and you'll see that there is a minimum grid in it. This great is actually in every word in your microwave. And that's what creates

[01:22:43] Dr. Daniel Elias Martin Herranz, PhD: [01:22:43] a

[01:22:43] Arthur Menard de Calenge: [01:22:43] completely closed off and closure. And with, um, just a

[01:22:48] Dr. Daniel Elias Martin Herranz, PhD: [01:22:48] little bit of sealer,

[01:22:50] Arthur Menard de Calenge: [01:22:50] for instance, um, which has conducted conductive material that allows us to do this type of shielding.

[01:22:56] If you're

[01:22:56] Dr. Daniel Elias Martin Herranz, PhD: [01:22:56] not,

[01:22:58] Arthur Menard de Calenge: [01:22:58] first of all, building your civil grid [01:23:00] the right way. And second of all, if you're not putting the silvery everywhere

[01:23:04] Dr. Daniel Elias Martin Herranz, PhD: [01:23:04] around the buddy,

[01:23:06] Arthur Menard de Calenge: [01:23:06] um, then you're actually doing the opposite effect,

[01:23:09] Dr. Daniel Elias Martin Herranz, PhD: [01:23:09] which is silver attracts radiation.

[01:23:11] Arthur Menard de Calenge: [01:23:11] You're turning yourself into an antenna, and what you do is you're effectively doing the opposite of what you're trying to

[01:23:17] Dr. Daniel Elias Martin Herranz, PhD: [01:23:17] achieve,

[01:23:17] Arthur Menard de Calenge: [01:23:17] right?

[01:23:18] Um.

[01:23:19] Dr. Daniel Elias Martin Herranz, PhD: [01:23:19] So I would

[01:23:20] Arthur Menard de Calenge: [01:23:20] highly recommend paying attention to this. Whenever I'm looking at products that have silver and,

[01:23:26] Dr. Daniel Elias Martin Herranz, PhD: [01:23:26] um, we have,

[01:23:27] Arthur Menard de Calenge: [01:23:27] like after launching lambs on the market, we've had a bunch of. Companies coming out with products, products that actually do not work because of that, because they use the silver because they don't get how the technology works.

[01:23:41] Um, and that's been, uh, some we've been warning people about quite a bit.

[01:23:46] Carl Lanore: [01:23:46] Yeah, exactly. And then, and then there's, uh, when you, when you have an antenna, there is something called a SIG. That energy must escape somewhere. And when that energy escapes, it also causes heat. [01:24:00] And so your body becomes the sink Sian, K for the, uh, for the RF that's accumulating if you're not completely enclosed, uh, and like you said, and you become an antenna.

[01:24:10] And that's why I questioned whether or not grounding sheets and grounding yourself is actually a good idea today given the fact that we have all this RF, uh, around us. Talk about some of the other studies that maybe I have missed. I, we know about infertility, we know about brain activity. We know about the microbiome.

[01:24:30] We and we all carry our own personal transmitter, so we don't even have to rely on the ambient RF in our, in our environment. We're doing the work for it. What other research have you seen recently that seems, uh, uh, very interesting.

[01:24:45] Arthur Menard de Calenge: [01:24:45] So I, one thing that I've been kinda digging into in the recent month is actually the molecular, or rather similar in this case, um, patterns as to what happens when a cell is exposed [01:25:00] to EMF radiation.

[01:25:01] Um, and here's the gist of whatever announce so far. Um, so membranes

[01:25:08] Dr. Daniel Elias Martin Herranz, PhD: [01:25:08] are

[01:25:08] Arthur Menard de Calenge: [01:25:08] polarized. Naturally and EMS sent for electromagnetic frequencies, then naturally polarized as well. And so

[01:25:17] Dr. Daniel Elias Martin Herranz, PhD: [01:25:17] when a wireless radiation

[01:25:18] Arthur Menard de Calenge: [01:25:18] such as your cell phone radiation or your wifi irrigation will get in contact with one of yourself or one of the cells of your body, this polarization of the wave, um, will mess with the integrating of the cell membrane.

