[2:40] what led to this research?

• The buzz about NAD in combination with the knowledge that the mitochondria takes it form the cytosol.

[5:01] could NAD supplementation be less useful without the transporting protein that carries it from cytosol to mitochondria?

• A deficiency of NAD would be the first thing to look at because if this transporter did not function, bigger problems would occur.

[7;50] What about supplementing with ribose and niacin?

• NAD would be a more direct path.

[8;59] this transporter is discussed.

• No previously known function for this gene.

[12:15] Could a slight deficiency of NAD trigger a hormetic strengthening of the mitochondria?

[13:00] No correlation between this gene and aging has been discovered yet.

[15:30] could senescent cells hinder the  production of this transporter?

[16;50] Are there any epigentic triggers?

• Possibly PGC-1 alpha.

[17:50] What disease states could this research affect?

• Various forms of cancer.
• Heart failure.
• Possibly sarcopenia
• Mitochondrial myopathy.

[29:50] exercise: does it affect MCART-1?

• No direct evidence but probably in a manner.

[43:20] MOT-C: Is it working in the same manner as NAD+

[58:05] CBD based workout products.

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