[00:00:00] Carl Lanore: [00:00:00] welcome back to another episode of super human radio today is, uh, July 8th. Uh, and that is, uh, 2020. And today's show is very, should be very interesting to most people and that is can attest predict when you're going to die. And, uh, we're going to discuss this with real serious. Um, implications and also look at the capabilities of testing today, which is changing dramatically right before our eyes.

[00:00:32] Very, very exciting stuff. Before we get started with that, I have to remind people that my title sponsor is legendary foods and, uh, they make the best seasoned nuts in the world. I kid you not. In fact, um, they have pizza flavor, tangy ranch, uh, bacon cheddar, not only. Is the seasoning so delicious, but they use the freshest almonds I have ever eaten.

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[00:02:04] So we have a, let me just remove that. We are joined by dr. Daniel Elias. Martine had bronze from chronometer. And dr.  hi Dan, to be back. And then of course, dr. Williams seeds, who literally just stepped out of the Orr, right? Literally you would just in the O R a little while ago. Yes. Okay. So dr. Seeds is a real, he's a real surgeon.

[00:02:31] He's not somebody who used to be a surgeon. Just want to put that out there. So anyway, good, good discussion here today. So we're doing this for two reasons. Number one, it's amazing how, uh, the dissemination of new information takes so slow, uh, for people to pick up that the early adopters need to hear things over and over again.

[00:02:53] So some of you may have heard this already, but we're doing it for a reason. Because not enough people really understand what [00:03:00] epigenetic testing is and what it's capable of doing. And so, you know, everybody thinks epigenetic testing is just genetic testing. Oh, I got my 23 and me already. I'm I'm done. I really don't need anything else.

[00:03:14] Um, But it is very, very different because it allows you to see what your genes will do in different environmental exposures and conditions. So let's start with this first, what exactly is epigenetic testing and how's it different than genetic testing? I'll go to

[00:03:33] Dr. Daniel Elias Martin Herranz: [00:03:33] the, no, that's a, that's a fundamental question.

[00:03:35] Call. Uh, thanks so much for, for the intro. Um, I think, yeah, people, as you said, I've heard a lot about genetics, right. And you know, people are familiar with the fact that genetics is something that is in there in the raw sequence of our DNA made of, you know, ATCs and GS Malian. So for them, and we inherited this information from our parents, uh, and this predisposes us to, you know, certain, [00:04:00] uh, conditions or, you know, certain traits like the color of our eyes.

[00:04:03] Uh, but the problem with that is obviously that the set of genes. That we inherited. It's not the entire picture. Um, we all know that, you know, as you said, lifestyle, environmental factors shape our health over time. And therefore, if we really want to understand, you know, what is our status of health right now understand our risk factors for different diseases.

[00:04:23] We need to capture this component of, you know, how our genes are being turned on or off by this different lifestyle and environmental factors over time. And this is really what, what epigenetics can do. So. Um, it'd be genetics is essentially the way that, uh, you know, our DNA is wrapped around and therefore how this genes are turned on or off at a specific time point.

[00:04:47] And there are different types of epigenetic marks, and I'm sure we'll, we'll go into details into this. Um, But basically they, the key take home message is that, you know, by quantifying epigenetics, as opposed to only the genetic information, [00:05:00] we can also capture oldest influences of lifestyle, environmental factors, which are fundamental for most complex diseases.

[00:05:08] Carl Lanore: [00:05:08] Dr. Seeds. So with the emergence of epigenetics and the science, it turns out that while we think our genes are the blueprint. It's highly influenceable. So if you, if you think you're building a house, you have a blueprint, the wall's going to go here and they follow the blueprint specifically. And the house turns out exactly as you intended it to, but let's say one day the brick layers came in and somebody made a wrong measurement and the wall is too far to the left.

[00:05:39] And now you have. An unwanted change, which we could also equate to disease state. Is that oversimplifying what epigenetics is, being able to identify where the wall was built in the wrong place to then reverse that mistake?

[00:05:55] Dr. William Seeds, MD: [00:05:55] No, I think that's a really, that's a great analogy too, for people to [00:06:00] understand what you just said is very important.

[00:06:03] That the epigenome, the genome is not reversible. The epigenome is reversible, meaning you can, you can do things to improve the changes that have been made that may be disadvantaged to the cell. So understanding that that would, uh, Danny did so well and telling everyone that the epigenome is influenced is influencing the genome.

[00:06:32] Too, so to speak, change the messaging of the cell, and that can be environmental. It can be nutrition, it can be exercise. It can be everything that can influence that. So your analogy was excellent. Uh, because you can go back now, it's going to be more work, right? It's going to be more work to go back and rebuild that wall.

[00:06:57] Um, but it can be done and. [00:07:00] I think that's the value in understanding this, where we can go with this type of science today to really measure and at, as a Danny also. So, so, well, uh, articulate the digital twin type of a scenario. You know, we can take that digital twin and really look at how we can make changes.

[00:07:28] Uh, that are influenced by changing our influences on the epigenome like alcohol exercise. Uh,

[00:07:39] Carl Lanore: [00:07:39] what about stress? Does the stress actually, we hear stress kills, but is that an intrinsic F effect from the onset of stress? Does it actually start to have epigenetic, uh, effects on our genes?

[00:07:57] Dr. Daniel Elias Martin Herranz: [00:07:57] Yeah, absolutely.

[00:07:58] There, there are [00:08:00] different studies that have done this. Um, basically looking at, you know, the epigenetic layer of information, measuring epigenetics for people that have been more or less stressed also at different periods during their lives. So there are periods where in our lives that we're, you know, more sensible to, to this, uh, for example, like traumatic experiences or, you know, different levels of stress such as, you know, when we're growing up.

[00:08:22] Um, and people have identified this, the signatures, and we know that they, you know, they are at least correlated with, with the chances of developing, uh, certain diseases, um, in the medium unlocked longterm. And you know, the reason why. Really epigenetics is picking this up is because, you know, when under normal circumstances, when we have different stressors coming to our body in our body mounts, a fight or flight response, some people call it to deal with those stressors.

[00:08:50] And if that is resolved quickly, it's a normal physiological process. But the problem is that when that is repeated, that continued a lot over time that can lead to damage in our [00:09:00] bodies due to this chronic stress. And that is what. And stop, you know, through hormones, through all their different processes, uh, altering the epigenetic state of, of some of our tissues and selves.

[00:09:12] Carl Lanore: [00:09:12] So dr. Seeds, it seems to me that, um, I'm sorry, go ahead. And then I have a question for you.

[00:09:18] Dr. William Seeds, MD: [00:09:18] I was just going to add to what Danny said is that we know in the cell that the inflamma zone, which is a receptor to environmental or nutritional or metabolic changes. Is actually is controlled by damage associated molecular patterns, which is a, a name for, and we have classified this, that there are receptors for stress itself and how it affects that Inflammatone and then how that carries over into changes in transcription in the cell that will produce these pro-inflammatory type of, uh, Uh, cytokines and chemo, kines.