[01:25:33] And this is taken

[01:25:35] Dr. Daniel Elias Martin Herranz, PhD: [01:25:35] by

[01:25:35] Arthur Menard de Calenge: [01:25:35] yourself as an external aggression. And so what we've done, um, this is actually a study that we've done here,

[01:25:44] Dr. Daniel Elias Martin Herranz, PhD: [01:25:44] um,

[01:25:44] Arthur Menard de Calenge: [01:25:44] is with studying the immune system reaction, uh, of your body when exposed to a cell phone call

[01:25:53] Dr. Daniel Elias Martin Herranz, PhD: [01:25:53] and

[01:25:53] Arthur Menard de Calenge: [01:25:53] what we see. So the best way to, uh, sort of. Your immune system, um, called, you probably knows [01:26:00] this better than I do, but you essentially have two modes, rest and digest or

[01:26:05] Dr. Daniel Elias Martin Herranz, PhD: [01:26:05] fight and flight,

[01:26:06] Arthur Menard de Calenge: [01:26:06] and, um, your body performs better when your immune system is either in check or in rest and digest.

[01:26:12] Most of the time in this year. Doing a particular supports, um, efforts. And so what we found is that the moment you make a cell phone call or you have a cell phone next to you, your buddies switches to be in, um, in fight and flight

[01:26:26] Dr. Daniel Elias Martin Herranz, PhD: [01:26:26] mode. And

[01:26:28] Arthur Menard de Calenge: [01:26:28] more interesting me if we shield your buddy from radiation using our fabric, um, and we put a cell phone next to you.

[01:26:38] We do not have, um, this switched to, um, fight and flight.

[01:26:43] Dr. Daniel Elias Martin Herranz, PhD: [01:26:43] And if we're in fight or flight,

[01:26:44] Arthur Menard de Calenge: [01:26:44] uh, within 10 minutes of being shielded, you go back to rest and digest. Um, and what's super interesting there is that your immune system not being in check leads to, um, inflammation, uh, altogether. And inflammation is the [01:27:00] root cause of so many of today's issues.

[01:27:04] Um, and it's, and it's. It's something that we see being increased by a number of things that we put in our bodies, including

[01:27:13] Dr. Daniel Elias Martin Herranz, PhD: [01:27:13] alcohol and

[01:27:14] Arthur Menard de Calenge: [01:27:14] tobacco, that we were just, that you guys were just discussing before. But this is also a factor. And, um, what increased inflammation and, um, and essentially accepted that oxidative stress.

[01:27:27] Leads to are, is, um, cancer, cardiovascular diseases, uh, neurological disorders have already have issues that in the longterm, um, are, are significantly impacting

[01:27:40] Carl Lanore: [01:27:40] our health. So let's talk specifically about the apparel. I know about the hat and the underwear. You have tee shirts. Do you have anything else, uh, that people can purchase that, that offers this perfect protective effect?

[01:27:53] So

[01:27:53] Arthur Menard de Calenge: [01:27:53] we're working on a lot of

[01:27:55] Dr. Daniel Elias Martin Herranz, PhD: [01:27:55] new products

[01:27:56] Arthur Menard de Calenge: [01:27:56] that will come out in the next few months. One of the things [01:28:00] that,

[01:28:00] Dr. Daniel Elias Martin Herranz, PhD: [01:28:00] uh, were

[01:28:00] Arthur Menard de Calenge: [01:28:00] key in us developing lens is that we take rates, plow applied to, sorry, in developing great products. And

[01:28:10] Dr. Daniel Elias Martin Herranz, PhD: [01:28:10] so we actually take

[01:28:12] Arthur Menard de Calenge: [01:28:12] a lot of time to come up with, uh, each in each and every single one of our products because we want to make sure that when you're wearing the shirt, when you're wearing the underwear or the beanie.

[01:28:22] Uh, you're wearing your best shirt, your best underwear has been, you're not making a trade off of like, Hey, I'm wearing that thing that is super uncomfortable, but it can be something expected for math. Um, like we're trying to give people a no brainer choice, which is, Hey, this is already a super comfortable t-shirts.

[01:28:42] It's also antibacterial because silver has natural antibacterial properties. Um, and he diffusing as well. So essentially just a better tee shirt in the first place. It feels good. It looks good. Um, and it blocks radiation. And so we have, um, a few new product lines that will come out this [01:29:00] year, um, constantly just surprise just yet.