[00:09:58] And [00:10:00] proteases that then as Danny said, over a period of time can have an effect on the, uh, on methylation issues on a satellite station. Um, semination, ubiquitination all these things that can happen. And that could be post-translational changes or chemical changes on the DNA, but mostly what we follow and look at are the methylation changes in the DNA.

[00:10:23] Carl Lanore: [00:10:23] Now, now, is it fair to say that COVID is having an effect on epigenetics because of the lack of. Ability to train at the gym, uh, being more sedentary, but also the stress of not knowing if you're going to get it and die. It wouldn't be a fair statement to say that we would probably see epigenetic changes if we tested somebody before the lockdown and after the lockdown, who wants to grab that?

[00:10:49] Dr. William Seeds, MD: [00:10:49] Well, I would say it just depends on where that person was. You know, they were at a tipping point and this was the, the added stressor that, that. Got that [00:11:00] level of where we're talking, where I just talked about the cellular changes that need to continue to, to actually work those changes. Absolutely. Um, it just depends on where that person is.

[00:11:12] Can't it can also be a, another part of that whole process of, of stress, which some people talk about as an Alice static load. They like

[00:11:23] Carl Lanore: [00:11:23] to use that word the other day.

[00:11:27] Dr. William Seeds, MD: [00:11:27] It's just the combination of everything together and still absolutely. Carl, I mean, of course.

[00:11:35] Carl Lanore: [00:11:35] So let's talk about what  actually does because I think the fascinating thing is there seems to be, uh, several, um, places now where you can get quote, unquote, epigenetic testing done.

[00:11:47] But I wanted to talk about what Kronos mix does because it's very, very different than what other groups do, right.

[00:11:56] Dr. Daniel Elias Martin Herranz: [00:11:56] Yeah, that's right. Um, I think the key differentiator of [00:12:00] what we do is that, you know, we're really pushing the limits of what is possible with epigenetic testing. And to do this, you need two things.

[00:12:08] So, first of all, you need a new technology or the latest technology to quantify as much as you can from the epi genome. So obviously we mentioned that there are millions of different places in your DNA that basically can get this chemical toxins Metfield tax attached or detached from them. And this is what affects in turn, uh, the regulation of, of your genes.

[00:12:30] So we quantify those, those Smithfield groups, those chemical tags. In more than 20 million positions in your DNA. So this is obviously a super comprehensive and a huge, uh, data set and a snapshot that we're taking from Europe, between them. Right on. If you compare this, uh, with, with other technologies, um, then, you know, in the latest versions of flux technologies, you normally measure around 850,000 positions.

[00:12:54] So. From the point of view of the, of the type of data that we're collecting, uh, you know, [00:13:00] w we've gone to use the latest technology, which is known as next year, some sequencing. Um, and then obviously what that means is that then you have a lot of data to see many different things that are going on in the FPT numb, right on the idea of what we want to do is essentially to take those epigenetic signatures and relate them to this different, uh, risk factors that we were, uh, discussing things, success, you know, the aging process, uh, which has quantified your biological age, uh, your exposure to different toxins.

[00:13:28] Like the ones that you find the smoke from tobacco or air pollution. Uh, how drinking over the years has affected you, um, how your metabolism is working, uh, uh, stress, all this different things. Uh, we'll leave this as specific epigenetic signatures that because we get this super wide picture or snapshot of, of your epi genome.

[00:13:48] And as dr. Seats says, um, you know, building a slowly your, your digital twin, uh, applying machine learning, we can derive. Oldest different measurements.

[00:13:57] Carl Lanore: [00:13:57] So there's one other thing I want to mention [00:14:00] and why we're doing this show now. So many of you heard the show originally months and months ago, and where we just talked about the chronometer test by itself.

[00:14:10] And my audience got an, a ridiculous 70% off this test to, to buy it direct so they can assess their own chronological age versus their biological age. And we had a lot of people respond. But there are still some tests left in the number that we allotted for this promotion. This once, once in a lifetime promotion, these tests will never be 70% off again.

[00:14:34] And in fact, in a short period of time, probably within a month of this show, airing, this offer will be retracted and no one will be able to get this test on their own at this. Just ridiculously inexpensive price. And this is such a valuable test. And we're going to talk about why this may actually be the future of all testing done, because let's face it.

[00:14:57] You can look at all this blood work and, but it doesn't predict [00:15:00] what is a bad actor in your life. This does. That's the big difference. If you go to the website seeds.md/epigenetic hyphen test slash put the third, the second slash audit, please you'll get saved. 70% on this test in the future. It will only be allowed available through clinicians and they will never offer you this price here to get it done.

[00:15:24] So if you've been sitting on the fence and thinking, you know, I keep hearing that ad run and I do want to do that. I do want the clock is ticking and this is not a high pressure thing that  needs to move to the next phase of their. They're marketing in the United States. And this offer to my audience is, is stopping things from happening.

[00:15:42] So about 30 days to today is July eight 30 days from now. You probably won't be able to get this offer anymore. So again, Seeds that MD slash epigenetic hyphen test slash for 70% off. All right. So now let's, let's talk a little bit about methylation for a second. W you know what [00:16:00] I do know about science is this what we think we know often turns out not to be what we know.

[00:16:05] So right now we think that methylation DNA methylation is everything. Uh, when it comes to changing that blueprint, that leads to the onset of diseases. Is it, is there any evidence that there's any unknown areas?

[00:16:20] Dr. Daniel Elias Martin Herranz: [00:16:20] Yeah, I think, I mean, obviously the reason why, you know, we have started and decided to choose DNA methylation as, as the place that we wanted to look at for, for data to build this different predictors for your health.

[00:16:33] Uh, you know, it's informed of, uh, part of the work that we did during our PhD. So the university of Cambridge. Uh, but also following on from, from there. And, uh, just to be clear, you know, we have a bioinformatics background, which means that we're very unbiased about the data types that we use. Right. And we had seen that, you know, with, with blood biomarkers, we could get to the same place.

[00:16:55] Uh, we would have chosen that first, um, instead of, of DNA methylation. Right. And [00:17:00] obviously the more data that you have is always the better, but our thinking is that you measure this. Uh, important risk factors for DCS, uh, such as the aging process. Um, it is clear that DNA methylation is the best data type to date, uh, to ask these questions.

[00:17:17] And the reason for that is very simple. And it's that, you know, if you look at the hierarchy of the cell, um, you know, we go from your genetic information, which is in your, in your DNA, then the epigenetic layer of information such as DNA methylation. Then this, this gets, you know, produce like the expression of your genes, which is known as RNA.

[00:17:37] And then that goes to proteins, which start to carry out functions. And then that goes to potentially metabolites like cholesterol, for example. So if you only measure at the top of the hierarchy, the DNA, that's, that's a static. If you only look at the genetic information, but the next layer of information that is controlling, what is below all of that, and that is controlling and integrating the effects of.

[00:17:58] Genetics and environment [00:18:00] is that it'd be genetically Europe information. So that's the reason why you'd capture so well. It's very complex processes that are happening now where, you know, we're still and tissues. And it's not only that theoretically that's the case is that there are now thousands of studies that have shown in large human populations that, you know, this is the best way to quantify things such as the aging process and how the front left side environmental factors are shaping.

[00:18:25] Our health over time. So, uh, I guess that's, that's the reason why we get so excited about, you know, what DNA methylation can do and what it will also be able to do in the, in the next months and years,

[00:18:36] Carl Lanore: [00:18:36] dr. Seeds. So is this technology reliable enough to predict or project a person's lifespan? If, if things are effected by epigenetics, Then we're still looking at a snapshot in time of your world, interacting with you that could change.

[00:18:58] And that would change the trajectory [00:19:00] of the balance of your life span. Would it not?

[00:19:03] Dr. William Seeds, MD: [00:19:03] Yes. Um, and, and it is that's, that's where this is going. That's the accuracy of this is I think what is the most intriguing part of being able to put a lot of factors together because as we know. It takes a lot of there's a lot of players that make something happen.

[00:19:24] It's like teamwork, a cell works because of a team of an orchestra, not a single instrument. The more we have input from all of those pieces of the orchestra in looking at what's going to come out as the music per se, we can predict. Based on that we can predict where that care is going to go with how we change each instrument or how we change the pitch of an instrument or how we change the input into the instrument.

[00:20:00] [00:20:00] I mean, those are personal type of aspects of looking at medicine that we've never had before. And when you can do that and see an outcome. Of a cell, but can also predict that when it's a predictable outcome, based on those changes, because of all the data you have, uh, that's the future, Carl, that is the future.

[00:20:25] And there's no doubt in my mind, that's where we are going. And that's what makes it so exciting because we deserve to be there because now we have that ability to gather this type of information.

[00:20:40] Carl Lanore: [00:20:40] So really what, where other tests are designed or are used maybe inappropriately, um, to take a snapshot in time, not of how your body is reacting to your environment, but instead these non-sequitur bits of information that don't come [00:21:00] together and play nicely to make any kind of predictions that the, the epigenetic chess, it sounds like will evolve to be something that people do.

[00:21:08] A few times a year to make sure that they're not introducing things that they didn't use before that are affecting their epigenome, uh, or that the desired effects of their current lifestyle are working out predictably. Uh, so where, where does, where does the concept of human lifespan come into this discussion?

[00:21:28] Do you think that epigenetic testing with on regular, on a regular basis. Could actually change our opinion of what we think the human lifespan is. I mean, right now it's pretty, it's pretty agreed by everybody with any sensibility. That's about 120 years. And even with that, we lost dr. Seeds. I don't know what happened.

[00:21:48] Let me get he disconnected for a second. Okay. Even with that, even with the 120 years, um, it's really not, uh, Hard and true because very few people [00:22:00] actually live to be 120 years old. You know, we think that that's the most, because most people die at 117 will epigenetic testing give us guided lifestyle information that actually will change our opinion of what the life of the human lifespan is.

[00:22:17] Dr. Daniel Elias Martin Herranz: [00:22:17] Yeah, no, I think that's, that's a really great question, Kyle. And he boasted a quarter off of what all of this is about. Right. I think first it's important to make the distinction between. Um, life span and health is fine, right? So life is fine. Is that the maximum amount of time that, that you live basically?

[00:22:34] Uh, but no one wants to leave 20 years more with a horrible chronic disease. Right? So what we really want to push is, is health span, which is that the time that we are free of disease, and there's some relation between, between those two. So sometimes, you know, people that live really long, like supercentenarians or centenarians.

[00:22:53] Um, you know, tend to tend to develop this, um, you know, diseases later in life. Uh, but again, [00:23:00] it's, it's still under study, but the key thing here is that, you know, we need a way, a reliable way to tell us a test, to tell us if you know, the different changes that we are doing to our lifestyle, our environment, you know, the supplements that were taken, potentially some, some drugs, uh, you know, whether that is really putting us into the right truck.

[00:23:20] And to be able to do that, you need a reliable way to measure things such as the aging process. So your age associated risk your risk of focused mortality on, you know, these are things that until a few years ago were completely impossible to measure awkwardly in humans. So this is a very new technology and you know, now it's got to the point where we can actually do exactly what you said.

[00:23:42] We have enough. Temporal resolution, uh, for people to go on and take a specific interventions and measured themselves before they take that intervention. And then after that intervention on objectively at the molecular level, determine whether they have managed to slow down the rate at which they are [00:24:00] aging or even potentially reverse it.

[00:24:02] Um, so, you know, it's very exciting times because for the first time we have actually the tools. To to answer this and you don't need to wait until people develop diseases later on, or you don't need to wait until they die. You can tell today. Where they are heading in terms of their aging process. And I know they're complex risk factors and act on them.

[00:24:22] Uh, and that's, that's really the exciting thing about, I think every genetic testing,

[00:24:26] Carl Lanore: [00:24:26] if a budget was not an issue, how frequently should someone test their epigenetics to see, like how long does it take for the, the changes in lifestyle to actually show up? So is it, is it twice a year? Is it four times a year?

[00:24:39] What do you think.

[00:24:41] Dr. Daniel Elias Martin Herranz: [00:24:41] Yeah. So interestingly enough, we have epigenetic changes that happen even in our circadian minor. So, you know, depending on, on the time of the day that we are in a, you know, you have changes in your epigenetic profile as well. Uh, but most of the changes, at least to what we know nowadays that are important for your health in the medium and [00:25:00] longterm, which is what this is really about.

[00:25:02] Um, they will happen in the order of, uh, you know, months towards a year. Um, so for example, things such as the aging process, uh, you know, for most of the interventions that people will do, uh, you know, testing twice a year or once a year, it's normally, you know what we recommend because before that it's difficult to, to farm, you know, a huge impact on your biological age, but things that says your metabolic risk or, you know, your exposure to these different toxins, et cetera, they're, you can see changes much faster.

[00:25:31] Um, and the exciting thing is that now you have final objective way to, to quantify these things. Uh, so it really depends on what's the goal of, of your interventions and you know, how much you stick to them and what they are, uh, by. Yeah. We envisioned that people will be, you know, people are already doing this already, uh, every few months, uh, and maybe one day we do it, you know, every day from our homes.

[00:25:54] Um, So, yeah, I think it's, it's very exciting too, to see where all this develops as well.