[01:29:02] But for people following us on social media or, um, who are chasing our now mailing lists, you'll get the news first. Um. And, uh,

[01:29:11] Dr. Daniel Elias Martin Herranz, PhD: [01:29:11] and yeah, we've, what's,

[01:29:12] Arthur Menard de Calenge: [01:29:12] what's been great is that we have a community of customers who are

[01:29:16] Dr. Daniel Elias Martin Herranz, PhD: [01:29:16] super engaged with,

[01:29:17] Arthur Menard de Calenge: [01:29:17] uh, lamps. And so we're able to, could develop our products with our customers, making sure that we address, um, the.

[01:29:25] Number one needs first. And um, and we have a very, very don't list now that we're working yet that we're working to, to, to, to get products out the door as soon as possible.

[01:29:39] Carl Lanore: [01:29:39] So obviously these, since it's made with silver, it's, it's, it's, it's somewhat more expensive, let's say that fruit of the loom t-shirts at target.

[01:29:50] How about the durability? Do they last a long time?

[01:29:54] Arthur Menard de Calenge: [01:29:54] So that's the beautiful thing about silver is that it

[01:29:57] Dr. Daniel Elias Martin Herranz, PhD: [01:29:57] actually makes the

[01:29:59] Arthur Menard de Calenge: [01:29:59] product more [01:30:00] resistant to washes and just overall being worn out. So,

[01:30:06] Dr. Daniel Elias Martin Herranz, PhD: [01:30:06] uh,

[01:30:06] Arthur Menard de Calenge: [01:30:06] my personal purse, I've been wearing the first prototypes for I think three years now. Um, and that's still fine.

[01:30:15] That's kinda crazy. The first prototypes were made with cotton, um, and the cuts on the starts to wear off now. But the silver is still perfectly fine. Um, so that's one of the good things about the, uh, the, the product itself, is that the job that it is also great.

[01:30:33] Dr. Daniel Elias Martin Herranz, PhD: [01:30:33] Um. But

[01:30:34] Arthur Menard de Calenge: [01:30:34] yeah, the, the cost of making these compared to a regular and where, I mean, we know where we stand work about 15 times more expensive to produce

[01:30:46] Carl Lanore: [01:30:46] silver.

[01:30:46] Silver is not cheap. Silver is, you know, a precious metal. I mean, you're wearing a precious metal. You really care about yourself. If you're wearing something made out of silver, let's be honest. Uh, the website is get Lamb's dot com G T L a [01:31:00] M B s.com. The code is SHR to save 20% off. Check them out. I think the underwear for men, and I think the skullcap is a must for everybody, given the fact that we live in literally a soup of radiofrequencies today.

[01:31:15] And no, no one's being honest about what these things are doing to you. They're really not. Uh, we're going to take a last commercial break and when we come back, I'm going to discuss, um, I don't know if it's purposeful, but there is a lot of misinformation going around. Five G is bad stuff. I will tell you that five G is bad stuff.

[01:31:36] I'm not, I'm not going to sugarcoat that, but it's not causing Corona virus. I can assure you of that as well. And I'm going to explain. In very simple terms, I'm going to bring everybody up to speed. Uh, we're going to have a little RF one Oh one class. Uh, we're going to say goodbye to author, author, thanks for being here today.

[01:31:54] Uh, love your products. Get lambs.com is the place to go. Use the code. SHR. Show them some love. They are a [01:32:00] sponsor. And you know, the only reason why this show is out and you get this information for free is because of the sponsors. So. Check them out. We'll take one quick commercial break. We'll be right back with more of the super human radio show.

[01:32:12] Stay tuned and we're ready.

[01:32:25] Hey, welcome back. So this is going to be a radio frequency engineering school one Oh one and we're going to do this show and this discussion. Specifically because there is some real stupid, stupid stuff going around the internet right now. And I'm not saying people are stupid. Don't get me wrong. People are worried.

[01:32:47] The truth is, so I get regularly asked if I think covert 19 is being caused by four four by five G. And it's absolutely not [01:33:00] happening that way. It definitely isn't. There's no doubt about it. And I think when we're finished talking, you'll feel the same way. Um, the reality is that five G is not a good thing.

[01:33:10] It's not four G wasn't a good thing. 3g wasn't a good thing. This a accumulation of radio frequencies in our environment. Or having an effect on us and they're taking a toll on our health. And it's the God's honest truth that maybe we won't know about it for another a hundred years until they find another way to make money on a radio communication.

[01:33:32] And they do away with it, but it, but the research is out there. The research is sound. It's, it's not baloney research. It's not being paid for by the cellular companies. In fact, I don't see any good research being paid for by the cellular companies cause they don't want people to be concerned about buying their next $1,200 iPhone.

[01:33:52] Um. But the reality is that while RF is bad and COBIT 19 a bed, they have zero to do with [01:34:00] anything with each other. So let's, let's start off with what real five G is. First of all, so four G goes up to 5.7 gigahertz and five G starts at. Um, I'm sorry, four G goes up to 6.7 gigahertz and 5g starts at six gigahertz, but it goes all the way up to 60 gigahertz, and the higher you go, the more dangerous it becomes because the smaller the frequency becomes, and because now it's able to excite.

[01:34:33] Uh, smaller, uh, cilia and smaller cells and tissue. Cause remember RF interacts through the phenomenon of attenuation and it's kind of a lock and key relationship loosely. And that means that in order for a radio frequency to stimulate something, it must be. An exact ratio [01:35:00] down to one, 100th of the wave form.

[01:35:03] So theoretically, if the wave form is, is a hundred millimeters from peak to trough, then an antenna, a successful antenna could be one millimeter and anything in between that, it's not going to be excited. Right? If you hit a one and a half millimeter a antenna, it's not going to be excited. Okay? So this is the phenomenon of attenuation.

[01:35:25] So the next thing I want to talk about is high five G is, uh, I'm sorry, low five G is five, five gigahertz of six gigahertz. I'm getting ahead of myself to 24 gigahertz, high five G, which is the future, 24 gigahertz to 60 gigahertz. Why, why, why are these frequencies, these higher and higher frequencies, uh, needed?

[01:35:49] And you have to look all the way back. To the days of modems. We all remember those of you old enough, remember, dial up, dial up sounds like static to you, doesn't it? [01:36:00] It's not the phenomenon of frequency shift keying was developed by Motorola for the military. What frequency shift keying is have you take a frequency of one Hertz.

[01:36:14] That means from trough to peak. One per second. That's one Hertz, one cycle per second. That means the trough to the peak from the trough to the peak is one cycle. So if you had a point and they were passing, one would pass completely every second. That's one Hertz. So what they discovered about frequency shift keying.

[01:36:39] Was that you could actually let data instead of analog sound. You can use frequencies to transmit digital. So the trough could be a zero and the peak could be a one. So you can actually put data on those peaks and troughs. And that was what frequency should shift. King was all about. [01:37:00] So then what we learned was that they.

[01:37:06] Lower frequencies couldn't carry near as much data as the higher frequencies. And this is why there's a race to introduce higher and higher frequency. So one Hertz is one oscillation per second, one trough, and one peak passing a 0.1 kilohertz, which you see all the time. I'm sure you see KFC. Uh, I think the KFC, you'll see a M radios, you'll see Hertz, H Z FM radios you'll see in kill a Hertz.

[01:37:33] So one kilohertz is 1000 oscillations per second. Now you're really talking. So you're seeing a thousand peaks and troughs past this point every second. So now if you're transmitting data, wow, that's a thousand bits of data. A second. You see where I'm going with this? Okay? So then we go right to gigahertz.

[01:37:53] Gigahertz is 1 billion data points per [01:38:00] second. Wow. So every second you can transfer 1 billion bits of data. And this is why there's a race to get up above six gigahertz. So five G is six gigahertz. Every second on a frequency, they can send 6 billion bits of data. We talk about bandwidth in the communications business as well as in the community computer business.

[01:38:28] Well, this is now you're really getting up there. So just imagine if you had 24 gigabyte gigabyte gigahertz, right? The top end of low five G. 24 billion bits of data per second. Now you can send high definition video. You can send real time stuff. You no glitches. Nope. I mean, we're talking about opening that pipe up of bandwidth, like a sewer pipe in the street.

[01:38:55] It's huge now. And when they get up to 60 gigahertz, they're saying, [01:39:00] and this is why it was developed. So people like to say, Oh, um. Five G is a weapon. The military uses it and you can use RF. There's no doubt about it. They use it all the way back in the fifties in the sixties they knew that they, if they pointed an antenna with enough frequency, a high and a frequency and enough power coming out of it, they could stunned people with it.

[01:39:20] That's just further evidence that RF does something to us. Right? So just keep that in the back of your mind. But the reason that the military wanted five G is because. They could get pinpoint data from an entire battlefield in real time. They could control drones and make them do. Now you're talking about being able to control thousands and tens of thousands of things and getting tens of thousands of data points back every second.