[00:25:59] Carl Lanore: [00:25:59] We're going to take a quick [00:26:00] commercial break. When we come back, we got lots more to talk about. We're discussing the epigenetic test emergence in science and what it actually potentially could mean for all of us. I'm very interested in this because I want to live longer.

[00:26:12] I'm waiting for my results to come back. It takes a long time. This is not. Like the 23andme test. This takes some time that they're testing 20 million different or 20 million plus different points of your DNA. And it's a saliva test, which means that you don't have to go bleed somewhere. You don't have to make yourself bleed at home.

[00:26:31] It's an ultra ultra simple. Uh, so let's do this. We'll take a quick commercial break. We'll tell you how you can take advantage of this unbelievable 70% off for a limited time. Stager is this superhuman chat.

[00:26:47] welcome back to supreme-a radio. Uh, for those of you watching live or, uh, listening live or whatever, uh, the place to go, if you want to take advantage of this, and this is the last time I'm going to give a during the show, [00:27:00] if you want to gauge your own. Chronological age against your biological age. If you go to the website seeds, S E D s.md/epigenetic hyphen test, you'll save 70% off of this test for limited time.

[00:27:14] This is it. We're pulling the plug on this most likely before the Oh, by the, uh, uh, this time next month. So today is July 8th and, um, a lot of people have already taken advantage of it. And I have, and I'm eagerly waiting to see not only where I am now, but the targets of change that I will be focusing on because most of us approach our health in a Willy nilly fashion.

[00:27:38] We try this, we read an article and then we try that and we read this and we try this, or we remove that and we never know what's really working and what's not. So just having. That information of the things that erode your health versus the things that build your health. That's like invaluable. That's so much wonderful information.

[00:27:59] So we have [00:28:00] a couple of things here. Uh, let's see here. So Brett also has a question. We'll put it up here. He says contests predict mental illness, which accounts for a decent percentage of the population. What do you think dr. Danny?

[00:28:12] Dr. Daniel Elias Martin Herranz: [00:28:12] Yeah, no, that's, that's a fantastic question. Um, I think, you know, there, there are many studies now, uh, pointing more and more towards the fact that, you know, obviously the epigenetics, uh, may play a role in, in different mental illnesses and, and the CCS and, uh, spread correctly medicines.

[00:28:30] It's becoming unfortunately more and more common in, in our populations. And, um, you know, what we do at the moment. Is to quantify risk factor for risk factors for mental illnesses. Right? So one of, one of the areas that we're really interested in is, for example, in quantifying, uh, I stress, we know that, you know, things like stress and onsite to really have, you know, a huge team back, for example, your risk of developing things like depression.

[00:28:55] So, um, you know, we are developing those, those [00:29:00] capabilities with genetic testing, uh, and we hope to, to make them available. Uh, very soon to, to our users. And I think the take home message here. Uh, just going back to what you, what we spoke before is the fact that, you know, because of the data that we're capturing before for you right now, even, uh, you know, in a few years time, you will be able to go back and look at that data and get many more insights from them.

[00:29:28] Uh, so as we, for example, discover new signatures that are associated with a specific. Uh, you know, mental illnesses, you would be able to

[00:29:35] Carl Lanore: [00:29:35] compare that data to yours. You have to overlay that onto yours. Dr. Seeds, are you using epigenetics now in the, in the instances of people who develop osteoarthritis or other types of muscle, uh, uh, musculoskeletal issues that you treat in your practice, are you starting to see connecting the dots of any epigenetic causes and things like that?

[00:29:58] Dr. William Seeds, MD: [00:29:58] Well, that's, that's [00:30:00] why I'm involved on the research side. That's where this is going right now. So I'm, that's, those are the type of, of, um, uh, of studies that we're looking at in particular, uh, to osteopenia osteoporosis, uh, uh, degenerative disease, um, and, uh, And from there, uh, other type of, uh, repair, uh, scenarios with a tendon ligament issues.

[00:30:31] So we're in the process of actually setting all those studies up presently, uh, to, you know, to manifest. And show that how powerful these markers are. So we can start validating. I mean, the whole, the key to this is really invalidating the therapies we have to treat disease and injury. It's a, this is the validation for all of that.

[00:30:58] And that's where [00:31:00] the power of this lies because we can definitively go in. And for instance, um, you know, I I've always been in the, again, this is just my opinion, but I've always been one of the opinion that people take way too many supplements and antioxidants, and they don't realize that there are finite times to take those and that taking them every day for a entire year is not in their best interest because it works against the cell actually, because you need that signaling, you need.

[00:31:35] Oxidant signaling to make things happen. So all of this type of testing that, uh, that we're describing is going to change the landscape of how everybody looks at this. It will completely change it because it's re it's predictable and reproducible and shows you definitively. Treatment protocols and the changes they [00:32:00] make.

[00:32:00] I mean, that's, I just, I can't tell you how exciting that is to be involved in that and to know where we're heading in looking at that and being able to take away. So many of these myths that are, I think are out there in regenerative medicine and in, in the health world that we can, we can totally nullify in.

[00:32:22] In no time. And I think that's the power of this process.

[00:32:26] Carl Lanore: [00:32:26] So w where

[00:32:28] Dr. Daniel Elias Martin Herranz: [00:32:28] maybe we come into that. Um, I think it's very clear when you explain this, for example, in the terms of, in terms of diseases, right? So when people get diagnosed at VCs, they get given a treatment and there are objective ways to quantify and see whether that treatment is working or not.

[00:32:46] Uh, but surprisingly enough, we don't have. That many ways of doing this before you develop the, so we don't have many ways to quantify pre disease or health estates on on, before we get to the point where we develop a cancer or type two diabetes, [00:33:00] have objective markers that will tell us, okay, you know, you, this new treatment that you're doing or this new lifestyle intervention, or, you know, potentially a new supplement, even dr.

[00:33:10] Seats, that's the thing. They, they, they worked out well. I mean, I agree I'm from diet. From that side of things. So, you know, maybe, uh, keeping it well researched and under control rather than taking everything that is out there. Uh, but the key thing about this is that you can objectively determine if it's working for you.

[00:33:28] Right. I'm in my will be true that some of those things work for other people, but not, not for you particularly. Uh, and now you have a way to see if that's the case, instead of just relying on population-based advice.

[00:33:40] Carl Lanore: [00:33:40] Right. Right. And I, and I'm a firm believer now at this stage of my life, that it's not what you introduce, but what you remove from your environment, your food, your supplements, your life.

[00:33:54] That has the greatest impact on you being healthy and strong. And so, I mean, I know it's true. I know what [00:34:00] should we see people with? Auto-immunity they take drugs that, that happens. And all of a sudden they removed this one thing from their diet and their, their ulcerative colitis just goes away in four months.

[00:34:08] It's like, wait a minute. So, you know, Ron Penna is the one who taught me this health is about subtracting, not adding when you come to the, uh, when you come to that knowledge. So what are the areas of. The unknown and blind spots that may provide some of the greatest breakthroughs. I think dr. Seeds, you really, you, you kind of indicated this on a, on a wide scale, but when, when you look at data, you're seeing emerging trends in our population, what do you, what do you see in the future that people would be surprised that epigenetic testing will actually have a great role in correcting a specific disease States?

[00:34:52] Danny.

[00:34:54] Dr. Daniel Elias Martin Herranz: [00:34:54] Yeah. So I think there, there are many areas that, you know, are our own folding, as [00:35:00] we speak in, in epigenetics research. And, you know, at crunch comics, we really are being part of, of that revolution and pushing the boundaries of what we can get from epigenetic data. And I think before we discuss, I think one of the areas that in my opinion is super interesting, which is.

[00:35:15] Uh, you know, mental health. And I think we know very little about mental health. Uh, most of the biomarkers that we have are not molecular, so they don't quantify specific molecules or things in yourselves. They, you know, are based on questionnaires or, you know, if you get a bit funnier, maybe in some cases, MRI, um, by, you know, most of the tools that we have are.

[00:35:36] You know, we can, we can improve them. And I think it'd be genetics because of the nature of the data. Um, I think it will, you know, it will help in that, in that revolution, regional health. And I think, yeah. Yeah. And I think, you know, besides that, obviously we've discussed all the things that it can do in preventative healthcare.

[00:35:54] Um, but I think another key thing is how we, uh, start to integrate this data as [00:36:00] well with, with other types of data. Right. And, and how this different parts of the puzzle. Uh, come together on how we want to fight them over time, uh, on how we derive actionable insights from them. And that's, that's very much a, you know, the vision that is behind kernel makes and it's even forge in the name of the company itself.

[00:36:21] Carl Lanore: [00:36:21] Dr. Seed's fibromyalgia is a big problem today as is Lyme disease. Do you think that epigenetic testing may give us insights into maybe not ways to cure it, but to stop it from progressing?

[00:36:37] Dr. William Seeds, MD: [00:36:37] Well, absolutely. Just as I said, in treatment and treatment modalities of, you know, I, I think this is going to get, as Danny knows, there, there are papers out there that are looking at the.

[00:36:50] That are getting so specific that we can look at the epigenome of just a STEM cell and what a STEM cell can do and how a STEM cell changes. And the [00:37:00] STEM cell is all about repairing injury, whether it's the brain, whether it's the heart. Um, it's an indicator of where the body is in, um, in age. In repair in basically everything and how it rejuvenates and how it repopulates.

[00:37:21] So getting that type of data and understanding how, what the epigenome is of a STEM cell, everything is gonna, everything's gonna fall into place in my opinion, when that happens, because then you've got, you really can look at w you know what Danny was just talking about the brain, uh, in health, because, you know, we know that.

[00:37:45] STEM cells are very important in repopulation and continuing the plasticity of the right. And yeah, it's absolute. And in the diseases you've just described, there is an inherent problem with STEM cell [00:38:00] repair and in any auto immune disease or any. 

[00:38:05] Carl Lanore: [00:38:05] see what you just said. So this is where the rubber meets the road.

[00:38:08] There's a large number of doctors out there telling their fibro patients that this is not an autoimmune disorder. And then there are a large number of physicians telling their patients that this is an autoimmune disorder. And I kind of feel like epigenetic testing would put that argument to the rest because once you looked at this person's epigenome and you realize.

[00:38:30] What is going wrong with them on a core level, on a cellular level, then by removing certain bad actors that we already know, cause some of these downstream effects from their diet, we should see them get better. And if they do, then we can stop this debate whether or not fibromyalgia is, is a, is a, uh, an autoimmune disorder or not.

[00:38:51] That's what I'm, I don't know.

[00:38:53] Dr. William Seeds, MD: [00:38:53] I'm not sure if there's a debate. I mean, we know the auto immune systems and intricately involved in. [00:39:00] Any in any disorder. I mean, it's, that's the bottom line. There is you name the disease and I'll show you the auto immune problem it's there. And we have the genetic, we have the genetic markers for that also in, in the, in, in the dysfunction of, you know, a specific type of, uh, cell signaling or, or receptors.

[00:39:19] So I don't know about, I would say that the people haven't. They're, they're not on the cutting edge of where research is right now because the epigenome is about this. And it's about everything in repair.

[00:39:33] Carl Lanore: [00:39:33] Right? I want to take our last commercial break in this interview. Then later on in the show, I have some things I want to talk about too, but we're going to take one commercial break, stay tuned evolution just got kicked up a notch.

[00:39:49] Welcome back. If you haven't heard already. You can get one of these epigenetic tests that literally 70% off. You'll see that if you go to the website, if you go to [00:40:00] seeds.md forward slash epigenetic hyphen test and add the last four, which let's just be on the safe side, you can order the test. They're shipped here from the UK.

[00:40:11] Um, the test is very simple. It's a saliva test and this is a big deal to me because. I remember when I used to do the ZRT blood tests, you know, I didn't want to prick my finger, who the hell wants to bleed. I mean, bleeding, isn't something that we all look forward to doing. And, and even worse than that, if you have to literally go to a, a lab in your neighborhood, To give blood for a test that's not convenient at all.

[00:40:34] The thing that I really love. I mean, the idea that this test is next generation so far advanced, uh, as opposed to anything else out there, but the fact that it's a saliva test, it's like there's there's no, no, no. I mean, it's so easy to get involved. You order the test, you spit in the bile, you send it out.

[00:40:52] Bam, it's all done. And the valuable information that you get could actually help you target. Disease reversal [00:41:00] most likely I'm hope hoping it does. So I got a question from a listener who wants to remain and remain anonymous. Can the chronometer data be used to create genealogy profiles? Hmm.

[00:41:16] Dr. Daniel Elias Martin Herranz: [00:41:16] Yeah. So genealogy profiles are normally, uh, done using more genetic information.

[00:41:22] Um, so that's not something that we focus a lot of time on, but having said that. Uh, the raw data that we are capturing contains both genetic and epigenetic information. Uh, so theoretically it would be possible if we have some of their, you know, makes users want to want to access that role data. They can, they can download it.

[00:41:43] And then, uh, I'm sure, you know, there will be some people out there that might be able to do that with, with that role, uh, data as well.

[00:41:52] Carl Lanore: [00:41:52] Dr. Seeds, if someone has this done. Do they get a lot of the same data they would get from 23 and me as well on [00:42:00] top of the fact that they're getting an epigenome test as well.