[01:39:46] This is going to revolutionize a automated warfare. This is why they want five G so, and that's the reason why the cellular industry wants five G. They want it because they'll sell you phones that'll do [01:40:00] stuff that you've never been able to do because they needed more bandwidth. Now let's talk about something separately.

[01:40:06] Let's switch. Switch the tone here for a second. Let's talk about covert 19 so people like to say, Oh, five G is causing inflammation. It possibly is, but covert 19 turns on the immune system to such a degree that we hear about cytokine storms. All the cytokine storm is, is the immune system is overreacting and lighting the body up like fire.

[01:40:31] It's lighting the body up like fire because the immune system has kicked in and it's going to scorch earth. It wants to kill everything at anything. It's not even sure what it wants to kill. The other problem with inflammation is it thickens the blood so. Those of, you know, people take aspirin to thin their blood out, things that are anti-inflammatory, thin, the blood, things that are proinflammatory thick in the blood.

[01:40:55] Stay with me. We're almost done here. We're almost done here. So a lot of people like to say, well, the five G is [01:41:00] causing thickening of the blood. No, it's not. It's not that the coven 19 is causing thickening of the blood because your immune system is kicked into gear and you are literally turning into a.

[01:41:11] Congealed mass because your blood is getting thicker and thicker. Anti-inflammatory thins, the blood. Pro-inflammatory thickens the blood fact, but here's the real. Fact that nobody's paying attention to when they listen to these people on the internet and these people say, yeah, five G is causing covert 19 this is w this, this isn't even critical thinking.

[01:41:36] This is just looking at simple data and asking yourself, could this be true? Currently, 34 countries have five G networks. Almost all of them in the low five G. But yet 150 plus countries have cases of covert 19 and deaths from covert [01:42:00] 19 so if if five G was driving covert 19 we would say 34 countries have covert 19 only the countries that have five G.

[01:42:11] So it's not, it's not logical. It doesn't make sense. Please don't follow this BS narrative. In fact, this narrative is hurting. It's hurting real information. People who are thinking, Oh, you know, my grandmother died from coven 19 it's because of five G it's not because of five G she got the virus. Five G isn't killing people.

[01:42:33] And they're not calling a covert 19 okay, but five G is dangerous. Don't ma, I'm not mixing my words here. Five G is dangerous and we just don't even really know yet what five G is going to do to us. Probably for another 30 or 40 years. We're going to start seeing new high levels of suspicious diseases like idiopathic mitral valve prolapse is like on the rise.

[01:42:56] Nobody understands why. I think it's from RF. I think it's from cell [01:43:00] phones. I do. And who knows, maybe all the gut dysbiosis that we're seeing in the population is from cell phones and not the foods we eat because there are good studies out there that show that RF, the same frequencies we're using in four G right now are causing changes in the landscape of the the gut microbiome.

[01:43:19] But the truth is nobody knows what five G is doing yet. We are the Guinea pigs right here. We are the experiment. However. I can assure you that it's not causing covert 19 so please don't pass that silly information around. That is your lesson today about RF. I hope you got something good out of it, and you can talk intelligently to people who say, I think, I think RF is causing covert 19 I think five G is doing it.

[01:43:48] Just ask them, well, there's only 34 countries that have a five G network up, but there's 150 plus countries that have. This virus. How do you explain that? [01:44:00] Believe me, they'll go searching for some BS explanation if they want to stick to their guns, but just don't buy it. I look, that's it for today. I'm off tomorrow.

[01:44:08] We'll see a Monday with more super Yuma radio. Please share the show. If you think you learned something today, please share the show and help someone else learn too. We'll see you next week. [01:45:00]



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Super Human Radio is the world's longest running broadcast dedicated to health, fitness & anti-aging with an emphasis on exercise, nutrition, and hormone management. This one of the most progressive podcasts for preventative & regenerative techniques designed to increase longevity. More

2908 Brownsboro Rd Ste 103
Louisville, Kentucky 40206

(502)-690-2200

SHR Logo

Super Human Radio is the world's longest running broadcast dedicated to fitness, health, and anti-aging with emphasis on exercise, nutrition, and hormone management. The most progressive source of information for preventative & regenerative techniques... More

2908 Brownsboro Rd Ste 103
Louisville, Kentucky 40206
United States of America

+1 502-690-2200