[00:42:04] Dr. William Seeds, MD: [00:42:04] No, this is really based more on the epigenome and specifically what, how it's influencing the, um, biological age. It's, it's specific to that. And to the other factors of what's being looked at like how exercise influences, how alcohol influences. How does the environment like smoking influence and, and again, this next, the next step of stress and how does that affect the epigenome?

[00:42:34] So it's really the epigenome, again, is something that's separate from the genome. The genome is, so we've learned, you know, that we started looking at the genome and we thought we were of the impression that by understanding the genome. Then we would be able to influence disease and how to treat it. And we, we, we quickly learned that that's not the case and [00:43:00] unless.

[00:43:00] You have one of those rare inheritable type of diseases, uh, that then you, you're better equipped, you know, to go after, but there's very, very few, uh, uh, it's a very low percentage. When you look at the general population of what is, you know, what is the, the real good of this information? Um, you know, how valuable it can it be for you?

[00:43:23] And my opinion of that is that it's not the genome, it's the epigenome, because that's what influences the changes in the cell that can take place. So that's, that's my short answer for that.

[00:43:36] Carl Lanore: [00:43:36] Yeah. Um, do you think that epigenetic testing will ever give insights into the ability to lose body fat or build muscle Daniel?

[00:43:48] The cause that's an area he's interested in. Well as well. I'm sure.

[00:43:54] Dr. Daniel Elias Martin Herranz: [00:43:54] Yeah. So obviously we already measure some aspects of this. So we, as part of the epigenetic tests that [00:44:00] we do, we have what we call the metabolic status. Uh, epigenetic indicator or biomarker on what this does is that it measures metabolic risk.

[00:44:08] And obviously metabolism is something that is quite complex. It has to do with, for example, our depository levels, uh, but also with how your insulin is being regulated, whether you're developing insulin resistance or not, or even like how your immune system is working. Are you developing inflammation?

[00:44:24] Because you've been for example, abuse for a long time, and these are all factors that contribute to. You know, the CCS like type two diabetes or, or metabolic syndrome. Um, so as part of that, this metabolic status indicator is Sable to, to provide with a barrier listic view on, on your metabolism. Uh, and therefore it's currently being used, for example, to assess, uh, whether specific weight loss interventions, or, you know, specific metabolic programs are work into to improve the health of people.

[00:44:54] And, you know, if you're familiar with like, for example, weight loss programs, um, This, you know, this Jojo [00:45:00] effect is something which is pretty common, which is like people lose weight, but then the metabolism takes a bit to catch up with that. Right. Um, so you know, the metabolic status indicator, uh, picks up better the state of metabolism rather than something, you know, that is only looking at a very specific aspects of it, which would be your, your weight

[00:45:19] Carl Lanore: [00:45:19] doctor seeds.

[00:45:20] What do you think about anibolism? What do you think can, can someone who's trying to build muscle. By understanding the epigenome have greater success and building muscle.

[00:45:31] Dr. William Seeds, MD: [00:45:31] Absolutely. Uh, and I think that's a, that's a great, you know, we know about responders and non-responders and how people reach plateaus, um, that, you know, that you're very familiar with Carla and in different types of training methods.

[00:45:49] And this is where I think from that side of it, of the, um, Of the physical culture, as you want to call it, that is going to be [00:46:00] tremendously valuable information and how we look at health. Uh, uh, high-intensity uh, aerobics influences pre-training before resistance training. You know, there is all these questions that like, like I've that are just, I'm just dying to answer where I get into these debates about, well, you know, there's this hit upregulate, that STEM cell.

[00:46:24] So when you do your resistance training, it is smarter to do it right after your hit, as opposed to people do hit after. You know, there are strength training, all of those issues like that. We're so familiar with, Oh my gosh, Karl it's, it's going to be a, it's going to be amazing of, you know, where we can go and start defining the type of, uh, we'll be looking at this.

[00:46:49] I, I can't even imagine actually where it will be, but I know we'll be looking at how we train in. It's totally different fashion and, and, um, And [00:47:00] it's going to be, you know, when you have that type of information, it's even more of an incentive to change. Right?

[00:47:06] Carl Lanore: [00:47:06] Right. That's a given yes. People, the reason people don't have success is they're doing something and they're really not sure if it's what they should be doing.

[00:47:15] And then if they don't see outward change as quickly, they hop to something else or they abandoned this. And if you knew you are like, It's like, it's the difference between leaving here and going to California without a map or leaving here and going California with, with the map without a map, you're not sure if you're really going in the right direction.

[00:47:31] So you're kind of half hearted trudging along. But if you knew that this is absolutely the right way, use zip, you go right to it. And you, you live with whatever the experiences are in the journey because you know, ultimately you're going the right way. Yeah.

[00:47:46] Dr. William Seeds, MD: [00:47:46] And how many. So how many different programs are there out there for instance, to improve your bench?

[00:47:56] Press probably a hundred,

[00:47:58] Dr. Daniel Elias Martin Herranz: [00:47:58] right?

[00:47:59] Dr. William Seeds, MD: [00:47:59] Right. [00:48:00] And probably every one of them has some valid aspect of it, of what it can do to improve your bench, press. Right. I think, I think what we're going to have is something that's going to give us a more concrete way of looking at timing of where some of these things may fit in the best, you know, report for that particular time in life with all of the environmental stressors.

[00:48:27] I mean, this is what it's hard for people to comprehend, but you're putting together a tremendous amount of data. To make predictions. And, um, that's, what's so exciting because it's, if you can think it right now in what we're talking about, I know people have to be sitting there going, well, maybe it could do this and I'm going to say, yes, it can.

[00:48:51] I'm going to say yes, it can. Cause because it's going to give us that kind of information. It already is giving us that information. And, and this is why you and [00:49:00] I, Carl, have to. Hang around for awhile to take advantage of all this information.

[00:49:04] Carl Lanore: [00:49:04] Oh, I know. I look, I, I, matter of fact, when I come back from the break, I'm going to tell everybody, I just started working with a fellow named bill taco, who trains.

[00:49:12] He does the diets for all the, a lot of pros have pros and I had to send them pictures this morning. And I looked at myself and I was. So disheartened to see how far off the path I've gotten. Now, I'm waiting for my chronometer test to come back. I'm going to be instituting some changes. So I'll have my tests done again, maybe in six months.

[00:49:29] And I'll look at my progress against what I learned from what I did at crunch comics. But I want to talk about that. I don't, I kind of gave the cat out of the bag for everybody, wants to sit through this, but my pictures this morning, I almost didn't send them. And I know how people feel, and that's what I want to talk about.

[00:49:45] If you feel like you're in a place where you can't get out of, it's not true, you can still get out of it. I look, the website is, is seeds.md/epigenetic hyphen test slash. If you want to take advantage of [00:50:00] this, it's got to be good for a month. So today is July 8th. So by the time, uh, August 10th rolls around, these won't be lasted.

[00:50:06] They won't be there. So if you want to do this, you better do it soon. Cause after this, the only way you get to do it is to go to a doctor and have him sell you the test. So this is a really, really unique opportunity that has never been done before. Uh, this type of this offer direct to the public. So check it out.

[00:50:23] Uh, Daniel, thank you for being here, dr. Seeds. Thanks for scrubbing up and getting the blood off of you before coming on camera today, I told him, I told him, I told Sean, I haven't bring a femur in with him.

[00:50:38] Alright. Good. See you guys. Thank you very much. Thank you.

[00:50:41] Dr. Daniel Elias Martin Herranz: [00:50:41] Alright, thanks so much.

[00:50:46] Carl Lanore: [00:50:46] And we're going to take one quick commercial break and we'll be right back for Braun and brain's finally meet

[00:50:56] welcome back. [00:51:00] So I am a officially. Taking control of both my health and my strength and fitness. And I really have, I've just been meandering. You know, I trained for four or five months in a row. I do a good job. I start seeing progress. I go off the deep end. I started drinking a lot on the weekends. I start living a life that isn't really the best for me.

[00:51:23] I abuse caffeine. I'm doing all these things that I know that are not in my own best interest. The fact that I am not sicker. It surprises me to be honest with you. Okay. But I have my challenges. And so I'm working with a fellow named bill taco. He's known as war, room nutrition. He's a wonderful human being.

[00:51:45] He, um, everybody he works with looks amazing. The guy knows what he's doing. He was introduced to me by Scott McNally. Thank you, Scott. And, uh, so. The deal was we talked yesterday. I says, okay, [00:52:00] you know, I'm ready to get serious. I'm going to ease into this. My, so one of my big problems is that I'm still very strong, but I have no conditioning.

[00:52:10] I don't think I could jog. A half mile right now. I mean, I could walk a mile, no problem, but I couldn't jog it. And that not being able to jog it is because of several reasons. It's not just my conditioning. It's the fact that my left leg is like a pirates peg leg after the surgeries that I had, uh, that can be changed.

[00:52:29] But right now, this is where I'm starting from. And I have the same neurological disorder that I've shared with all of you over the years that listen to the show that really killed my sister, but what killed her? Wasn't the disease. It's the, it was the way she was treated. She was treated for Parkinson's disease.

[00:52:42] She was given inappropriate drugs that wrecked her. Uh, she had deep brain implant for stimulation. Didn't do anything cause she never had Parkinson's disease. Okay. So the fact that I'm not using any of those drugs is part of the reason why I am fairing better than she did, but I have the same thing and it's [00:53:00] definitely, there's no doubt in my mind, that's autoimmune in nature.

[00:53:05] I think caffeine has something to do with it. I gotta be honest with you because like right now I'm back up to 600 milligrams of caffeine, anhydrous, or more a day to bang energy drinks in the morning or a total war and a bang later on in the day. And it's just, I don't, I have this addictive. Personality.

[00:53:22] I've known about it since I was a young man. I never injected heroin, but I snorted a lot of heroin and I snorted a lot of cocaine and I did a lot of drugs all growing up. I've been self medicating myself as something for some reason, for a long time. Well, caffeine is just become one of those, those drugs.

[00:53:39] I abuse it. I've been abusing it for years. I can go back to 2002. I was doing a gram of, uh, of, uh, caffeine anhydrous, uh, 600 milligrams before going into the gym and training 300 milligrams after training. Um, Why I haven't had a stroke or a heart attack yet just is a [00:54:00] Testament to my constitution, to be honest with you, but it's got me again.

[00:54:04] Caffeine has me again, and I know it's no good. And so I have to take my health seriously. Now I'm 62 years old and I run the risk of really having. Developing some serious problems. And hopefully I haven't developed them already. I talked about Alice static load the other day, an Allo status. If you caught the show less a week or so ago, where I finished up the show with the discussion about Alice static load analysis, I I'm managing a lot of things right now.

[00:54:30] I'm managing, um, nerve issues in my lower body. That have not only caused me to lose pretty much all the, um, lower body muscle that I've held for so, so long, but make it hard for me, increasingly harder for me to do simple things. Some days walking is a challenge. Um, and on top of that, you know, I have, uh, The lack of fitness that has [00:55:00] occurred from my lack of training consistently, and my lack of doing things that are good for me or eliminating things that are bad.

[00:55:08] So I made the decision to do this. I'm announcing it too, right? Because now I can't turn back. That's another thing, right? I'm not going to be able to say this and then not do something about it. And I'm working with bill taco. And he's going to get my nutrition straightened out. We've even talked about training, but I think I have the training part down and I'm sensible about my training.

[00:55:30] Like I said before, I'm still really strong, but I'm not fit. What puts me at a very rare risk of hurting myself of tearing something or injuring myself. So I'm really starting out really low. Like the bar is very low. My goal right now for the first month is to literally show up to the gym. Every day I'm supposed to and train whatever it is that I've decided I'm going to do that day.

[00:55:55] And I'm not going to be doing anything heavy. I'm going to do predominantly [00:56:00] machine movements and I'm going to start doing cardio again today. I got up yesterday. I got up in the morning and did cardio today. I got, you know, when you live with chronic pain jumping out of bed and running downstairs and getting on the treadmill or the Airdyne bike, just isn't exciting.

[00:56:16] It's not like we used to be. I used to, I, I would wake up in the morning. I would throw my sneakers on and my gym clothes on. I'd run out. When I lived in the apartment, when I was going through my divorce, I'd go to the, the little gym room they had there. Walk through the freezing cold in the middle of winter and get on the treadmill or get on the elliptical and turn on the TV, watch MTV.

[00:56:36] And I would do my 45 minutes hour of, of faster cardio consistently, religiously every single morning for years. And I exploited all of that health equity. That I had from doing that for a day. I mean, it took me a decade to go from 330 fat heartbroken, disgusting pounds to my [00:57:00] 50th birthday. Uh, where I was lean and muscular and felt amazing and had a great outlook on life and had no pain I lived with no, no chronic pain.

[00:57:09] Now, can I reverse a completely, I don't know. I have some tricks up my sleeve. You know, I have the knowledge of peptides. I'm getting ready to start a round a 28 day round of ARA two 90 or submitted tide. I am absolutely cutting the caffeine down. I can't just stop. Even bill taco said to me, So, what are you going to do to wean yourself off?

[00:57:32] Like it was, it was a given you can't just stop when you are addicted to caffeine, to this, this level of consumption. And so Aaron Singerman has been kind enough to send me a case of total war and I'm going to drink one only one a day. In the morning and I'm going to do that for probably the better part of a week or so.

[00:57:52] And then I'm going to cut it down to a half a bottle a day and I'll do that for the better part of a week or so. And then I'm gonna cut that down to a third and then eventually I'm going to wean it [00:58:00] off. Um, but bill said, okay, so here's what I need from you. He says, I need your pictures. I want them to be taken with the flash on.

[00:58:09] I don't want you to take advantage of the light to make you look better. I want to see your body, everything. You know, so I can really see where the fat is and all that sort of stuff. And last night, when I talked to him, I was like, yeah, no big deal. So this morning I got up, I weighed myself. I'm 228 pounds right now.

[00:58:27] And, uh, I've lost 10 pounds. It's all lower body. It's my glutes. My glutes are gone. I have no ass at all anymore. My hamstrings and my quads have shrunken. My left calf looks like a piece of wood. We cost it's shrunken so much from the two surgeries. Uh, so, you know, I got, I look like Frankenstein. I really, I looked at my body this morning.

[00:58:50] I almost didn't send the pictures. I almost said, Oh, F this, you know what? I don't need to be. I'll train myself, do my own diet. I'll just do an, obviously that [00:59:00] doesn't work. Cause I've been doing that for the past 10 years and sliding down this, this Hill slowly, you know, two steps forward. One step back.

[00:59:09] And I almost didn't want to send them, at least it says, just send it. You're not going to look like that forever. That's the goal. And I was like, okay. And I sent them to him and I looked at them and I am so sad. I can't tell you how sad I am when I see what I've let my body become just by not tending to it.

[00:59:27] And you know, and the older you get, the harder it is to keep it. That's true. And I was doing two steps forward, one step back always. And it's, it's really, I look at myself now and I go, Oh my God. Like, can I even, can I even turn this around? And so I sent the pictures to bill and he was just, you know, he's just such a kind decent guy.

[00:59:48] He's not gonna say, Holy crap, you hit the wall hard. My belly is bloated. I got to start hanging again. Cause my spine looks like it's getting a little crooked. Um, You know, I've got [01:00:00] fat around my back at the lower part of my back, where my ass used to be, and I don't even have an ass now. Um, you know, I got the peg leg on the left hand side with a foot that looks like it's a inch and a half shorter than it used to be.

[01:00:11] It looks like a claw. I mean, I'm a wreck. I'm a wreck, but I'm being positive. Uh, he, bill, bill said, boy, this is going to be such an exciting time for you. And I kind of feel like, well, he tells everybody that otherwise they just back out, they go, you know what? Forget about it. I don't want to do this after all.

[01:00:28] I don't want to, I don't want to admit to myself what I look like right now. Um, it's clear that I have a lot of muscle. There's no doubt about that. I mean, I look like a frigging gorilla. I really do. My stomach is somewhat distended. I really believe the caffeine has been contributing a lot to that. I am, we will see I'm going to get my epigenetic tests back and I'm working with Joel green a little bit too.

[01:00:52] I look like a frigging gorilla. I really do. Um, it's, it's just sad. And, uh, and, and this is [01:01:00] going to be a really long and arduous journey for me because I, when I did this the first time when I was in my forties, I had the energy and I didn't have the pain and I didn't have anything that was broken, ready.

[01:01:14] And I, you know, it was now. Motivating myself to get up and get downstairs on the treadmill. First thing in the morning is like, ridiculously, I mean, this morning, I just said to myself, Oh, F it, man, just lets go. And like I, I thought to myself, okay. So I feel like crap, I have pain that I need to walk for a while for it to go away.

[01:01:35] I'll just get on the treadmill and walk for a while. And that's what I did. And then I went, I had my protein drink. And within an hour, I was at the gym and I did my training session and exactly what I was supposed to do. Nothing heavy. Just show up, be there, be cognizant of what the task is. And I did it.

[01:01:52] And now it's one day at a time it's one day at a time, but I feel anybody out there who feels like. [01:02:00] They've just let it go too far. It's like, I almost drowned one time when I was in Yugoslavia, I was drinking all day and I swam really, really far out. It was a really, really hot day. And when I turned around and realized how far away the seashore was, cause I swam for so long, like an idiot, I kind of panicked a little bit.

[01:02:20] And right then when I panicked, I vomited. And H T if you're ever in deep water and you vomit, you go under you, cause you can't tread water while you're vomiting, right? It's this gut wrenching reaction, your body just folds up and becomes a weight sinker. And I swam back to the top of the water. I remember having vomited my hair because I had to swim back through my own vomit, uh, to find that th th the arrogant and I was about to vomit again.

[01:02:45] And I thought, Oh my God, I'm going to drown. And I remember there was a blink of a moment where I, I thought to myself, this may be it. And it's almost like I always got relaxed. Cause I thought, Oh, well I can't fight this. I'm going to [01:03:00] drown. That's kind of how I felt this morning. When I looked at those pictures, I kind of felt like, Oh my God, I'm too far under.

[01:03:08] I can't save myself. This is an impossibility. Like I have not only do I look at a shape and know what, not only is my fitness quote and it's so low, which that's not the thing, but I'm bouncing, battling against some autoimmune disorder that really like wants to continue to etch away at my health while I'm trying to add to it.

[01:03:29] So I am going to do this. If you're doing this right now, and you feel like you're in a hole and you need some, uh, mutual support, I'm thinking about starting a small zoom group. Email me at, on This email address is being protected from spambots. You need JavaScript enabled to view it.. If you're about to face a challenge, I don't care what that challenge is. Cure your own cancer.

[01:03:51] Get back in shape, uh, you know, overcome some sort of adversity in your life. I think we all can use each [01:04:00] other to kind of motivate us from different ways. We each have a gift to give and we each can give a gift. And so if you're at a point in your life, Where you feel like I'm going to really need something to get past this.

[01:04:14] I'm going to really need something to fix this. Email This email address is being protected from spambots. You need JavaScript enabled to view it., no matter what your challenges are and we'll work together. And we'll find other people to work together and we'll pull our resources and, and gather our hope and motivate each other because I need it, man. I'm serious.

[01:04:34] Um, This is a hard pill for me to swallow. It really is. I look, we're going to end the show on that note. And hope everybody enjoyed today's show. Don't forget, take advantage of this epigenetic test. It really is an amazing test and you're never going to get it for this price. I think it's like $200 to $250 or something like that on their website.

[01:04:52] It's only a thousand dollar test. Go to seeds.md. Slash epigenetic [01:05:00] dash test and another slash to sign up and get this first test done, uh, because I'm I'm, I am so hopeful about this test, giving me the map of the things I need to get out of my life that are the reason for my auto-immunity. I'm very, very confident it's going to help.

[01:05:21] Okay. See you tomorrow. Thanks for watching and listening today. Please share this show. Uh, the greatest thing you can do, uh, to not only help me and other people. But it really is, is to share the show. Okay, thanks. See you tomorrow. [01:06:00] .